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2000年論文 tieteellinen artikkeli 2000年论文 2000 nî lūn-bûn 2000年论文 artigo científico (publicado na 2000) scientific article published in July 2000 2000年の論文 2000年论文 научна статия artigo científico (publicado na 2000) مقالة علمية نشرت في يوليو 2000 article scientifique (publié 2000) articolo scientifico artikulong pang-agham 2000年論文 2000 թուականի Յուլիսին հրատարակուած գիտական յօդուած مقالهٔ علمی 2000年论文 2000 թվականի հուլիսին հրատարակված գիտական հոդված article scientific artículo científico publicado en 2000 научни чланак bài báo khoa học article científic vitskapeleg artikkel scienca artikolo tudományos cikk мақолаи илмӣ научни чланак სამეცნიერო სტატია мақолаи илмӣ videnskabelig artikel (udgivet 2000) 2000年論文 2000年论文 wetenschappelijk artikel سائنسی مضمون наукова стаття, опублікована в липні 2000 vetenskaplig artikel artikel ilmiah บทความทางวิทยาศาสตร์ artikull shkencor naučni članak artykuł naukowy 2000年論文 2000년 논문 articol științific 2000年論文 vědecký článek 2000年论文 vitenskapelig artikkel научная статья vedecký článok mokslinis straipsnis artículu científicu espublizáu en 2000 επιστημονικό άρθρο scientific article published in July 2000 teaduslik artikkel scientific article published in July 2000 wissenschaftlicher Artikel מאמר מדעי artigo científico ২০০০-এ প্রকাশিত বৈজ্ঞানিক নিবন্ধ bilimsel makale
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Adipose tissue deficiency, glucose intolerance, and increased atherosclerosis result from mutation in the mouse fatty liver dystrophy (fld) gene. Adipose tissue deficiency, glucose intolerance, and increased atherosclerosis result from mutation in the mouse fatty liver dystrophy (fld) gene. Adipose tissue deficiency, glucose intolerance, and increased atherosclerosis result from mutation in the mouse fatty liver dystrophy (fld) gene.
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Adipose tissue deficiency, glucose intolerance, and increased atherosclerosis result from mutation in the mouse fatty liver dystrophy (fld) gene. Adipose tissue deficiency, glucose intolerance, and increased atherosclerosis result from mutation in the mouse fatty liver dystrophy (fld) gene. Adipose tissue deficiency, glucose intolerance, and increased atherosclerosis result from mutation in the mouse fatty liver dystrophy (fld) gene.
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Adipose tissue deficiency, glucose intolerance, and increased atherosclerosis result from mutation in the mouse fatty liver dystrophy (fld) gene. Adipose tissue deficiency, glucose intolerance, and increased atherosclerosis result from mutation in the mouse fatty liver dystrophy (fld) gene. Adipose tissue deficiency, glucose intolerance, and increased atherosclerosis result from mutation in the mouse fatty liver dystrophy (fld) gene.
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Adipose tissue deficiency, glucose intolerance, and increased atherosclerosis result from mutation in the mouse fatty liver dystrophy (fld) gene
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