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vitskapeleg artikkel 1999年論文 artikull shkencor bài báo khoa học мақолаи илмӣ artykuł naukowy 1999年論文 գիտական հոդված հրատարակված 1999 թվականի սեպտեմբերին научни чланак wetenschappelijk artikel vědecký článek artigo científico 1999年論文 scienca artikolo scientific article published on September 1999 articolo scientifico 1999 nî lūn-bûn 1999年论文 artículu científicu espublizáu en 1999 1999년 논문 1999年论文 artikel ilmiah article scientifique artikulong pang-agham teaduslik artikkel naučni članak სამეცნიერო სტატია наукова стаття, опублікована у вересні 1999 1999年论文 bilimsel makale artículo científico publicado en 1999 scientific article published on September 1999 บทความทางวิทยาศาสตร์ wissenschaftlicher Artikel article científic научна статия tieteellinen artikkeli scientific article published on September 1999 1999年論文 1999年の論文 مقالة علمية نشرت في سبتمبر 1999 articol științific 1999年论文 vedecký článok επιστημονικό άρθρο article scientific vitenskapelig artikkel videnskabelig artikel tudományos cikk vetenskaplig artikel artigo científico מאמר מדעי সেপ্টেম্বর ১৯৯৯-এ প্রকাশিত বৈজ্ঞানিক নিবন্ধ 1999年論文 научни чланак научная статья artigo científico 1999年论文 1999年论文
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The new apolipoprotein A-I variant leu(174) --> Ser causes hereditary cardiac amyloidosis, and the amyloid fibrils are constituted by the 93-residue N-terminal polypeptide The new apolipoprotein A-I variant leu(174) --> Ser causes hereditary cardiac amyloidosis, and the amyloid fibrils are constituted by the 93-residue N-terminal polypeptide
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The new apolipoprotein A-I variant leu(174) --> Ser causes hereditary cardiac amyloidosis, and the amyloid fibrils are constituted by the 93-residue N-terminal polypeptide The new apolipoprotein A-I variant leu(174) --> Ser causes hereditary cardiac amyloidosis, and the amyloid fibrils are constituted by the 93-residue N-terminal polypeptide
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The new apolipoprotein A-I variant leu(174) --> Ser causes hereditary cardiac amyloidosis, and the amyloid fibrils are constituted by the 93-residue N-terminal polypeptide The new apolipoprotein A-I variant leu(174) --> Ser causes hereditary cardiac amyloidosis, and the amyloid fibrils are constituted by the 93-residue N-terminal polypeptide
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The new apolipoprotein A-I variant leu(174) --> Ser causes hereditary cardiac amyloidosis, and the amyloid fibrils are constituted by the 93-residue N-terminal polypeptide
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