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teaduslik artikkel سائنسی مضمون vetenskaplig artikel บทความทางวิทยาศาสตร์ vitskapeleg artikkel 2016년 논문 научна статия 2016年學術文章 bài báo khoa học 2016年学术文章 artikull shkencor научни чланак article científic wetenschappelijk artikel 2016年學術文章 2016年学术文章 wissenschaftlicher Artikel tudományos cikk مقالة علمية επιστημονικό άρθρο 2016年學術文章 bilimsel makale artigo científico scientific article מאמר מדעי artigo científico (publicado na 2016) სამეცნიერო სტატია գիտական հոդված 2016 nî lūn-bûn 2016年學術文章 tieteellinen artikkeli artículu científicu espublizáu en 2016 мақолаи илмӣ artykuł naukowy artículo científico publicado en 2016 articol științific ২০১৬-এ প্রকাশিত বৈজ্ঞানিক নিবন্ধ artigo científico (publicado na 2016) vedecký článok مقالهٔ علمی научная статья 2016年學術文章 2016年学术文章 articolo scientifico наукова стаття, опублікована в листопаді 2016 article scientific artikulong pang-agham 2016年学术文章 2016年の論文 videnskabelig artikel (udgivet 2016) naučni članak научни чланак scienca artikolo vědecký článek vitenskapelig artikkel mokslinis straipsnis 2016年學術文章 мақолаи илмӣ 2016年学术文章 article scientifique (publié 2016)
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Kang-Ling Wang Huai-Chih Chiang Shih-Jie Chou Yann-Jang Chen Wen-Chung Yu Yung-Yang Liu Hsin-Yang Li Hsin-Bang Leu Yueh Chien Yen-Jen Sung Wei-Chao Chang Ying-Hsiu Lai Chian-Shiu Chien Kuan-Hsuan Chen Kai-Hsi Lu Yuh-Lih Chang
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Interleukin-18 deteriorates Fabry cardiomyopathy and contributes to the development of left ventricular hypertrophy in Fabry patients with GLA IVS4+919 G>A mutation Interleukin-18 deteriorates Fabry cardiomyopathy and contributes to the development of left ventricular hypertrophy in Fabry patients with GLA IVS4+919 G>A mutation Interleukin-18 deteriorates Fabry cardiomyopathy and contributes to the development of left ventricular hypertrophy in Fabry patients with GLA IVS4+919 G>A mutation.
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Interleukin-18 deteriorates Fabry cardiomyopathy and contributes to the development of left ventricular hypertrophy in Fabry patients with GLA IVS4+919 G>A mutation Interleukin-18 deteriorates Fabry cardiomyopathy and contributes to the development of left ventricular hypertrophy in Fabry patients with GLA IVS4+919 G>A mutation. Interleukin-18 deteriorates Fabry cardiomyopathy and contributes to the development of left ventricular hypertrophy in Fabry patients with GLA IVS4+919 G>A mutation
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Interleukin-18 deteriorates Fabry cardiomyopathy and contributes to the development of left ventricular hypertrophy in Fabry patients with GLA IVS4+919 G>A mutation Interleukin-18 deteriorates Fabry cardiomyopathy and contributes to the development of left ventricular hypertrophy in Fabry patients with GLA IVS4+919 G>A mutation Interleukin-18 deteriorates Fabry cardiomyopathy and contributes to the development of left ventricular hypertrophy in Fabry patients with GLA IVS4+919 G>A mutation.
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Interleukin-18 deteriorates Fabry cardiomyopathy and contributes to the development of left ventricular hypertrophy in Fabry patients with GLA IVS4+919 G>A mutation
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