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videnskabelig artikel (udgivet 2010) научни чланак 2010 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած научни чланак articolo scientifico tudományos cikk 2010年论文 vitskapeleg artikkel 2010 nî lūn-bûn artigo científico 2010年论文 научная статья مقالة علمية 2010年論文 bài báo khoa học vetenskaplig artikel vitenskapelig artikkel artigo científico (publicado na 2010) מאמר מדעי სამეცნიერო სტატია 2010年论文 article científic vedecký článok ২০১০-এ প্রকাশিত বৈজ্ঞানিক নিবন্ধ articol științific 2010年論文 artikull shkencor tieteellinen artikkeli مقالهٔ علمی naučni članak 2010年论文 vědecký článek artículo científico publicado en 2010 2010年の論文 wetenschappelijk artikel επιστημονικό άρθρο 2010년 논문 2010 թվականի նոյեմբերին հրատարակված գիտական հոդված article scientific teaduslik artikkel บทความทางวิทยาศาสตร์ 2010年論文 мақолаи илмӣ scientific article scienca artikolo artigo científico (publicado na 2010) научна статия سائنسی مضمون 2010年论文 wissenschaftlicher Artikel artykuł naukowy наукова стаття, опублікована в листопаді 2010 2010年論文 artikulong pang-agham article scientifique (publié 2010) mokslinis straipsnis 2010年论文 bilimsel makale artículu científicu espublizáu en 2010 2010年論文 мақолаи илмӣ
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The role of germline AIP, MEN1, PRKAR1A, CDKN1B and CDKN2C mutations in causing pituitary adenomas in a large cohort of children, adolescents, and patients with genetic syndromes. The role of germline AIP, MEN1, PRKAR1A, CDKN1B and CDKN2C mutations in causing pituitary adenomas in a large cohort of children, adolescents, and patients with genetic syndromes. The role of germline AIP, MEN1, PRKAR1A, CDKN1B and CDKN2C mutations in causing pituitary adenomas in a large cohort of children, adolescents, and patients with genetic syndromes.
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The role of germline AIP, MEN1, PRKAR1A, CDKN1B and CDKN2C mutations in causing pituitary adenomas in a large cohort of children, adolescents, and patients with genetic syndromes. The role of germline AIP, MEN1, PRKAR1A, CDKN1B and CDKN2C mutations in causing pituitary adenomas in a large cohort of children, adolescents, and patients with genetic syndromes. The role of germline AIP, MEN1, PRKAR1A, CDKN1B and CDKN2C mutations in causing pituitary adenomas in a large cohort of children, adolescents, and patients with genetic syndromes.
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The role of germline AIP, MEN1, PRKAR1A, CDKN1B and CDKN2C mutations in causing pituitary adenomas in a large cohort of children, adolescents, and patients with genetic syndromes. The role of germline AIP, MEN1, PRKAR1A, CDKN1B and CDKN2C mutations in causing pituitary adenomas in a large cohort of children, adolescents, and patients with genetic syndromes. The role of germline AIP, MEN1, PRKAR1A, CDKN1B and CDKN2C mutations in causing pituitary adenomas in a large cohort of children, adolescents, and patients with genetic syndromes.
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