About: Michael D. Taylor

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An Entity of Type : wikibase:Item, within Data Space : wikidata.demo.openlinksw.com associated with source document(s)

Canadian pediatric neurosurgeon

Attributes	Values
rdf:type	Item
field of work	wds:Q88072573-169eebf0-431a-4676-fc8e-92621a44e5ed
field of work	brain cancer
description	ikertzailea (eu) 연구원 (ko) wetenschapper (nl) дослідник (uk) 研究者 (zh) kanadischer Forscher (de) Canadian pediatric neurosurgeon (en)
rdfs:label	Michael D. Taylor (en) M D Taylor (nl) মাইকেল ডি টেলর (bn)
skos:prefLabel	Michael D. Taylor (en) M D Taylor (nl) মাইকেল ডি টেলর (bn)
name	Michael D. Taylor (en) M D Taylor (nl) মাইকেল ডি টেলর (bn)
given name	wds:Q88072573-BB3804F0-3D9B-4631-9346-89AB792D63E3
given name	Michael
image	wds:Q88072573-17c84ef5-4a6f-6801-352f-cc28ea132781
instance of	wds:Q88072573-5C916AE0-6271-4169-B387-D03E8FB3EE98
image
instance of	Homo sapiens
award received	wds:Q88072573-a2ce71dd-4f14-4252-eef1-00b5f268b492 wds:Q88072573-bc056de5-4543-1529-8dcf-970535e985fe
award received	Robert L. Noble Prize Zülch Award
Commons category	wds:Q88072573-8086c187-42fa-41cb-ad1e-676e3dce6eca
Commons category	Michael D. Taylor
skos:altLabel	Michael Taylor (en)
occupation	wds:Q88072573-35135110-428a-f027-d6ce-6da52fef77c7 wds:Q88072573-7F301CDE-6BCD-4901-944C-F85EB3791672
employer	wds:Q88072573-cd00976e-44da-a785-1b06-ae8d3f690f89 wds:Q88072573-cd5b3c6c-4a92-31f0-7a0f-8e15f722faef
sex or gender	wds:Q88072573-AAAC650D-753A-4987-BB7F-4A835044E945
VIAF ID	wds:Q88072573-b2503791-5100-4f6f-9e92-c82db06216b8
GND ID	wds:Q88072573-9ad28124-0887-4ab4-8572-1c9c173a6445
country of citizenship	wds:Q88072573-2bbb02b5-436e-217d-f02f-08c879841fb0
family name	wds:Q88072573-40F6609B-E17B-468F-A9D4-E2DEA3EA5F7F
VIAF ID	http://viaf.org/viaf/8806155769104127880000
GND ID	https://d-nb.info/gnd/1185584242
occupation	pediatric neurosurgeon researcher
employer	University of Toronto Hospital for Sick Children
sex or gender	male
VIAF ID	8806155769104127880000
GND ID	1185584242
country of citizenship	Malawi
family name	Taylor
ORCID iD	wds:Q88072573-E4D0BF25-953E-4B69-96F3-5F146E67D9D5
Dimensions author ID	wds:Q88072573-AEED223C-294C-4320-93DB-FB7B49522BB3
ORCID iD	https://orcid.org/0000-0001-7009-3466
ORCID iD	0000-0001-7009-3466
Dimensions author ID	014000552037.51
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q88072573 Category:Michael D. Taylor
is author of	International network of cancer genome projects Medulloblastoma comprises four distinct molecular variants Molecular Classification of Ependymal Tumors across All CNS Compartments, Histopathological Grades, and Age Groups Rapid, reliable, and reproducible molecular sub-grouping of clinical medulloblastoma samples TERT promoter mutations are highly recurrent in SHH subgroup medulloblastoma Methylation of the TERT promoter and risk stratification of childhood brain tumours: an integrative genomic and molecular study. Genome sequencing of pediatric medulloblastoma links catastrophic DNA rearrangements with TP53 mutations The eEF2 kinase confers resistance to nutrient deprivation by blocking translation elongation Biological and clinical heterogeneity of MYCN-amplified medulloblastoma. Preclinical target validation using patient-derived cells The G protein α subunit Gαs is a tumor suppressor in Sonic hedgehog-driven medulloblastoma New Brain Tumor Entities Emerge from Molecular Classification of CNS-PNETs Role of LIM and SH3 protein 1 (LASP1) in the metastatic dissemination of medulloblastoma Divergent clonal selection dominates medulloblastoma at recurrence Clonal selection drives genetic divergence of metastatic medulloblastoma Aberrant patterns of H3K4 and H3K27 histone lysine methylation occur across subgroups in medulloblastoma. Embryonal tumor with abundant neuropil and true rosettes (ETANTR), ependymoblastoma, and medulloepithelioma share molecular similarity and comprise a single clinicopathological entity Recurrent somatic alterations of FGFR1 and NTRK2 in pilocytic astrocytoma Pediatric and adult sonic hedgehog medulloblastomas are clinically and molecularly distinct Medulloblastoma exome sequencing uncovers subtype-specific somatic mutations Molecular subgroups of medulloblastoma: the current consensus Dissecting the genomic complexity underlying medulloblastoma Subgroup-specific structural variation across 1,000 medulloblastoma genomes Molecular subgroups of medulloblastoma: an international meta-analysis of transcriptome, genetic aberrations, and clinical data of WNT, SHH, Group 3, and Group 4 medulloblastomas An animal model of MYC-driven medulloblastoma Subgroup-specific prognostic implications of TP53 mutation in medulloblastoma Medulloblastomics: the end of the beginning Cytogenetic prognostication within medulloblastoma subgroups Recurrence patterns across medulloblastoma subgroups: an integrated clinical and molecular analysis Pleiotropic role for MYCN in medulloblastoma. Robust molecular subgrouping and copy-number profiling of medulloblastoma from small amounts of archival tumour material using high-density DNA methylation arrays Hotspot mutations in H3F3A and IDH1 define distinct epigenetic and biological subgroups of glioblastoma. TP53 mutation is frequently associated with CTNNB1 mutation or MYCN amplification and is compatible with long-term survival in medulloblastoma. Genome sequencing of SHH medulloblastoma predicts genotype-related response to smoothened inhibition Risk stratification of childhood medulloblastoma in the molecular era: the current consensus Decoding the regulatory landscape of medulloblastoma using DNA methylation sequencing Atypical Teratoid/Rhabdoid Tumors Are Comprised of Three Epigenetic Subgroups with Distinct Enhancer Landscapes. FSTL5 is a marker of poor prognosis in non-WNT/non-SHH medulloblastoma Molecular subgroups of medulloblastoma The clinical implications of medulloblastoma subgroups Somatostatin receptor subtype 2 (sst₂) is a potential prognostic marker and a therapeutic target in medulloblastoma.

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