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artikel ilmiah videnskabelig artikel vedecký článok 2008年学术文章 artikull shkencor 2008年學術文章 bilimsel makale научни чланак наукова стаття, опублікована в жовтні 2008 অক্টোবর ২০০৮-এ প্রকাশিত বৈজ্ঞানিক নিবন্ধ 2008年學術文章 artigo científico научна статия scientific article published in October 2008 artykuł naukowy naučni članak vetenskaplig artikel wetenschappelijk artikel vitenskapelig artikkel articol științific 2008年学术文章 2008年の論文 2008年学术文章 2008年学术文章 article scientific wissenschaftlicher Artikel artigo científico 2008年學術文章 article científic 2008년 논문 2008年學術文章 vědecký článek tudományos cikk bài báo khoa học vitskapeleg artikkel 2008年学术文章 מאמר מדעי บทความทางวิทยาศาสตร์ scienca artikolo επιστημονικό άρθρο мақолаи илмӣ სამეცნიერო სტატია 2008年学术文章 artikulong pang-agham artículo científico publicado en 2008 artículu científicu article scientifique tieteellinen artikkeli scientific article published in October 2008 2008年學術文章 artigo científico научная статья научни чланак scientific article published in October 2008 articolo scientifico teaduslik artikkel 2008 nî lūn-bûn مقالة علمية نشرت في أكتوبر 2008
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Bruno Levy Chantal Montemont Sebastien Gibot
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Increased aerobic glycolysis through beta2 stimulation is a common mechanism involved in lactate formation during shock states Increased aerobic glycolysis through beta2 stimulation is a common mechanism involved in lactate formation during shock states
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Increased aerobic glycolysis through beta2 stimulation is a common mechanism involved in lactate formation during shock states Increased aerobic glycolysis through beta2 stimulation is a common mechanism involved in lactate formation during shock states
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Increased aerobic glycolysis through beta2 stimulation is a common mechanism involved in lactate formation during shock states Increased aerobic glycolysis through beta2 stimulation is a common mechanism involved in lactate formation during shock states
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Increased aerobic glycolysis through beta2 stimulation is a common mechanism involved in lactate formation during shock states
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10.1097/SHK.0B013E318167378F