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επιστημονικό άρθρο tudományos cikk articolo scientifico научна статия mokslinis straipsnis vědecký článek vitenskapelig artikkel 2010年论文 article scientific scienca artikolo مقالهٔ علمی artículo científico publicado en 2010 2010 nî lūn-bûn artigo científico (publicado na 2010) artikull shkencor videnskabelig artikel (udgivet 2010) tieteellinen artikkeli article scientifique (publié 2010) vitskapeleg artikkel 2010년 논문 2010年論文 artigo científico мақолаи илмӣ scientific article 2010 թուականի Յունուարին հրատարակուած գիտական յօդուած บทความทางวิทยาศาสตร์ 2010年論文 article científic научная статья 2010年論文 ২০১০-এ প্রকাশিত বৈজ্ঞানিক নিবন্ধ სამეცნიერო სტატია 2010年论文 bài báo khoa học artículu científicu espublizáu en 2010 2010年の論文 artykuł naukowy vedecký článok научни чланак teaduslik artikkel artikulong pang-agham 2010年论文 2010年論文 artigo científico (publicado na 2010) سائنسی مضمون 2010 թվականի հունվարին հրատարակված գիտական հոդված naučni članak 2010年论文 مقالة علمية 2010年论文 наукова стаття, опублікована в січні 2010 מאמר מדעי vetenskaplig artikel 2010年論文 articol științific wetenschappelijk artikel bilimsel makale 2010年论文 wissenschaftlicher Artikel мақолаи илмӣ научни чланак
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Loss of function of ATXN1 increases amyloid beta-protein levels by potentiating beta-secretase processing of beta-amyloid precursor protein. Loss of function of ATXN1 increases amyloid beta-protein levels by potentiating beta-secretase processing of beta-amyloid precursor protein. Loss of function of ATXN1 increases amyloid beta-protein levels by potentiating beta-secretase processing of beta-amyloid precursor protein.
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Loss of function of ATXN1 increases amyloid beta-protein levels by potentiating beta-secretase processing of beta-amyloid precursor protein. Loss of function of ATXN1 increases amyloid beta-protein levels by potentiating beta-secretase processing of beta-amyloid precursor protein. Loss of function of ATXN1 increases amyloid beta-protein levels by potentiating beta-secretase processing of beta-amyloid precursor protein.
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Loss of function of ATXN1 increases amyloid beta-protein levels by potentiating beta-secretase processing of beta-amyloid precursor protein. Loss of function of ATXN1 increases amyloid beta-protein levels by potentiating beta-secretase processing of beta-amyloid precursor protein. Loss of function of ATXN1 increases amyloid beta-protein levels by potentiating beta-secretase processing of beta-amyloid precursor protein.
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Loss of function of ATXN1 increases amyloid beta-protein levels by potentiating beta-secretase processing of beta-amyloid precursor protein
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