About: Global gene expression profiles during acute pathogen-induced pulmonary inflammation reveal divergent roles for Th1 and Th2 responses in tissue repair

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About: Global gene expression profiles during acute pathogen-induced pulmonary inflammation reveal divergent roles for Th1 and Th2 responses in tissue repair Goto Sponge NotDistinct Permalink

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scientific article published on 01 October 2003

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rdf:type	Item
description	wetenschappelijk artikel (nl) наукова стаття, опублікована в жовтні 2003 (uk) مقالة علمية تنشرت ف 1 أكتوبر 2003 (ary) im Oktober 2003 veröffentlichter wissenschaftlicher Artikel (de) article scientifique publié en 2003 (fr) artículu científicu espublizáu n'ochobre de 2003 (ast) scientific article published on 01 October 2003 (en)
publication date	wds:Q79074374-8451C3A7-8CF7-46E1-ADAC-1394C41815F7
publication date	2003-10-01 00:00:00Z (xsd:dateTime)
language of work or name	wds:Q79074374-093EEF5E-5643-4305-B44A-94F68F45ED3D
language of work or name	English
author name string	wds:Q79074374-08BBC3DC-5483-47E8-A2E3-4BDCF1AE1BEA wds:Q79074374-B9D2BDEA-0CEC-421C-AF09-D973DAD25F4E wds:Q79074374-F8DB7A3E-5785-4CE9-9EC7-2C061F1FB4DD
author name string	Allen W Cheever Margaret M Mentink-Kane Netanya G Sandler
rdfs:label	Global gene expression profiles during acute pathogen-induced pulmonary inflammation reveal divergent roles for Th1 and Th2 responses in tissue repair (en) Global gene expression profiles during acute pathogen-induced pulmonary inflammation reveal divergent roles for Th1 and Th2 responses in tissue repair (nl)
skos:prefLabel	Global gene expression profiles during acute pathogen-induced pulmonary inflammation reveal divergent roles for Th1 and Th2 responses in tissue repair (en) Global gene expression profiles during acute pathogen-induced pulmonary inflammation reveal divergent roles for Th1 and Th2 responses in tissue repair (nl)
name	Global gene expression profiles during acute pathogen-induced pulmonary inflammation reveal divergent roles for Th1 and Th2 responses in tissue repair (en) Global gene expression profiles during acute pathogen-induced pulmonary inflammation reveal divergent roles for Th1 and Th2 responses in tissue repair (nl)
author	wds:Q79074374-05FD5598-02E0-4192-906C-C2B44453B2B5
author	Thomas A. Wynn
title	wds:Q79074374-B11144B5-86E5-4878-89CD-6F7CF0454AD6
title	Global gene expression profiles during acute pathogen-induced pulmonary inflammation reveal divergent roles for Th1 and Th2 responses in tissue repair (en)
page(s)	wds:Q79074374-A83974CB-F1E8-4DA0-8AD9-718958FE0A02
page(s)	3655-3667
instance of	wds:Q79074374-3DF0047B-E252-4E67-A05C-BB488D0A6E81
instance of	scholarly article
PubMed ID	wds:Q79074374-FD0D0BA4-0E14-4FD3-AEE0-17FF7F435B9E
PubMed ID	http://rdf.ncbi.nlm.nih.gov/pubchem/reference/14500663
PubMed ID	14500663
published in	wds:Q79074374-AC775E00-DA30-4E97-8161-03984C226396
published in	Journal of Immunology
issue	wds:Q79074374-118D0C92-BFF8-4374-BDC1-087EFD400558
volume	wds:Q79074374-63115FC5-BA67-46C0-BFAD-7E286F8180B2
issue	7
volume	171
DOI	wds:Q79074374-48488BFD-A58B-4776-BAA2-C3697DA0F7F5
DOI	http://dx.doi.org/10.4049/JIMMUNOL.171.7.3655
DOI	10.4049/JIMMUNOL.171.7.3655
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q79074374
is cites work of	Allergic asthma: influence of genetic and environmental factors The quiescin sulfhydryl oxidase (hQSOX1b) tunes the expression of resistin-like molecule alpha (RELM-α or mFIZZ1) in a wheat germ cell-free extract Novel effector molecules in type 2 inflammation: lessons drawn from helminth infection and allergy New concepts of IL-10-induced lung fibrosis: fibrocyte recruitment and M2 activation in a CCL2/CCR2 axis Cellular and molecular mechanisms of fibrosis Role of IL-17 and Th17 cells in liver diseases Differential liver protein expression during schistosomiasis Innate immune responses to lung-stage helminth infection induce alternatively activated alveolar macrophages Chitinase and Fizz family members are a generalized feature of nematode infection with selective upregulation of Ym1 and Fizz1 by antigen-presenting cells The IL-21 receptor augments Th2 effector function and alternative macrophage activation Alternatively activated macrophages in helminth infections Identification of novel genes in intestinal tissue that are regulated after infection with an intestinal nematode parasite Regulatory T cells: a role in the control of helminth-driven intestinal pathology and worm survival Arginase-1-expressing macrophages are dispensable for resistance to infection with the gastrointestinal helminth Trichuris muris. Identification and distribution of developing innate lymphoid cells in the fetal mouse intestine Small proline-rich proteins (SPRR) function as SH3 domain ligands, increase resistance to injury and are associated with epithelial-mesenchymal transition (EMT) in cholangiocytes Macrophages: master regulators of inflammation and fibrosis Fibrotic disease and the T(H)1/T(H)2 paradigm Obstacles and opportunities for understanding macrophage polarization Arginase and arginine dysregulation in asthma Alternative activation is an innate response to injury that requires CD4+ T cells to be sustained during chronic infection Protective immune mechanisms in helminth infection Thioredoxin peroxidase secreted by Fasciola hepatica induces the alternative activation of macrophages Transcriptional profiling of the bladder in urogenital schistosomiasis reveals pathways of inflammatory fibrosis and urothelial compromise Alternatively activated macrophage-derived RELM-{alpha} is a negative regulator of type 2 inflammation in the lung. Type 2 immunity and wound healing: evolutionary refinement of adaptive immunity by helminths. An essential role for TH2-type responses in limiting acute tissue damage during experimental helminth infection Type 2 cytokines: mechanisms and therapeutic strategies Expression profiles for macrophage alternative activation genes in AD and in mouse models of AD Heterogeneity of microglial activation in the innate immune response in the brain Characteristic pattern of skeletal muscle remodelling in different mouse strains Development of "-omics" research in Schistosoma spp. and -omics-based new diagnostic tools for schistosomiasis Evolving concepts in the pathogenesis of NASH: beyond steatosis and inflammation Macrophages in Tissue Repair, Regeneration, and Fibrosis New weapons in the war on worms: identification of putative mechanisms of immune-mediated expulsion of gastrointestinal nematodes Matrix metalloproteinase Mmp-1a is dispensable for normal growth and fertility in mice and promotes lung cancer progression by modulating inflammatory responses The protease allergen Pen c 13 induces allergic airway inflammation and changes in epithelial barrier integrity and function in a murine model. Matrix metalloproteinase 12-deficiency augments extracellular matrix degrading metalloproteinases and attenuates IL-13-dependent fibrosis Local and systemic gene expression responses of Atlantic salmon (Salmo salar L.) to infection with the salmon louse (Lepeophtheirus salmonis). Diabetes adversely affects macrophages during atherosclerotic plaque regression in mice Arginase strongly impairs neuronal nitric oxide-mediated airway smooth muscle relaxation in allergic asthma Chitins and chitosans as immunoadjuvants and non-allergenic drug carriers Excreted/secreted Schistosoma mansoni venom allergen-like 9 (SmVAL9) modulates host extracellular matrix remodelling gene expression. Temporal expression of chemokines dictates the hepatic inflammatory infiltrate in a murine model of schistosomiasis Retnla (relmalpha/fizz1) suppresses helminth-induced Th2-type immunity Adoptive transfer of IL-4Rα+ macrophages is sufficient to enhance eosinophilic inflammation in a mouse model of allergic lung inflammation Role of CCR5 in IFN-gamma-induced and cigarette smoke-induced emphysema Thrombospondin-1 deficiency causes a shift from fibroproliferative to inflammatory kidney disease and delays onset of renal failure Arginase and pulmonary diseases T cell-specific deletion of the inositol phosphatase SHIP reveals its role in regulating Th1/Th2 and cytotoxic responses Th17 down-regulation is involved in reduced progression of schistosomiasis fibrosis in ICOSL KO mice Fibrosis is regulated by Th2 and Th17 responses and by dynamic interactions between fibroblasts and macrophages Immune interactions in hepatic fibrosis. Genetic ablation of arginase 1 in macrophages and neutrophils enhances clearance of an arthritogenic alphavirus IL-31-IL-31R interactions negatively regulate type 2 inflammation in the lung

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