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scientific article научни чланак 1995年论文 article scientifique (publié 1995) наукова стаття, опублікована у вересні 1995 1995年論文 bilimsel makale سائنسی مضمون 1995년 논문 1995 nî lūn-bûn 1995 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած article scientific мақолаи илмӣ artikull shkencor bài báo khoa học 1995年论文 1995年論文 artículo científico publicado en 1995 научная статья 1995 թվականի սեպտեմբերին հրատարակված գիտական հոդված artigo científico (publicado na 1995) wetenschappelijk artikel (gepubliceerd in 1995-09) vedecký článok (publikovaný 1995-09) 1995年论文 מאמר מדעי 1995年論文 επιστημονικό άρθρο naučni članak tieteellinen artikkeli teaduslik artikkel artigo científico 1995年论文 scienca artikolo wissenschaftlicher Artikel videnskabelig artikel (udgivet 1995) ১৯৯৫-এ প্রকাশিত বৈজ্ঞানিক নিবন্ধ مقالة علمية (نشرت في سبتمبر 1995) 1995年論文 vitenskapelig artikkel tudományos cikk vědecký článek publikovaný v roce 1995 мақолаи илмӣ مقالهٔ علمی artykuł naukowy 1995年论文 article científic სამეცნიერო სტატია 1995年の論文 научни чланак (објављен 1995-09) mokslinis straipsnis articol științific vitskapeleg artikkel научна статия artigo científico (publicado na 1995) vetenskaplig artikel articolo scientifico artikulong pang-agham บทความทางวิทยาศาสตร์ 1995年论文 1995年論文 artículu científicu espublizáu en 1995

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K R Groom G J Gao C K Kassenbrock M G Douglas N C Martin

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RPM2, independently of its mitochondrial RNase P function, suppresses an ISP42 mutant defective in mitochondrial import and is essential for normal growth RPM2, independently of its mitochondrial RNase P function, suppresses an ISP42 mutant defective in mitochondrial import and is essential for normal growth RPM2, independently of its mitochondrial RNase P function, suppresses an ISP42 mutant defective in mitochondrial import and is essential for normal growth

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RPM2, independently of its mitochondrial RNase P function, suppresses an ISP42 mutant defective in mitochondrial import and is essential for normal growth RPM2, independently of its mitochondrial RNase P function, suppresses an ISP42 mutant defective in mitochondrial import and is essential for normal growth RPM2, independently of its mitochondrial RNase P function, suppresses an ISP42 mutant defective in mitochondrial import and is essential for normal growth

schema:name

RPM2, independently of its mitochondrial RNase P function, suppresses an ISP42 mutant defective in mitochondrial import and is essential for normal growth RPM2, independently of its mitochondrial RNase P function, suppresses an ISP42 mutant defective in mitochondrial import and is essential for normal growth RPM2, independently of its mitochondrial RNase P function, suppresses an ISP42 mutant defective in mitochondrial import and is essential for normal growth

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RPM2, independently of its mitochondrial RNase P function, suppresses an ISP42 mutant defective in mitochondrial import and is essential for normal growth

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