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artículu científicu espublizáu en 2010 научни чланак tieteellinen artikkeli 2010年論文 scientific article 2010年論文 мақолаи илмӣ vědecký článek 2010年论文 2010年の論文 artikulong pang-agham artykuł naukowy artigo científico (publicado na 2010) tudományos cikk наукова стаття, опублікована в травні 2010 vedecký článok teaduslik artikkel 2010年论文 2010年論文 επιστημονικό άρθρο scienca artikolo vetenskaplig artikel article scientifique (publié 2010) artículo científico publicado en 2010 2010년 논문 artigo científico vitenskapelig artikkel bài báo khoa học articolo scientifico naučni članak научная статья мақолаи илмӣ mokslinis straipsnis articol științific مقالة علمية bilimsel makale 2010 nî lūn-bûn artikull shkencor article científic artigo científico (publicado na 2010) vitskapeleg artikkel wissenschaftlicher Artikel 2010年论文 2010 թվականի մայիսին հրատարակված գիտական հոդված מאמר מדעי videnskabelig artikel (udgivet 2010) научни чланак 2010 թուականի Մայիսին հրատարակուած գիտական յօդուած 2010年論文 2010年论文 2010年论文 سائنسی مضمون ২০১০-এ প্রকাশিত বৈজ্ঞানিক নিবন্ধ บทความทางวิทยาศาสตร์ სამეცნიერო სტატია 2010年論文 2010年论文 научна статия wetenschappelijk artikel article scientific مقالهٔ علمی
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Low doses of lipopolysaccharide and minimally oxidized low-density lipoprotein cooperatively activate macrophages via nuclear factor kappa B and activator protein-1: possible mechanism for acceleration of atherosclerosis by subclinical endotoxemia Low doses of lipopolysaccharide and minimally oxidized low-density lipoprotein cooperatively activate macrophages via nuclear factor kappa B and activator protein-1: possible mechanism for acceleration of atherosclerosis by subclinical endotoxemia Low doses of lipopolysaccharide and minimally oxidized low-density lipoprotein cooperatively activate macrophages via nuclear factor kappa B and activator protein-1: possible mechanism for acceleration of atherosclerosis by subclinical endotoxemia
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Low doses of lipopolysaccharide and minimally oxidized low-density lipoprotein cooperatively activate macrophages via nuclear factor kappa B and activator protein-1: possible mechanism for acceleration of atherosclerosis by subclinical endotoxemia Low doses of lipopolysaccharide and minimally oxidized low-density lipoprotein cooperatively activate macrophages via nuclear factor kappa B and activator protein-1: possible mechanism for acceleration of atherosclerosis by subclinical endotoxemia Low doses of lipopolysaccharide and minimally oxidized low-density lipoprotein cooperatively activate macrophages via nuclear factor kappa B and activator protein-1: possible mechanism for acceleration of atherosclerosis by subclinical endotoxemia
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Low doses of lipopolysaccharide and minimally oxidized low-density lipoprotein cooperatively activate macrophages via nuclear factor kappa B and activator protein-1: possible mechanism for acceleration of atherosclerosis by subclinical endotoxemia Low doses of lipopolysaccharide and minimally oxidized low-density lipoprotein cooperatively activate macrophages via nuclear factor kappa B and activator protein-1: possible mechanism for acceleration of atherosclerosis by subclinical endotoxemia Low doses of lipopolysaccharide and minimally oxidized low-density lipoprotein cooperatively activate macrophages via nuclear factor kappa B and activator protein-1: possible mechanism for acceleration of atherosclerosis by subclinical endotoxemia
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Low doses of lipopolysaccharide and minimally oxidized low-density lipoprotein cooperatively activate macrophages via nuclear factor kappa B and activator protein-1: possible mechanism for acceleration of atherosclerosis by subclinical endotoxemia
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