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наукова стаття, опублікована в жовтні 2010 2010年論文 videnskabelig artikel naučni članak научна статия artikulong pang-agham 2010年论文 artículu científicu espublizáu en 2010 teaduslik artikkel scientific article published on 18 October 2010 2010年論文 vedecký článok scienca artikolo vědecký článek bilimsel makale научни чланак επιστημονικό άρθρο 2010년 논문 artykuł naukowy 2010年の論文 tieteellinen artikkeli scientific article published on 18 October 2010 2010年論文 מאמר מדעי articol științific บทความทางวิทยาศาสตร์ wetenschappelijk artikel 2010年論文 научная статья artigo científico 2010年论文 wissenschaftlicher Artikel bài báo khoa học ১৮ অক্টোবর ২০১০-এ প্রকাশিত বৈজ্ঞানিক নিবন্ধ article scientific scientific article published on 18 October 2010 مقالة علمية نشرت في 18 أكتوبر 2010 2010年論文 научни чланак artikull shkencor 2010年论文 գիտական հոդված հրատարակված 2010 թվականի հոկտեմբերի 18-ին სამეცნიერო სტატია artículo científico publicado en 2010 article científic articolo scientifico vitenskapelig artikkel vetenskaplig artikel artigo científico мақолаи илмӣ 2010年论文 artikel ilmiah 2010年论文 2010 nî lūn-bûn vitskapeleg artikkel tudományos cikk artigo científico 2010年论文 article scientifique
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Anti-Inflammatory benefits of antibiotic-induced neutrophil apoptosis: tulathromycin induces caspase-3-dependent neutrophil programmed cell death and inhibits NF-kappaB signaling and CXCL8 transcription Anti-Inflammatory benefits of antibiotic-induced neutrophil apoptosis: tulathromycin induces caspase-3-dependent neutrophil programmed cell death and inhibits NF-kappaB signaling and CXCL8 transcription Anti-Inflammatory benefits of antibiotic-induced neutrophil apoptosis: tulathromycin induces caspase-3-dependent neutrophil programmed cell death and inhibits NF-kappaB signaling and CXCL8 transcription
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Anti-Inflammatory benefits of antibiotic-induced neutrophil apoptosis: tulathromycin induces caspase-3-dependent neutrophil programmed cell death and inhibits NF-kappaB signaling and CXCL8 transcription Anti-Inflammatory benefits of antibiotic-induced neutrophil apoptosis: tulathromycin induces caspase-3-dependent neutrophil programmed cell death and inhibits NF-kappaB signaling and CXCL8 transcription Anti-Inflammatory benefits of antibiotic-induced neutrophil apoptosis: tulathromycin induces caspase-3-dependent neutrophil programmed cell death and inhibits NF-kappaB signaling and CXCL8 transcription
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