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2005 թվականի նոյեմբերին հրատարակված գիտական հոդված სამეცნიერო სტატია bài báo khoa học 2005 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած artigo científico (publicado na 2005) 2005年論文 artículu científicu espublizáu en 2005 articol științific مقالة علمية scientific article научна статия наукова стаття, опублікована в листопаді 2005 2005년 논문 videnskabelig artikel (udgivet 2005) bilimsel makale επιστημονικό άρθρο article scientifique (publié 2005) artykuł naukowy 2005年论文 мақолаи илмӣ 2005年論文 мақолаи илмӣ บทความทางวิทยาศาสตร์ artikulong pang-agham מאמר מדעי mokslinis straipsnis vědecký článek 2005 nî lūn-bûn vitskapeleg artikkel 2005年论文 scienca artikolo vedecký článok научни чланак article científic سائنسی مضمون tieteellinen artikkeli 2005年論文 vitenskapelig artikkel 2005年论文 artigo científico (publicado na 2005) ২০০৫-এ প্রকাশিত বৈজ্ঞানিক নিবন্ধ artigo científico научная статья wetenschappelijk artikel article scientific مقالهٔ علمی wissenschaftlicher Artikel 2005年论文 2005年論文 artículo científico publicado en 2005 2005年の論文 articolo scientifico artikull shkencor teaduslik artikkel 2005年论文 vetenskaplig artikel научни чланак naučni članak 2005年论文 2005年論文 tudományos cikk
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Richard Schretzenmair William J Swiggard Ted Theodosopoulos Jihong Dai Clifford Baytop Jianqing J Yu Una O'Doherty Chuanzhao Li
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Human immunodeficiency virus type 1 can establish latent infection in resting CD4+ T cells in the absence of activating stimuli Human immunodeficiency virus type 1 can establish latent infection in resting CD4+ T cells in the absence of activating stimuli Human immunodeficiency virus type 1 can establish latent infection in resting CD4+ T cells in the absence of activating stimuli
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Human immunodeficiency virus type 1 can establish latent infection in resting CD4+ T cells in the absence of activating stimuli Human immunodeficiency virus type 1 can establish latent infection in resting CD4+ T cells in the absence of activating stimuli Human immunodeficiency virus type 1 can establish latent infection in resting CD4+ T cells in the absence of activating stimuli
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Human immunodeficiency virus type 1 can establish latent infection in resting CD4+ T cells in the absence of activating stimuli Human immunodeficiency virus type 1 can establish latent infection in resting CD4+ T cells in the absence of activating stimuli Human immunodeficiency virus type 1 can establish latent infection in resting CD4+ T cells in the absence of activating stimuli
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Human immunodeficiency virus type 1 can establish latent infection in resting CD4+ T cells in the absence of activating stimuli
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