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επιστημονικό άρθρο 2003年论文 мақолаи илмӣ wissenschaftlicher Artikel teaduslik artikkel artikull shkencor naučni članak artikel ilmiah vitenskapelig artikkel научни чланак bilimsel makale научна статия article científic scientific article published on October 2003 tieteellinen artikkeli наукова стаття, опублікована в жовтні 2003 2003年论文 article scientifique научни чланак vitskapeleg artikkel অক্টোবর ২০০৩-এ প্রকাশিত বৈজ্ঞানিক নিবন্ধ 2003年論文 บทความทางวิทยาศาสตร์ 2003年论文 2003年の論文 2003年论文 vědecký článek article scientific tudományos cikk artykuł naukowy artikulong pang-agham artigo científico 2003年論文 2003年論文 2003年論文 articolo scientifico scientific article published on October 2003 vedecký článok научная статья artículu científicu espublizáu en 2003 2003年論文 مقالة علمية تنشرت ف 1 أكتوبر 2003 2003年论文 wetenschappelijk artikel bài báo khoa học scienca artikolo מאמר מדעי 2003年论文 artigo científico 2003년 논문 artículo científico publicado en 2003 videnskabelig artikel مقالة علمية نشرت في أكتوبر 2003 vetenskaplig artikel 2003 nî lūn-bûn articol științific scientific article published on October 2003 სამეცნიერო სტატია artigo científico
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Astrocytic ceruloplasmin expression, which is induced by IL-1beta and by traumatic brain injury, increases in the absence of the IL-1 type 1 receptor Astrocytic ceruloplasmin expression, which is induced by IL-1beta and by traumatic brain injury, increases in the absence of the IL-1 type 1 receptor
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Astrocytic ceruloplasmin expression, which is induced by IL-1beta and by traumatic brain injury, increases in the absence of the IL-1 type 1 receptor Astrocytic ceruloplasmin expression, which is induced by IL-1beta and by traumatic brain injury, increases in the absence of the IL-1 type 1 receptor
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Astrocytic ceruloplasmin expression, which is induced by IL-1beta and by traumatic brain injury, increases in the absence of the IL-1 type 1 receptor Astrocytic ceruloplasmin expression, which is induced by IL-1beta and by traumatic brain injury, increases in the absence of the IL-1 type 1 receptor
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Astrocytic ceruloplasmin expression, which is induced by IL-1beta and by traumatic brain injury, increases in the absence of the IL-1 type 1 receptor
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