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artículo científico publicado en 2002 vedecký článok мақолаи илмӣ 2002年学术文章 scienca artikolo artikulong pang-agham articol științific научная статья мақолаи илмӣ مقالهٔ علمی artigo científico (publicado na 2002) научни чланак teaduslik artikkel 2002年學術文章 2002年学术文章 سائنسی مضمون wissenschaftlicher Artikel 2002 nî lūn-bûn наукова стаття, опублікована у квітні 2002 2002年学术文章 article scientifique (publié 2002) tudományos cikk գիտական հոդված artigo científico 2002年の論文 vetenskaplig artikel מאמר מדעי wetenschappelijk artikel ২০০২-এ প্রকাশিত বৈজ্ঞানিক নিবন্ধ article científic 2002年學術文章 บทความทางวิทยาศาสตร์ artikull shkencor 2002年学术文章 научна статия 2002년 논문 vitskapeleg artikkel artículu científicu espublizáu en 2002 vědecký článek mokslinis straipsnis article scientific 2002年學術文章 επιστημονικό άρθρο 2002年學術文章 scientific article vitenskapelig artikkel tieteellinen artikkeli bài báo khoa học 2002年學術文章 2002年学术文章 videnskabelig artikel (udgivet 2002) naučni članak articolo scientifico bilimsel makale artigo científico (publicado na 2002) مقالة علمية 2002年學術文章 artykuł naukowy სამეცნიერო სტატია научни чланак
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Dieter Swandulla Johannes Schenkel Hanns-Ulrich Zeilhofer Jörg von Wegerer Hans Weiher
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Disease-specific human glycine receptor alpha1 subunit causes hyperekplexia phenotype and impaired glycine- and GABA(A)-receptor transmission in transgenic mice. Disease-specific human glycine receptor alpha1 subunit causes hyperekplexia phenotype and impaired glycine- and GABA
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Disease-specific human glycine receptor alpha1 subunit causes hyperekplexia phenotype and impaired glycine- and GABA(A)-receptor transmission in transgenic mice. Disease-specific human glycine receptor alpha1 subunit causes hyperekplexia phenotype and impaired glycine- and GABA
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Disease-specific human glycine receptor alpha1 subunit causes hyperekplexia phenotype and impaired glycine- and GABA Disease-specific human glycine receptor alpha1 subunit causes hyperekplexia phenotype and impaired glycine- and GABA(A)-receptor transmission in transgenic mice.
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Disease-specific human glycine receptor alpha1 subunit causes hyperekplexia phenotype and impaired glycine- and GABA(A)-receptor transmission in transgenic mice
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