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научни чланак 2016年學術文章 2016年学术文章 vitskapeleg artikkel tieteellinen artikkeli 2016 nî lūn-bûn artigo científico (publicado na 2016) vedecký článok wissenschaftlicher Artikel artigo científico επιστημονικό άρθρο мақолаи илмӣ artículu científicu espublizáu en 2016 artigo científico (publicado na 2016) videnskabelig artikel (udgivet 2016) bilimsel makale 2016年學術文章 articol științific naučni članak ২০১৬-এ প্রকাশিত বৈজ্ঞানিক নিবন্ধ artikull shkencor مقالهٔ علمی 2016年学术文章 scienca artikolo บทความทางวิทยาศาสตร์ سائنسی مضمون научни чланак სამეცნიერო სტატია wetenschappelijk artikel 2016年學術文章 vetenskaplig artikel 2016年學術文章 מאמר מדעי научная статья 2016年学术文章 наукова стаття, опублікована в листопаді 2016 artículo científico publicado en 2016 2016年學術文章 مقالة علمية mokslinis straipsnis 2016年学术文章 գիտական հոդված scientific article teaduslik artikkel artykuł naukowy bài báo khoa học article científic 2016年の論文 article scientifique (publié 2016) tudományos cikk 2016년 논문 article scientific artikulong pang-agham 2016年學術文章 articolo scientifico научна статия 2016年学术文章 мақолаи илмӣ vitenskapelig artikkel vědecký článek
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Sulforaphane Improves Ischemia-Induced Detrusor Overactivity by Downregulating the Enhancement of Associated Endoplasmic Reticulum Stress, Autophagy, and Apoptosis in Rat Bladder. Sulforaphane Improves Ischemia-Induced Detrusor Overactivity by Downregulating the Enhancement of Associated Endoplasmic Reticulum Stress, Autophagy, and Apoptosis in Rat Bladder. Sulforaphane Improves Ischemia-Induced Detrusor Overactivity by Downregulating the Enhancement of Associated Endoplasmic Reticulum Stress, Autophagy, and Apoptosis in Rat Bladder.
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Sulforaphane Improves Ischemia-Induced Detrusor Overactivity by Downregulating the Enhancement of Associated Endoplasmic Reticulum Stress, Autophagy, and Apoptosis in Rat Bladder. Sulforaphane Improves Ischemia-Induced Detrusor Overactivity by Downregulating the Enhancement of Associated Endoplasmic Reticulum Stress, Autophagy, and Apoptosis in Rat Bladder. Sulforaphane Improves Ischemia-Induced Detrusor Overactivity by Downregulating the Enhancement of Associated Endoplasmic Reticulum Stress, Autophagy, and Apoptosis in Rat Bladder.
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