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scientific journal article 2001 թվականի մայիսին հրատարակված գիտական հոդված 2001 թուականի Մայիսին հրատարակուած գիտական յօդուած مقالة علمية (نشرت في 22-5-2001) article publié dans les Proceedings of the National Academy of Sciences of the United States of America wetenschappelijk artikel (gepubliceerd op 2001/05/22) 2001年學術文章 im Mai 2001 veröffentlichter wissenschaftlicher Artikel vedecký článok (publikovaný 2001/05/22) vědecký článek publikovaný v roce 2001 наукова стаття, опублікована в травні 2001 artículu científicu espublizáu en 2001
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A. R. Kimmel J. T. Tansey C. X. Deng M. L. Reitman C. Li O. Gavrilova J. Gruia-Gray J. V. Zee D. L. Roush C. Sztalryd C. Londos
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Perilipin ablation results in a lean mouse with aberrant adipocyte lipolysis, enhanced leptin production, and resistance to diet-induced obesity Perilipin ablation results in a lean mouse with aberrant adipocyte lipolysis, enhanced leptin production, and resistance to diet-induced obesity Perilipin ablation results in a lean mouse with aberrant adipocyte lipolysis, enhanced leptin production, and resistance to diet-induced obesity
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Perilipin ablation results in a lean mouse with aberrant adipocyte lipolysis, enhanced leptin production, and resistance to diet-induced obesity Perilipin ablation results in a lean mouse with aberrant adipocyte lipolysis, enhanced leptin production, and resistance to diet-induced obesity Perilipin ablation results in a lean mouse with aberrant adipocyte lipolysis, enhanced leptin production, and resistance to diet-induced obesity
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Perilipin ablation results in a lean mouse with aberrant adipocyte lipolysis, enhanced leptin production, and resistance to diet-induced obesity Perilipin ablation results in a lean mouse with aberrant adipocyte lipolysis, enhanced leptin production, and resistance to diet-induced obesity Perilipin ablation results in a lean mouse with aberrant adipocyte lipolysis, enhanced leptin production, and resistance to diet-induced obesity
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Perilipin ablation results in a lean mouse with aberrant adipocyte lipolysis, enhanced leptin production, and resistance to diet-induced obesity
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