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2006년 논문 επιστημονικό άρθρο 2006年学术文章 2006年學術文章 artigo científico artículo científico publicado en 2006 tudományos cikk article scientifique სამეცნიერო სტატია научна статия научни чланак scienca artikolo artykuł naukowy 2006年学术文章 bài báo khoa học 2006年学术文章 2006年学术文章 artikull shkencor vedecký článok article scientific vitskapeleg artikkel article científic vědecký článek мақолаи илмӣ 2006年の論文 научни чланак articol științific מאמר מדעי scientific article published on 27 February 2006 artigo científico bilimsel makale ২৭ ফেব্রুয়ারি ২০০৬-এ প্রকাশিত বৈজ্ঞানিক নিবন্ধ научная статья teaduslik artikkel wissenschaftlicher Artikel artikel ilmiah 2006年学术文章 artikulong pang-agham наукова стаття, опублікована в лютому 2006 vitenskapelig artikkel videnskabelig artikel 2006年學術文章 articolo scientifico naučni članak บทความทางวิทยาศาสตร์ scientific article published on 27 February 2006 scientific article published on 27 February 2006 artículu científicu artigo científico tieteellinen artikkeli vetenskaplig artikel مقالة علمية نشرت في 27 فبراير 2006 2006年学术文章 2006年學術文章 2006年學術文章 wetenschappelijk artikel 2006年學術文章 2006 nî lūn-bûn
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Sumio Sugano Masashi Ikushima Satoshi Takeda Toshio Ogihara Masaharu Kaibe Mitsuru Ohishi Atsushi Shiota Norihisa Ito Yuji Tatara Koichi Yamamoto Tomohiro Katsuya
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Deletion of angiotensin-converting enzyme 2 accelerates pressure overload-induced cardiac dysfunction by increasing local angiotensin II. Deletion of angiotensin-converting enzyme 2 accelerates pressure overload-induced cardiac dysfunction by increasing local angiotensin II.
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Deletion of angiotensin-converting enzyme 2 accelerates pressure overload-induced cardiac dysfunction by increasing local angiotensin II. Deletion of angiotensin-converting enzyme 2 accelerates pressure overload-induced cardiac dysfunction by increasing local angiotensin II.
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Deletion of angiotensin-converting enzyme 2 accelerates pressure overload-induced cardiac dysfunction by increasing local angiotensin II. Deletion of angiotensin-converting enzyme 2 accelerates pressure overload-induced cardiac dysfunction by increasing local angiotensin II.
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Deletion of angiotensin-converting enzyme 2 accelerates pressure overload-induced cardiac dysfunction by increasing local angiotensin II.
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