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artikel ilmiah 2005年論文 2005年論文 artikulong pang-agham научна статия scientific article published on 22 March 2005 article científic мақолаи илмӣ tudományos cikk naučni članak 2005年論文 wissenschaftlicher Artikel научни чланак article scientific articol științific vitenskapelig artikkel บทความทางวิทยาศาสตร์ სამეცნიერო სტატია 2005年論文 scientific article published on 22 March 2005 2005年论文 tieteellinen artikkeli vedecký článok scienca artikolo 2005年论文 научная статья teaduslik artikkel 2005年论文 artigo científico наукова стаття, опублікована в березні 2005 επιστημονικό άρθρο wetenschappelijk artikel 2005年论文 artykuł naukowy vědecký článek vitskapeleg artikkel مقالة علمية نشرت في 22 مارس 2005 artigo científico vetenskaplig artikel научни чланак bilimsel makale 2005년 논문 artikull shkencor artículu científicu espublizáu en 2005 2005年论文 2005年の論文 bài báo khoa học articolo scientifico scientific article published on 22 March 2005 2005年論文 article scientifique 2005年论文 artigo científico artículo científico publicado en 2005 ২২ মার্চ ২০০৫-এ প্রকাশিত বৈজ্ঞানিক নিবন্ধ videnskabelig artikel מאמר מדעי 2005 nî lūn-bûn
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Peer B Jacobson Bradley Zinker James T Link Marlena Grynfarb Bach Nga-Nguyen Benjamin C Lane Steven Fung Phong T Nguyen Thomas Fey Noah Tu Marie Osterland Terry J Opgenorth David W A Beno Amanda Mika Suad Efendic Denise Wilcox Thomas W von Geldern Philip R Kym Kennan Marsh Jiahong Wang Tomas Barkhem Lars Ohman Annika Goos-Nilsson
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Hepatic glucocorticoid receptor antagonism is sufficient to reduce elevated hepatic glucose output and improve glucose control in animal models of type 2 diabetes Hepatic glucocorticoid receptor antagonism is sufficient to reduce elevated hepatic glucose output and improve glucose control in animal models of type 2 diabetes
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Hepatic glucocorticoid receptor antagonism is sufficient to reduce elevated hepatic glucose output and improve glucose control in animal models of type 2 diabetes Hepatic glucocorticoid receptor antagonism is sufficient to reduce elevated hepatic glucose output and improve glucose control in animal models of type 2 diabetes
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Hepatic glucocorticoid receptor antagonism is sufficient to reduce elevated hepatic glucose output and improve glucose control in animal models of type 2 diabetes Hepatic glucocorticoid receptor antagonism is sufficient to reduce elevated hepatic glucose output and improve glucose control in animal models of type 2 diabetes
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Hepatic glucocorticoid receptor antagonism is sufficient to reduce elevated hepatic glucose output and improve glucose control in animal models of type 2 diabetes
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