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artigo científico (publicado na 2017) 2017年学术文章 2017 nî lūn-bûn videnskabelig artikel (udgivet 2017) scienca artikolo 2017年学术文章 articolo scientifico 2017年學術文章 artículu científicu espublizáu en 2017 vedecký článok wetenschappelijk artikel artikull shkencor vitenskapelig artikkel სამეცნიერო სტატია bilimsel makale article científic مقالهٔ علمی գիտական հոդված 2017年學術文章 2017年学术文章 scientific article published on February 2017 vitskapeleg artikkel 2017年学术文章 naučni članak научни чланак articol științific 2017年學術文章 мақолаи илмӣ מאמר מדעי 2017年の論文 научни чланак article scientific artykuł naukowy научна статия artículo científico publicado en 2017 tudományos cikk 2017년 논문 artikulong pang-agham bài báo khoa học наукова стаття, опублікована в лютому 2017 سائنسی مضمون 2017年學術文章 2017年學術文章 2017年學術文章 teaduslik artikkel мақолаи илмӣ tieteellinen artikkeli ২০১৭-এ প্রকাশিত বৈজ্ঞানিক নিবন্ধ مقالة علمية نشرت في فبراير 2017 mokslinis straipsnis artigo científico vědecký článek επιστημονικό άρθρο vetenskaplig artikel 2017年学术文章 article scientifique publié en 2017 научная статья wissenschaftlicher Artikel artigo científico (publicado na 2017) บทความทางวิทยาศาสตร์
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HuCNS-SC Human NSCs Fail to Differentiate, Form Ectopic Clusters, and Provide No Cognitive Benefits in a Transgenic Model of Alzheimer's Disease. HuCNS-SC Human NSCs Fail to Differentiate, Form Ectopic Clusters, and Provide No Cognitive Benefits in a Transgenic Model of Alzheimer's Disease. HuCNS-SC Human NSCs Fail to Differentiate, Form Ectopic Clusters, and Provide No Cognitive Benefits in a Transgenic Model of Alzheimer's Disease.
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HuCNS-SC Human NSCs Fail to Differentiate, Form Ectopic Clusters, and Provide No Cognitive Benefits in a Transgenic Model of Alzheimer's Disease. HuCNS-SC Human NSCs Fail to Differentiate, Form Ectopic Clusters, and Provide No Cognitive Benefits in a Transgenic Model of Alzheimer's Disease. HuCNS-SC Human NSCs Fail to Differentiate, Form Ectopic Clusters, and Provide No Cognitive Benefits in a Transgenic Model of Alzheimer's Disease.
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HuCNS-SC Human NSCs Fail to Differentiate, Form Ectopic Clusters, and Provide No Cognitive Benefits in a Transgenic Model of Alzheimer's Disease. HuCNS-SC Human NSCs Fail to Differentiate, Form Ectopic Clusters, and Provide No Cognitive Benefits in a Transgenic Model of Alzheimer's Disease. HuCNS-SC Human NSCs Fail to Differentiate, Form Ectopic Clusters, and Provide No Cognitive Benefits in a Transgenic Model of Alzheimer's Disease.
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