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научни чланак artikulong pang-agham articol științific artikel ilmiah artigo científico scientific article published in January 2005 scientific article published in January 2005 article scientific vedecký článok 2005年學術文章 2005年学术文章 vědecký článek artículo científico publicado en 2005 บทความทางวิทยาศาสตร์ 2005年學術文章 2005年学术文章 مقالة علمية نشرت في يناير 2005 vitenskapelig artikkel 2005年学术文章 мақолаи илмӣ artykuł naukowy 2005年学术文章 artikull shkencor научная статья article científic videnskabelig artikel scientific article published in January 2005 wetenschappelijk artikel articolo scientifico scienca artikolo artigo científico tudományos cikk 2005年学术文章 מאמר מדעי სამეცნიერო სტატია naučni članak vetenskaplig artikel 2005年の論文 vitskapeleg artikkel artículu científicu জানুয়ারি ২০০৫-এ প্রকাশিত বৈজ্ঞানিক নিবন্ধ научни чланак 2005年学术文章 2005 nî lūn-bûn bilimsel makale article scientifique 2005년 논문 artigo científico научна статия tieteellinen artikkeli 2005年學術文章 bài báo khoa học наукова стаття, опублікована в січні 2005 2005年學術文章 wissenschaftlicher Artikel επιστημονικό άρθρο teaduslik artikkel 2005年學術文章
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Lourdes Massieu Octavio García
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Increased mitochondrial respiration maintains the mitochondrial membrane potential and promotes survival of cerebellar neurons in an endogenous model of glutamate receptor activation. Increased mitochondrial respiration maintains the mitochondrial membrane potential and promotes survival of cerebellar neurons in an endogenous model of glutamate receptor activation.
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Increased mitochondrial respiration maintains the mitochondrial membrane potential and promotes survival of cerebellar neurons in an endogenous model of glutamate receptor activation. Increased mitochondrial respiration maintains the mitochondrial membrane potential and promotes survival of cerebellar neurons in an endogenous model of glutamate receptor activation.
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Increased mitochondrial respiration maintains the mitochondrial membrane potential and promotes survival of cerebellar neurons in an endogenous model of glutamate receptor activation. Increased mitochondrial respiration maintains the mitochondrial membrane potential and promotes survival of cerebellar neurons in an endogenous model of glutamate receptor activation.
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Increased mitochondrial respiration maintains the mitochondrial membrane potential and promotes survival of cerebellar neurons in an endogenous model of glutamate receptor activation
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