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1989年学术文章 სამეცნიერო სტატია article científic artículo científico publicado en 1989 1989年學術文章 مقالة علمية mokslinis straipsnis article scientific 1989年学术文章 vitenskapelig artikkel επιστημονικό άρθρο tieteellinen artikkeli artikulong pang-agham научни чланак vedecký článok 1989年学术文章 videnskabelig artikel (udgivet 1989) im März 1989 veröffentlichter wissenschaftlicher Artikel article scientifique publié en 1989 1989年學術文章 1989年学术文章 artigo científico (publicado na 1989) vitskapeleg artikkel 1989年の論文 наукова стаття, опублікована в березні 1989 vetenskaplig artikel teaduslik artikkel мақолаи илмӣ مقالهٔ علمی سائنسی مضمون bilimsel makale 1989 nî lūn-bûn articol științific artigo científico (publicado na 1989) գիտական հոդված научни чланак 1989年學術文章 научна статия บทความทางวิทยาศาสตร์ wetenschappelijk artikel artigo científico 1989年学术文章 1989年學術文章 bài báo khoa học מאמר מדעי scienca artikolo artykuł naukowy artículu científicu espublizáu en marzu de 1989 artikull shkencor naučni članak scientific article published on 01 March 1989 vědecký článek tudományos cikk мақолаи илмӣ 1989年學術文章 articolo scientifico научная статья ১৯৮৯-এ প্রকাশিত বৈজ্ঞানিক নিবন্ধ 1989年學術文章 1989년 논문
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Steinmann B Royce PM Moser U
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Ehlers-Danlos syndrome type VI with normal lysyl hydroxylase activity cannot be explained by a defect in cellular uptake of ascorbic acid Ehlers-Danlos syndrome type VI with normal lysyl hydroxylase activity cannot be explained by a defect in cellular uptake of ascorbic acid
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Ehlers-Danlos syndrome type VI with normal lysyl hydroxylase activity cannot be explained by a defect in cellular uptake of ascorbic acid Ehlers-Danlos syndrome type VI with normal lysyl hydroxylase activity cannot be explained by a defect in cellular uptake of ascorbic acid
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Ehlers-Danlos syndrome type VI with normal lysyl hydroxylase activity cannot be explained by a defect in cellular uptake of ascorbic acid Ehlers-Danlos syndrome type VI with normal lysyl hydroxylase activity cannot be explained by a defect in cellular uptake of ascorbic acid
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Ehlers-Danlos syndrome type VI with normal lysyl hydroxylase activity cannot be explained by a defect in cellular uptake of ascorbic acid
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