About: HIV-1 genes vpr and nef synergistically damage podocytes, leading to glomerulosclerosis

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scientific article published on 20 September 2006

Attributes	Values
rdf:type	Item
description	wetenschappelijk artikel (nl) article scientifique publié en 2006 (fr) наукова стаття, опублікована у вересні 2006 (uk) im September 2006 veröffentlichter wissenschaftlicher Artikel (de) scientific article published on 20 September 2006 (en) artículu científicu espublizáu en setiembre de 2006 (ast)
publication date	wds:Q80305257-39C3F3A3-B12C-4CEB-B6CD-E5498C06781A
publication date	2006-09-20 00:00:00Z (xsd:dateTime)
author name string	wds:Q80305257-00AEF1C4-49DF-4D45-94B4-1D23F7FF859B wds:Q80305257-2808E79D-E9D8-4811-90E6-8A7D7F666680 wds:Q80305257-2AF3CC49-7C3E-467A-94CE-4B988BCB0E73 wds:Q80305257-44A5FEC1-550E-4ADB-B238-02EF3F5E39AB wds:Q80305257-4CAE0C41-441D-45A0-B5F5-1313C7DCC051 wds:Q80305257-69152A6F-6DEA-44BF-B4F0-8984C18DC024 wds:Q80305257-78750FDD-ED57-4457-A44B-00A8EDCB8D46 wds:Q80305257-7CD3C6CE-C0FB-4131-A31F-2343DA0E8760
author name string	Ji Ma Agnes B Fogo Yiqin Zuo Zaher Hanna Iekuni Ichikawa Jianyong Zhong Taiji Matsusaka Li-jun Ma
rdfs:label	HIV-1 genes vpr and nef synergistically damage podocytes, leading to glomerulosclerosis (en) HIV-1 genes vpr and nef synergistically damage podocytes, leading to glomerulosclerosis (nl)
skos:prefLabel	HIV-1 genes vpr and nef synergistically damage podocytes, leading to glomerulosclerosis (en) HIV-1 genes vpr and nef synergistically damage podocytes, leading to glomerulosclerosis (nl)
name	HIV-1 genes vpr and nef synergistically damage podocytes, leading to glomerulosclerosis (en) HIV-1 genes vpr and nef synergistically damage podocytes, leading to glomerulosclerosis (nl)
author	wds:Q80305257-93225230-00D9-4204-AA65-2E1E7A892F8D
author	Paul Jolicoeur
title	wds:Q80305257-1B830623-77E0-46B5-B384-1271D9995947
title	HIV-1 genes vpr and nef synergistically damage podocytes, leading to glomerulosclerosis (en)
page(s)	wds:Q80305257-A1F4815B-A162-484D-88DB-B1566CF4880F
page(s)	2832-2843
instance of	wds:Q80305257-F1A706A3-C997-4329-8BA4-9CA4CA1E6F0D
instance of	scholarly article
main subject	wds:Q80305257-BE095136-73A5-491D-AFE2-4B70BB334B28
main subject	glomerulosclerosis
PubMed ID	wds:Q80305257-B2475728-9276-4A97-BB14-E4E241A9E9A8
PubMed ID	http://rdf.ncbi.nlm.nih.gov/pubchem/reference/16988066
PubMed ID	16988066
published in	wds:Q80305257-ECBDE70D-2CDE-4EAB-AFBF-53ED1406F25D
published in	Journal of the American Society of Nephrology
issue	wds:Q80305257-53101AB1-770E-4368-8D94-922F31696728
volume	wds:Q80305257-0E299790-02FB-4A8E-8FA5-033BD5682D96
issue	10
volume	17
DOI	wds:Q80305257-77D227FA-2063-4822-9F74-4EBE74AD02F2
DOI	http://dx.doi.org/10.1681/ASN.2005080878
DOI	10.1681/ASN.2005080878
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q80305257
is cites work of	Advances in the pathogenesis of HIV-associated kidney diseases FAT10: a novel mediator of Vpr-induced apoptosis in human immunodeficiency virus-associated nephropathy HIV-1 viral protein r induces ERK and caspase-8-dependent apoptosis in renal tubular epithelial cells HIV-1 Nef disrupts the podocyte actin cytoskeleton by interacting with diaphanous interacting protein Gene-gene and gene-environment interactions in HIV-associated nephropathy: A focus on the MYH9 nephropathy susceptibility gene Kidney disease in children and adolescents with perinatal HIV-1 infection HIV-1 accessory protein Vpr: relevance in the pathogenesis of HIV and potential for therapeutic intervention HIV-associated nephropathies: epidemiology, pathology, mechanisms and treatment Serious Non-AIDS events: Immunopathogenesis and interventional strategies Recent developments in HIV-related kidney disease Podocin inactivation in mature kidneys causes focal segmental glomerulosclerosis and nephrotic syndrome Transgenic mouse models of mitochondrial toxicity associated with HIV/AIDS and antiretrovirals Inhibition of Notch pathway attenuates the progression of human immunodeficiency virus-associated nephropathy Reversible cell-cycle entry in adult kidney podocytes through regulated control of telomerase and Wnt signaling Podocytes Models of chronic kidney disease Expression of HIV transgene aggravates kidney injury in diabetic mice Accelerated development of collapsing glomerulopathy in mice congenic for the HIVAN1 locus Advances in our understanding of the pathogenesis of HIV-1 associated nephropathy in children APOL1 variants increase risk for FSGS and HIVAN but not IgA nephropathy Role of the retinoic acid receptor-α in HIV-associated nephropathy HIV and chronic kidney disease HIV-1 Vpr activates the DNA damage response in renal tubule epithelial cells Human immunodeficiency virus-1 (HIV-1)-mediated apoptosis: new therapeutic targets Controversies in the pathogenesis of HIV-associated renal diseases HIV-1 and kidney cells: better understanding of viral interaction. HIV-1 upregulates VEGF in podocytes Causes and pathogenesis of focal segmental glomerulosclerosis Nef interaction with actin compromises human podocyte actin cytoskeletal integrity Update on current management of chronic kidney disease in patients with HIV infection DC-specific ICAM-3-grabbing nonintegrin mediates internalization of HIV-1 into human podocytes. Podocyte injury caused by indoxyl sulfate, a uremic toxin and aryl-hydrocarbon receptor ligand Common Mechanisms of Viral Injury to the Kidney Genetic susceptibility to HIV-associated nephropathy Functional genetic variation in aminopeptidase A (ENPEP): lack of clear association with focal and segmental glomerulosclerosis (FSGS). Non-productive HIV-1 infection of human glomerular and urinary podocytes Taking a hard look at the pathogenesis of childhood HIV-associated nephropathy Human immunodeficiency virus downregulates podocyte apoE expression Pathophysiology and treatment of focal segmental glomerulosclerosis: the role of animal models Molecular Mechanisms of Injury in HIV-Associated Nephropathy. Role of Podocyte Injury in Glomerulosclerosis APOL1 Renal Risk Variants: Fertile Soil for HIV-Associated Nephropathy. Nef as a Proliferative Factor for Kidney Epithelial Cells in HIV-Associated Nephropathy ARB protects podocytes from HIV-1 nephropathy independently of podocyte AT1. Viral Subversion Mechanisms in Chronic Kidney Disease Pathogenesis Polyploidy and Mitotic Cell Death Are Two Distinct HIV-1 Vpr-Driven Outcomes in Renal Tubule Epithelial Cells.

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