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article scientifique artículo científico publicado en 2003 artigo científico мақолаи илмӣ article científic 2003年の論文 scienca artikolo 2003年学术文章 artikel ilmiah 2003年學術文章 מאמר מדעי tieteellinen artikkeli 2003年學術文章 bilimsel makale teaduslik artikkel 2003 nî lūn-bûn tudományos cikk 2003年學術文章 scientific article published on 9 October 2003 научни чланак articol științific 2003년 논문 2003 թվականի հոկտեմբերի 9-ին հրատարակված գիտական հոդված artikulong pang-agham artículu científicu 2003年學術文章 научни чланак scientific article published on 9 October 2003 научная статья scientific article published on 9 October 2003 wissenschaftlicher Artikel บทความทางวิทยาศาสตร์ vitskapeleg artikkel 2003年学术文章 επιστημονικό άρθρο vedecký článok 2003年学术文章 наукова стаття, опублікована в жовтні 2003 სამეცნიერო სტატია artigo científico 2003年学术文章 ৯ অক্টোবর ২০০৩-এ প্রকাশিত বৈজ্ঞানিক নিবন্ধ vetenskaplig artikel videnskabelig artikel vitenskapelig artikkel 2003年学术文章 bài báo khoa học artikull shkencor artykuł naukowy wetenschappelijk artikel научна статия artigo científico 2003年學術文章 2003年学术文章 article scientific articolo scientifico naučni članak مقالة علمية نشرت في 9 أكتوبر 2003 vědecký článek
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Per Hellstrand Andreas Bergdahl Karl Dreja Jonas Broman
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Cholesterol depletion impairs vascular reactivity to endothelin-1 by reducing store-operated Ca2+ entry dependent on TRPC1. Cholesterol depletion impairs vascular reactivity to endothelin-1 by reducing store-operated Ca2+ entry dependent on TRPC1.
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Cholesterol depletion impairs vascular reactivity to endothelin-1 by reducing store-operated Ca2+ entry dependent on TRPC1. Cholesterol depletion impairs vascular reactivity to endothelin-1 by reducing store-operated Ca2+ entry dependent on TRPC1.
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Cholesterol depletion impairs vascular reactivity to endothelin-1 by reducing store-operated Ca2+ entry dependent on TRPC1. Cholesterol depletion impairs vascular reactivity to endothelin-1 by reducing store-operated Ca2+ entry dependent on TRPC1.
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Cholesterol depletion impairs vascular reactivity to endothelin-1 by reducing store-operated Ca2+ entry dependent on TRPC1.
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