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artigo científico scientific article published on 26 March 2012 wissenschaftlicher Artikel tieteellinen artikkeli 2012年論文 مقالة علمية نشرت في 26 مارس 2012 artículo científico publicado en 2012 наукова стаття, опублікована в березні 2012 2012年论文 мақолаи илмӣ 2012年论文 мақолаи илмӣ מאמר מדעי artigo científico bài báo khoa học naučni članak 2012年論文 სამეცნიერო სტატია artikel ilmiah mokslinis straipsnis vedecký článok teaduslik artikkel artikull shkencor article científic scienca artikolo 2012年论文 գիտական հոդված научни чланак tudományos cikk 2012年論文 научна статия videnskabelig artikel 2012年の論文 wetenschappelijk artikel articolo scientifico 2012年论文 scientific article published on 26 March 2012 ২০১২-এ প্রকাশিত বৈজ্ঞানিক নিবন্ধ articol științific 2012年論文 научни чланак 2012年论文 artikulong pang-agham vitenskapelig artikkel vetenskaplig artikel artigo científico scientific article published on 26 March 2012 2012 nî lūn-bûn article scientifique vědecký článek article scientific artículu científicu espublizáu en 2012 2012년 논문 vitskapeleg artikkel научная статья บทความทางวิทยาศาสตร์ artykuł naukowy 2012年論文 bilimsel makale επιστημονικό άρθρο 2012年论文
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Targeting Chk1 in p53-deficient triple-negative breast cancer is therapeutically beneficial in human-in-mouse tumor models. Targeting Chk1 in p53-deficient triple-negative breast cancer is therapeutically beneficial in human-in-mouse tumor models. Targeting Chk1 in p53-deficient triple-negative breast cancer is therapeutically beneficial in human-in-mouse tumor models.
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Targeting Chk1 in p53-deficient triple-negative breast cancer is therapeutically beneficial in human-in-mouse tumor models. Targeting Chk1 in p53-deficient triple-negative breast cancer is therapeutically beneficial in human-in-mouse tumor models. Targeting Chk1 in p53-deficient triple-negative breast cancer is therapeutically beneficial in human-in-mouse tumor models.
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Targeting Chk1 in p53-deficient triple-negative breast cancer is therapeutically beneficial in human-in-mouse tumor models. Targeting Chk1 in p53-deficient triple-negative breast cancer is therapeutically beneficial in human-in-mouse tumor models. Targeting Chk1 in p53-deficient triple-negative breast cancer is therapeutically beneficial in human-in-mouse tumor models.
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Targeting Chk1 in p53-deficient triple-negative breast cancer is therapeutically beneficial in human-in-mouse tumor models
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