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scienca artikolo 2008年学术文章 naučni članak مقالهٔ علمی artículo científico publicado en 2008 บทความทางวิทยาศาสตร์ articolo scientifico научная статья teaduslik artikkel 2008年學術文章 2008年学术文章 vetenskaplig artikel 2008년 논문 2008 nî lūn-bûn наукова стаття, опублікована в січні 2008 tudományos cikk scientific article artikull shkencor επιστημονικό άρθρο научни чланак 2008年學術文章 article scientifique (publié 2008) ২০০৮-এ প্রকাশিত বৈজ্ঞানিক নিবন্ধ artigo científico (publicado na 2008) wissenschaftlicher Artikel article scientific სამეცნიერო სტატია 2008年の論文 vědecký článek מאמר מדעי bài báo khoa học 2008年學術文章 vitenskapelig artikkel artigo científico научна статия wetenschappelijk artikel 2008年学术文章 2008年學術文章 mokslinis straipsnis article científic tieteellinen artikkeli vitskapeleg artikkel 2008年學術文章 2008年学术文章 vedecký článok سائنسی مضمون articol științific artikulong pang-agham artykuł naukowy мақолаи илмӣ научни чланак 2008年學術文章 2008年学术文章 գիտական հոդված artigo científico (publicado na 2008) bilimsel makale videnskabelig artikel (udgivet 2008) artículu científicu مقالة علمية мақолаи илмӣ
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Lepu Zhou Kai Schönig Hermann Bujard Inna Khlistunova Jacek Biernat Gabriele Rune Eva-Maria Mandelkow Katrin Eckermann Dagmar Drexler Olga Petrova Maria-Magdalena Mocanu Eckhard Mandelkow Astrid Nissen
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The potential for beta-structure in the repeat domain of tau protein determines aggregation, synaptic decay, neuronal loss, and coassembly with endogenous Tau in inducible mouse models of tauopathy. The potential for beta-structure in the repeat domain of tau protein determines aggregation, synaptic decay, neuronal loss, and coassembly with endogenous Tau in inducible mouse models of tauopathy.
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The potential for beta-structure in the repeat domain of tau protein determines aggregation, synaptic decay, neuronal loss, and coassembly with endogenous Tau in inducible mouse models of tauopathy. The potential for beta-structure in the repeat domain of tau protein determines aggregation, synaptic decay, neuronal loss, and coassembly with endogenous Tau in inducible mouse models of tauopathy.
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The potential for beta-structure in the repeat domain of tau protein determines aggregation, synaptic decay, neuronal loss, and coassembly with endogenous Tau in inducible mouse models of tauopathy. The potential for beta-structure in the repeat domain of tau protein determines aggregation, synaptic decay, neuronal loss, and coassembly with endogenous Tau in inducible mouse models of tauopathy.
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The potential for beta-structure in the repeat domain of tau protein determines aggregation, synaptic decay, neuronal loss, and coassembly with endogenous Tau in inducible mouse models of tauopathy
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