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1999年學術文章 1999年学术文章 مقالهٔ علمی 1999年の論文 artykuł naukowy vedecký článok επιστημονικό άρθρο 1999年学术文章 artikulong pang-agham мақолаи илмӣ ১৯৯৯-এ প্রকাশিত বৈজ্ঞানিক নিবন্ধ מאמר מדעי articol științific مقالة علمية 1999年學術文章 article scientific articolo scientifico bilimsel makale artículu científicu 1999年學術文章 1999년 논문 научни чланак наукова стаття, опублікована в лютому 1999 1999年学术文章 videnskabelig artikel (udgivet 1999) บทความทางวิทยาศาสตร์ wissenschaftlicher Artikel научна статия научни чланак mokslinis straipsnis 1999年學術文章 vědecký článek teaduslik artikkel artigo científico (publicado na 1999) 1999年学术文章 سائنسی مضمون գիտական հոդված vitskapeleg artikkel 1999 nî lūn-bûn wetenschappelijk artikel 1999年學術文章 article científic artigo científico (publicado na 1999) naučni članak სამეცნიერო სტატია vetenskaplig artikel bài báo khoa học artikull shkencor tieteellinen artikkeli scientific article article scientifique (publié 1999) scienca artikolo tudományos cikk vitenskapelig artikkel 1999年學術文章 1999年学术文章 artigo científico мақолаи илмӣ научная статья artículo científico publicado en 1999
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Tissue-specific knockout of the insulin receptor in pancreatic beta cells creates an insulin secretory defect similar to that in type 2 diabetes. Tissue-specific knockout of the insulin receptor in pancreatic beta cells creates an insulin secretory defect similar to that in type 2 diabetes.
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Tissue-specific knockout of the insulin receptor in pancreatic beta cells creates an insulin secretory defect similar to that in type 2 diabetes. Tissue-specific knockout of the insulin receptor in pancreatic beta cells creates an insulin secretory defect similar to that in type 2 diabetes.
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Tissue-specific knockout of the insulin receptor in pancreatic beta cells creates an insulin secretory defect similar to that in type 2 diabetes. Tissue-specific knockout of the insulin receptor in pancreatic beta cells creates an insulin secretory defect similar to that in type 2 diabetes.
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Tissue-specific knockout of the insulin receptor in pancreatic beta cells creates an insulin secretory defect similar to that in type 2 diabetes.
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