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scientific article published on 27 February 2008 научна статия wissenschaftlicher Artikel 2008年論文 article scientifique artigo científico 2008年論文 2008年论文 научни чланак artículo científico publicado en 2008 artikulong pang-agham 2008年论文 videnskabelig artikel article scientific наукова стаття, опублікована в лютому 2008 artykuł naukowy 2008年論文 scienca artikolo vitenskapelig artikkel vetenskaplig artikel artículu científicu espublizáu en 2008 2008年论文 vedecký článok vitskapeleg artikkel artigo científico scientific article published on 27 February 2008 2008 nî lūn-bûn wetenschappelijk artikel 2008年论文 articolo scientifico teaduslik artikkel bilimsel makale научная статья ২৭ ফেব্রুয়ারি ২০০৮-এ প্রকাশিত বৈজ্ঞানিক নিবন্ধ 2008年論文 2008年论文 artikull shkencor מאמר מדעי articol științific tieteellinen artikkeli мақолаи илмӣ 2008年論文 artigo científico 2008年の論文 2008년 논문 naučni članak επιστημονικό άρθρο บทความทางวิทยาศาสตร์ bài báo khoa học 2008年论文 სამეცნიერო სტატია vědecký článek научни чланак scientific article published on 27 February 2008 tudományos cikk مقالة علمية نشرت في 27 فبراير 2008 article científic artikel ilmiah
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Neuroinflammation mediated by IL-1beta increases susceptibility of dopamine neurons to degeneration in an animal model of Parkinson's disease Neuroinflammation mediated by IL-1beta increases susceptibility of dopamine neurons to degeneration in an animal model of Parkinson's disease Neuroinflammation mediated by IL-1beta increases susceptibility of dopamine neurons to degeneration in an animal model of Parkinson's disease
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Neuroinflammation mediated by IL-1beta increases susceptibility of dopamine neurons to degeneration in an animal model of Parkinson's disease Neuroinflammation mediated by IL-1beta increases susceptibility of dopamine neurons to degeneration in an animal model of Parkinson's disease Neuroinflammation mediated by IL-1beta increases susceptibility of dopamine neurons to degeneration in an animal model of Parkinson's disease
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Neuroinflammation mediated by IL-1beta increases susceptibility of dopamine neurons to degeneration in an animal model of Parkinson's disease Neuroinflammation mediated by IL-1beta increases susceptibility of dopamine neurons to degeneration in an animal model of Parkinson's disease Neuroinflammation mediated by IL-1beta increases susceptibility of dopamine neurons to degeneration in an animal model of Parkinson's disease
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Neuroinflammation mediated by IL-1beta increases susceptibility of dopamine neurons to degeneration in an animal model of Parkinson's disease
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