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мақолаи илмӣ artículo científico publicado en 2014 2014年学术文章 научни чланак 2014年学术文章 artigo científico ১৭ সেপ্টেম্বর ২০১৪-এ প্রকাশিত বৈজ্ঞানিক নিবন্ধ scientific article published on 17 September 2014 2014年學術文章 2014年学术文章 artikull shkencor 2014年學術文章 scientific article published on 17 September 2014 artigo científico vědecký článek บทความทางวิทยาศาสตร์ مقالة علمية نشرت في 17 سبتمبر 2014 article scientifique articolo scientifico επιστημονικό άρθρο artículu científicu article científic 2014年の論文 vedecký článok tieteellinen artikkeli 2014 nî lūn-bûn 2014년 논문 artikel ilmiah 2014年學術文章 article scientific vitenskapelig artikkel 2014年学术文章 wetenschappelijk artikel 2014年学术文章 научная статья wissenschaftlicher Artikel artykuł naukowy teaduslik artikkel vitskapeleg artikkel scientific article published on 17 September 2014 articol științific artigo científico videnskabelig artikel tudományos cikk scienca artikolo სამეცნიერო სტატია 2014年學術文章 artikulong pang-agham bài báo khoa học научни чланак bilimsel makale 2014年學術文章 naučni članak наукова стаття, опублікована у вересні 2014 vetenskaplig artikel научна статия 2014年学术文章 מאמר מדעי
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Xianming Lin Chunling Chen Heather O'Malley Monique Foster Glenn I Fishman Mingliang Zhang Akshay Shekhar
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Scn1b deletion leads to increased tetrodotoxin-sensitive sodium current, altered intracellular calcium homeostasis and arrhythmias in murine hearts. Scn1b deletion leads to increased tetrodotoxin-sensitive sodium current, altered intracellular calcium homeostasis and arrhythmias in murine hearts.
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Scn1b deletion leads to increased tetrodotoxin-sensitive sodium current, altered intracellular calcium homeostasis and arrhythmias in murine hearts. Scn1b deletion leads to increased tetrodotoxin-sensitive sodium current, altered intracellular calcium homeostasis and arrhythmias in murine hearts.
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Scn1b deletion leads to increased tetrodotoxin-sensitive sodium current, altered intracellular calcium homeostasis and arrhythmias in murine hearts. Scn1b deletion leads to increased tetrodotoxin-sensitive sodium current, altered intracellular calcium homeostasis and arrhythmias in murine hearts.
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Scn1b deletion leads to increased tetrodotoxin-sensitive sodium current, altered intracellular calcium homeostasis and arrhythmias in murine hearts.
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