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1995年論文 1995年論文 1995年论文 articolo scientifico scientific article published on March 1995 artigo científico tudományos cikk bài báo khoa học videnskabelig artikel vitenskapelig artikkel 1995年の論文 мақолаи илмӣ 1995年论文 scientific article published on March 1995 мақолаи илмӣ artikel ilmiah artículo científico publicado en 1995 artigo científico teaduslik artikkel 1995年論文 1995年论文 article científic научна статия מאמר מדעי tieteellinen artikkeli articol științific 1995年论文 наукова стаття, опублікована в березні 1995 মার্চ ১৯৯৫-এ প্রকাশিত বৈজ্ঞানিক নিবন্ধ artikull shkencor scienca artikolo article scientifique artykuł naukowy 1995年論文 wissenschaftlicher Artikel 1995年论文 vedecký článok vetenskaplig artikel article scientific научни чланак wetenschappelijk artikel 1995 թվականի մարտին հրատարակված գիտական հոդված scientific article published on March 1995 artículu científicu espublizáu en 1995 artigo científico 1995年論文 1995년 논문 научни чланак bilimsel makale научная статья 1995 nî lūn-bûn บทความทางวิทยาศาสตร์ 1995年论文 1995 թուականի Մարտին հրատարակուած գիտական յօդուած artikulong pang-agham mokslinis straipsnis naučni članak vitskapeleg artikkel επιστημονικό άρθρο სამეცნიერო სტატია vědecký článek مقالة علمية نشرت في مارس 1995
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A nonsense mutation 1669Glu-->Ter within the regulatory domain of human erythroid ankyrin leads to a selective deficiency of the major ankyrin isoform (band 2.1) and a phenotype of autosomal dominant hereditary spherocytosis A nonsense mutation 1669Glu-->Ter within the regulatory domain of human erythroid ankyrin leads to a selective deficiency of the major ankyrin isoform (band 2.1) and a phenotype of autosomal dominant hereditary spherocytosis A nonsense mutation 1669Glu-->Ter within the regulatory domain of human erythroid ankyrin leads to a selective deficiency of the major ankyrin isoform (band 2.1) and a phenotype of autosomal dominant hereditary spherocytosis
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A nonsense mutation 1669Glu-->Ter within the regulatory domain of human erythroid ankyrin leads to a selective deficiency of the major ankyrin isoform (band 2.1) and a phenotype of autosomal dominant hereditary spherocytosis A nonsense mutation 1669Glu-->Ter within the regulatory domain of human erythroid ankyrin leads to a selective deficiency of the major ankyrin isoform (band 2.1) and a phenotype of autosomal dominant hereditary spherocytosis A nonsense mutation 1669Glu-->Ter within the regulatory domain of human erythroid ankyrin leads to a selective deficiency of the major ankyrin isoform (band 2.1) and a phenotype of autosomal dominant hereditary spherocytosis
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A nonsense mutation 1669Glu-->Ter within the regulatory domain of human erythroid ankyrin leads to a selective deficiency of the major ankyrin isoform (band 2.1) and a phenotype of autosomal dominant hereditary spherocytosis A nonsense mutation 1669Glu-->Ter within the regulatory domain of human erythroid ankyrin leads to a selective deficiency of the major ankyrin isoform (band 2.1) and a phenotype of autosomal dominant hereditary spherocytosis A nonsense mutation 1669Glu-->Ter within the regulatory domain of human erythroid ankyrin leads to a selective deficiency of the major ankyrin isoform (band 2.1) and a phenotype of autosomal dominant hereditary spherocytosis
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A nonsense mutation 1669Glu-->Ter within the regulatory domain of human erythroid ankyrin leads to a selective deficiency of the major ankyrin isoform (band 2.1) and a phenotype of autosomal dominant hereditary spherocytosis
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10.1172/JCI117802
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