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naučni članak 2013年學術文章 2013年の論文 artikulong pang-agham vitskapeleg artikkel 2013年学术文章 научна статия מאמר מדעי artikel ilmiah artículu científicu 2013年学术文章 наукова стаття, опублікована у квітні 2013 مقالة علمية نشرت في 22 أبريل 2013 სამეცნიერო სტატია bài báo khoa học tieteellinen artikkeli tudományos cikk 2013년 논문 2013年学术文章 ২২ এপ্রিল ২০১৩-এ প্রকাশিত বৈজ্ঞানিক নিবন্ধ 2013 nî lūn-bûn scienca artikolo 2013年学术文章 επιστημονικό άρθρο 2013年學術文章 wissenschaftlicher Artikel artigo científico articol științific научная статья videnskabelig artikel artykuł naukowy 2013年學術文章 artigo científico bilimsel makale scientific article published on 22 April 2013 article científic 2013年學術文章 artikull shkencor artículo científico publicado en 2013 teaduslik artikkel научни чланак article scientifique vědecký článek article scientific vitenskapelig artikkel บทความทางวิทยาศาสตร์ vetenskaplig artikel 2013年学术文章 scientific article published on 22 April 2013 2013年學術文章 artigo científico мақолаи илмӣ научни чланак wetenschappelijk artikel vedecký článok 2013年学术文章 articolo scientifico scientific article published on 22 April 2013
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Yung-Mei Chao Ming-Derg Lai Julie Y H Chan
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Redox-sensitive endoplasmic reticulum stress and autophagy at rostral ventrolateral medulla contribute to hypertension in spontaneously hypertensive rats. Redox-sensitive endoplasmic reticulum stress and autophagy at rostral ventrolateral medulla contribute to hypertension in spontaneously hypertensive rats.
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Redox-sensitive endoplasmic reticulum stress and autophagy at rostral ventrolateral medulla contribute to hypertension in spontaneously hypertensive rats. Redox-sensitive endoplasmic reticulum stress and autophagy at rostral ventrolateral medulla contribute to hypertension in spontaneously hypertensive rats.
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Redox-sensitive endoplasmic reticulum stress and autophagy at rostral ventrolateral medulla contribute to hypertension in spontaneously hypertensive rats. Redox-sensitive endoplasmic reticulum stress and autophagy at rostral ventrolateral medulla contribute to hypertension in spontaneously hypertensive rats.
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Redox-sensitive endoplasmic reticulum stress and autophagy at rostral ventrolateral medulla contribute to hypertension in spontaneously hypertensive rats.
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