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2017年学术文章 2017年學術文章 vitenskapelig artikkel სამეცნიერო სტატია artigo científico (publicado na 2017) научная статья artículu científicu espublizáu en 2017 artykuł naukowy artikulong pang-agham videnskabelig artikel (udgivet 2017) 2017年学术文章 2017年學術文章 2017年学术文章 مقالة علمية نشرت في 15 يناير 2017 naučni članak artigo científico (publicado na 2017) scientific article published on 15 January 2017 ২০১৭-এ প্রকাশিত বৈজ্ঞানিক নিবন্ধ article scientific wetenschappelijk artikel 2017年学术文章 scienca artikolo επιστημονικό άρθρο vedecký článok مقالهٔ علمی tieteellinen artikkeli bilimsel makale vědecký článek artikel ilmiah 2017 nî lūn-bûn 2017年学术文章 2017年學術文章 wissenschaftlicher Artikel 2017年學術文章 мақолаи илмӣ научни чланак գիտական հոդված บทความทางวิทยาศาสตร์ teaduslik artikkel scientific article published on 15 January 2017 artículo científico publicado en 2017 article científic 2017年學術文章 научни чланак 2017年學術文章 научна статия 2017년 논문 vetenskaplig artikel artikull shkencor سائنسی مضمون наукова стаття, опублікована в січні 2017 bài báo khoa học article scientifique (publié 2017) artigo científico articol științific מאמר מדעי мақолаи илмӣ mokslinis straipsnis scientific article published on 15 January 2017 vitskapeleg artikkel articolo scientifico tudományos cikk 2017年の論文
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Diltiazem prevents stress-induced contractile deficits in cardiomyocytes, but does not reverse the cardiomyopathy phenotype in Mybpc3-knock-in mice Diltiazem prevents stress-induced contractile deficits in cardiomyocytes, but does not reverse the cardiomyopathy phenotype in Mybpc3-knock-in mice
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Diltiazem prevents stress-induced contractile deficits in cardiomyocytes, but does not reverse the cardiomyopathy phenotype in Mybpc3-knock-in mice Diltiazem prevents stress-induced contractile deficits in cardiomyocytes, but does not reverse the cardiomyopathy phenotype in Mybpc3-knock-in mice
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Diltiazem prevents stress-induced contractile deficits in cardiomyocytes, but does not reverse the cardiomyopathy phenotype in Mybpc3-knock-in mice Diltiazem prevents stress-induced contractile deficits in cardiomyocytes, but does not reverse the cardiomyopathy phenotype in Mybpc3-knock-in mice
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