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2005年論文 scientific article published on 22 December 2005 article científic artikull shkencor bài báo khoa học artykuł naukowy tieteellinen artikkeli vědecký článek научни чланак vedecký článok 2005년 논문 artículu científicu espublizáu en 2005 2005年论文 wetenschappelijk artikel 2005年论文 επιστημονικό άρθρο 2005年論文 სამეცნიერო სტატია scientific article published on 22 December 2005 vitenskapelig artikkel научная статья article scientifique artigo científico 2005年论文 мақолаи илмӣ 2005年論文 articolo scientifico artículo científico publicado en 2005 article scientific ২২ ডিসেম্বর ২০০৫-এ প্রকাশিত বৈজ্ঞানিক নিবন্ধ 2005年论文 научни чланак artikulong pang-agham vitskapeleg artikkel 2005年论文 2005年の論文 наукова стаття, опублікована в грудні 2005 vetenskaplig artikel videnskabelig artikel teaduslik artikkel tudományos cikk articol științific wissenschaftlicher Artikel bilimsel makale naučni članak 2005 nî lūn-bûn 2005年论文 2005年論文 บทความทางวิทยาศาสตร์ scientific article published on 22 December 2005 artikel ilmiah научна статия artigo científico 2005年論文 מאמר מדעי artigo científico scienca artikolo مقالة علمية نشرت في 22 ديسمبر 2005
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Vivian M Rumjanek Vivian M Coqueiro
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Cyclosporin A does not protect the disruption of the inner mitochondrial membrane potential induced by potassium ionophores in intact K562 cells. Cyclosporin A does not protect the disruption of the inner mitochondrial membrane potential induced by potassium ionophores in intact K562 cells.
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Cyclosporin A does not protect the disruption of the inner mitochondrial membrane potential induced by potassium ionophores in intact K562 cells. Cyclosporin A does not protect the disruption of the inner mitochondrial membrane potential induced by potassium ionophores in intact K562 cells.
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Cyclosporin A does not protect the disruption of the inner mitochondrial membrane potential induced by potassium ionophores in intact K562 cells. Cyclosporin A does not protect the disruption of the inner mitochondrial membrane potential induced by potassium ionophores in intact K562 cells.
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Cyclosporin A does not protect the disruption of the inner mitochondrial membrane potential induced by potassium ionophores in intact K562 cells
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