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1999年論文 სამეცნიერო სტატია vědecký článek 1999년 논문 1999年の論文 1999年论文 מאמר מדעי مقالة علمية mokslinis straipsnis bài báo khoa học مقالهٔ علمی научная статья scienca artikolo articolo scientifico vitskapeleg artikkel artykuł naukowy artikulong pang-agham 1999年論文 vetenskaplig artikel 1999年论文 wissenschaftlicher Artikel article scientific мақолаи илмӣ vitenskapelig artikkel videnskabelig artikel (udgivet 1999) 1999年論文 บทความทางวิทยาศาสตร์ vedecký článok artículo científico publicado en 1999 ১৯৯৯-এ প্রকাশিত বৈজ্ঞানিক নিবন্ধ научни чланак artigo científico (publicado na 1999) 1999年论文 wetenschappelijk artikel 1999 nî lūn-bûn artigo científico (publicado na 1999) سائنسی مضمون tieteellinen artikkeli 1999年論文 article científic naučni članak научна статия наукова стаття, опублікована у квітні 1999 artículu científicu espublizáu en 1999 научни чланак artikull shkencor article scientifique (publié 1999) 1999年论文 bilimsel makale 1999年论文 мақолаи илмӣ 1999年論文 scientific article επιστημονικό άρθρο tudományos cikk 1999年论文 articol științific teaduslik artikkel 1999 թուականի Ապրիլին հրատարակուած գիտական յօդուած artigo científico 1999 թվականի ապրիլին հրատարակված գիտական հոդված
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Modular variations of the human major histocompatibility complex class III genes for serine/threonine kinase RP, complement component C4, steroid 21-hydroxylase CYP21, and tenascin TNX (the RCCX module). A mechanism for gene deletions and disease as Modular variations of the human major histocompatibility complex class III genes for serine/threonine kinase RP, complement component C4, steroid 21-hydroxylase CYP21, and tenascin TNX (the RCCX module). A mechanism for gene deletions and disease as Modular variations of the human major histocompatibility complex class III genes for serine/threonine kinase RP, complement component C4, steroid 21-hydroxylase CYP21, and tenascin TNX (the RCCX module). A mechanism for gene deletions and disease as
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Modular variations of the human major histocompatibility complex class III genes for serine/threonine kinase RP, complement component C4, steroid 21-hydroxylase CYP21, and tenascin TNX (the RCCX module). A mechanism for gene deletions and disease as Modular variations of the human major histocompatibility complex class III genes for serine/threonine kinase RP, complement component C4, steroid 21-hydroxylase CYP21, and tenascin TNX (the RCCX module). A mechanism for gene deletions and disease as Modular variations of the human major histocompatibility complex class III genes for serine/threonine kinase RP, complement component C4, steroid 21-hydroxylase CYP21, and tenascin TNX (the RCCX module). A mechanism for gene deletions and disease as
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Modular variations of the human major histocompatibility complex class III genes for serine/threonine kinase RP, complement component C4, steroid 21-hydroxylase CYP21, and tenascin TNX (the RCCX module). A mechanism for gene deletions and disease as Modular variations of the human major histocompatibility complex class III genes for serine/threonine kinase RP, complement component C4, steroid 21-hydroxylase CYP21, and tenascin TNX (the RCCX module). A mechanism for gene deletions and disease as Modular variations of the human major histocompatibility complex class III genes for serine/threonine kinase RP, complement component C4, steroid 21-hydroxylase CYP21, and tenascin TNX (the RCCX module). A mechanism for gene deletions and disease as
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Modular variations of the human major histocompatibility complex class III genes for serine/threonine kinase RP, complement component C4, steroid 21-hydroxylase CYP21, and tenascin TNX (the RCCX module). A mechanism for gene deletions and disease as
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