About: Mikael Dohlsten

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An Entity of Type : wikibase:Item, within Data Space : wikidata.demo.openlinksw.com associated with source document(s)

Swedish researcher

Attributes	Values
rdf:type	Item
description	연구원 (ko) araştırmacı (tr) эрдэмтэн (mn) изилдөөчү (ky) ikertzaile suediarra (eu) investigador sueco (es) باحث سويدي (ar) svensk forsker (nb) Zweeds onderzoeker (nl) chercheur suédois (fr) investigador suec (ca) investigador sueco (gl) investigador sueco (pt) investigador suecu (ast) Swedish researcher (en) ricercatore svedese (it)
rdfs:label	Mikael Dohlsten (da) Mikael Dohlsten (de) Mikael Dohlsten (en) Mikael Dohlsten (es) Mikael Dohlsten (fr) Mikael Dohlsten (nb) Mikael Dohlsten (nl) Mikael Dohlsten (nn) Mikael Dohlsten (sl) Mikael Dohlsten (sv) Mikael Dohlsten (ast)
skos:prefLabel	Mikael Dohlsten (da) Mikael Dohlsten (de) Mikael Dohlsten (en) Mikael Dohlsten (es) Mikael Dohlsten (fr) Mikael Dohlsten (nb) Mikael Dohlsten (nl) Mikael Dohlsten (nn) Mikael Dohlsten (sl) Mikael Dohlsten (sv) Mikael Dohlsten (ast)
name	Mikael Dohlsten (da) Mikael Dohlsten (de) Mikael Dohlsten (en) Mikael Dohlsten (es) Mikael Dohlsten (fr) Mikael Dohlsten (nb) Mikael Dohlsten (nl) Mikael Dohlsten (nn) Mikael Dohlsten (sl) Mikael Dohlsten (sv) Mikael Dohlsten (ast)
given name	wds:Q76859361-1E0E3455-2C0A-4141-8FDA-1EE19572CE92
given name	Mikael
instance of	wds:Q76859361-EA7DB60C-0878-4A91-B066-26FA9A90831D
instance of	Homo sapiens
occupation	wds:Q76859361-e28f4821-4cdf-d66e-1bc5-d080de430818
employer	wds:Q76859361-ffa33529-4eab-3324-89a6-e36ccf1d3f15
sex or gender	wds:Q76859361-d5edbc50-4c79-1ee0-e00a-6c2e8e82b22f
country of citizenship	wds:Q76859361-d522a12f-4159-ca48-a155-026ad4d35f24
occupation	researcher
employer	Pfizer
sex or gender	male
country of citizenship	Sweden
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q76859361
is author of	Characterization of two distinct MHC class II binding sites in the superantigen staphylococcal enterotoxin A. Antibody-targeted superantigen therapy induces tumor-infiltrating lymphocytes, excessive cytokine production, and apoptosis in human colon carcinoma Activation of alloreactive natural killer cells is resistant to cyclosporine IL-2 unresponsiveness in anergic CD4+ T cells is due to defective signaling through the common gamma-chain of the IL-2 receptor Costimulation of human CD4+ T lymphocytes with B7 and lymphocyte function-associated antigen-3 results in distinct cell activation profiles. Monoclonal antibody-superantigen fusion proteins: tumor-specific agents for T-cell-based tumor therapy Therapy of human non-small-cell lung carcinoma using antibody targeting of a modified superantigen Superantigen-based immunotherapy: a phase I trial of PNU-214565, a monoclonal antibody-staphylococcal enterotoxin A recombinant fusion protein, in advanced pancreatic and colorectal cancer Binding of staphylococcal enterotoxin A to HLA-DR on B cell lines Immune response during tumor therapy with antibody-superantigen fusion proteins Repeated treatment with antibody-targeted superantigens strongly inhibits tumor growth Staphylococcal protein A (SpA) does not induce production of interferon-gamma in human mononuclear blood cells Crystal structure of the superantigen staphylococcal enterotoxin type A Long-term survival and complete cures of B16 melanoma-carrying animals after therapy with tumor-targeted IL-2 and SEA Histamine inhibits interleukin 1 production by lipopolysaccharide-stimulated human peripheral blood monocytes Histamine acts directly on human T cells to inhibit interleukin-2 and interferon-gamma production Human naive and memory T-helper cells display distinct adhesion properties to ICAM-1, LFA-3 and B7 molecules. Immunopharmacology of the superantigen staphylococcal enterotoxin A in T-cell receptor V beta 3 transgenic mice T cell activation pathways: B7, LFA-3, and ICAM-1 shape unique T cell profiles. Immunotherapy of human colon cancer by antibody-targeted superantigens Induction of interleukin-1 in human monocytes by the superantigen staphylococcal enterotoxin A requires the participation of T cells. Production of TNF-alpha and TNF-beta by staphylococcal enterotoxin A activated human T cells Vaccination with B16 melanoma cells expressing a secreted form of interleukin-1beta induces tumor growth inhibition and an enhanced immunity against the wild-type B16 tumor. In vivo induction of gamma/delta T cells with highly potent and selective anti-tumor cytotoxicity Stimulation of human naive and memory T helper cells with bacterial superantigen. Naive CD4+45RA+ T cells require a costimulatory signal mediated through the LFA-1/ICAM-1 pathway Superantigens anergize cytokine production but not cytotoxicity in vivo A recombinant C-terminal fragment of staphylococcal enterotoxin A binds to human MHC class II products but does not activate T cells Characterization of individual tumor necrosis factor alpha-and beta-producing cells after polyclonal T cell activation In vivo anergized CD4+ T cells express perturbed AP-1 and NF-kappa B transcription factors Selective induction of p38 mitogen-activated protein kinase activity following A6H co-stimulation in primary human CD4(+) T cells Histamine modulates the production of interferon-gamma and interleukin-2 by mitogen-activated human mononuclear blood cells Staphylococcal enterotoxin-dependent cell-mediated cytotoxicity Monoclonal antibody-targeted superantigens: a different class of anti-tumor agents Human major histocompatibility complex class II-negative colon carcinoma cells present staphylococcal superantigens to cytotoxic T lymphocytes: evidence for a novel enterotoxin receptor The LFA-3 adhesion pathway is differently utilized by superantigen-activated human CD4+ T-cell subsets Superantigen-induced cytokines suppress growth of human colon-carcinoma cells ICAM-1 costimulation induces IL-2 but inhibits IL-10 production in superantigen-activated human CD4+ T cells

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