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2016年论文 2016年論文 article scientifique naučni članak научни чланак 2016年论文 scientific article published on 25 October 2016 наукова стаття, опублікована в жовтні 2016 artikulong pang-agham 2016年论文 wetenschappelijk artikel article scientific tieteellinen artikkeli 2016年論文 επιστημονικό άρθρο ২৫ অক্টোবর ২০১৬-এ প্রকাশিত বৈজ্ঞানিক নিবন্ধ научная статья bilimsel makale artigo científico 2016년 논문 სამეცნიერო სტატია 2016年論文 articolo scientifico scienca artikolo 2016 nî lūn-bûn 2016年论文 vitskapeleg artikkel vědecký článek 2016年論文 บทความทางวิทยาศาสตร์ vetenskaplig artikel 2016年论文 научни чланак wissenschaftlicher Artikel videnskabelig artikel artigo científico teaduslik artikkel artikull shkencor מאמר מדעי 2016年论文 vitenskapelig artikkel scientific article published on 25 October 2016 artículu científicu espublizáu en 2016 scientific article published on 25 October 2016 مقالة علمية نشرت في 25 أكتوبر 2016 articol științific artykuł naukowy 2016年論文 bài báo khoa học мақолаи илмӣ artículo científico publicado en 2016 artikel ilmiah научна статия 2016年の論文 vedecký článok artigo científico article científic tudományos cikk

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2016-10-25T00:00:00Z

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Andreas Lamkowski Markus Priller Annette Arndt Alexis Rump Matthias Port Eva Wardelmann Wassiliki Daskalaki Stefan Eder

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Baseline MAPK signaling activity confers intrinsic radioresistance to KRAS-mutant colorectal carcinoma cells by rapid upregulation of heterogeneous nuclear ribonucleoprotein K (hnRNP K). Baseline MAPK signaling activity confers intrinsic radioresistance to KRAS-mutant colorectal carcinoma cells by rapid upregulation of heterogeneous nuclear ribonucleoprotein K (hnRNP K). Baseline MAPK signaling activity confers intrinsic radioresistance to KRAS-mutant colorectal carcinoma cells by rapid upregulation of heterogeneous nuclear ribonucleoprotein K (hnRNP K).

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Baseline MAPK signaling activity confers intrinsic radioresistance to KRAS-mutant colorectal carcinoma cells by rapid upregulation of heterogeneous nuclear ribonucleoprotein K (hnRNP K). Baseline MAPK signaling activity confers intrinsic radioresistance to KRAS-mutant colorectal carcinoma cells by rapid upregulation of heterogeneous nuclear ribonucleoprotein K (hnRNP K). Baseline MAPK signaling activity confers intrinsic radioresistance to KRAS-mutant colorectal carcinoma cells by rapid upregulation of heterogeneous nuclear ribonucleoprotein K (hnRNP K).

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Baseline MAPK signaling activity confers intrinsic radioresistance to KRAS-mutant colorectal carcinoma cells by rapid upregulation of heterogeneous nuclear ribonucleoprotein K (hnRNP K). Baseline MAPK signaling activity confers intrinsic radioresistance to KRAS-mutant colorectal carcinoma cells by rapid upregulation of heterogeneous nuclear ribonucleoprotein K (hnRNP K). Baseline MAPK signaling activity confers intrinsic radioresistance to KRAS-mutant colorectal carcinoma cells by rapid upregulation of heterogeneous nuclear ribonucleoprotein K (hnRNP K).

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Baseline MAPK signaling activity confers intrinsic radioresistance to KRAS-mutant colorectal carcinoma cells by rapid upregulation of heterogeneous nuclear ribonucleoprotein K (hnRNP K).

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n12:J.CANLET.2016.10.027

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10.1016/J.CANLET.2016.10.027