About: Phenotypic and functional characterization of mice that lack the type I receptor for IL-1

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About: Phenotypic and functional characterization of mice that lack the type I receptor for IL-1 Goto Sponge NotDistinct Permalink

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scientific article published on 01 October 1997

Attributes	Values
rdf:type	Item
description	wetenschappelijk artikel (nl) наукова стаття, опублікована в жовтні 1997 (uk) im Oktober 1997 veröffentlichter wissenschaftlicher Artikel (de) artículu científicu espublizáu n'ochobre de 1997 (ast) scientific article published on 01 October 1997 (en)
publication date	wds:Q73748033-195D53E2-FBFC-463A-9455-EDF0334B78F6
publication date	1997-10-01 00:00:00Z (xsd:dateTime)
language of work or name	wds:Q73748033-FC0426DB-0F1C-4A58-89AC-8DF018B6DB59
language of work or name	English
author name string	wds:Q73748033-320FEB5E-3F76-4510-843B-75150167D485 wds:Q73748033-3B3FBCE2-5C7B-4926-96F2-3198947C6134 wds:Q73748033-52A9BD70-C4EE-4705-84B8-EF25D8D244A3 wds:Q73748033-8889EC1F-6D1F-4B2C-B389-D8CF5202CFED wds:Q73748033-8FD98C77-60E5-4379-9D53-525E2CD27C52 wds:Q73748033-9054E4A4-0CBE-4237-9678-AE08180B4D47 wds:Q73748033-E2139461-0730-4261-95FB-8489A2487F46 wds:Q73748033-E73C1F36-33D4-4C8B-92BC-CF95D47C19B2 wds:Q73748033-EF752489-F0AC-4D41-9A35-E640062FE81E
author name string	Smith JL Livingston DJ Morrissey PJ Maliszewski C Willis CR Peschon JJ Charrier K Stocking KL Glaccum MB
rdfs:label	Phenotypic and functional characterization of mice that lack the type I receptor for IL-1 (en) Phenotypic and functional characterization of mice that lack the type I receptor for IL-1 (nl)
skos:prefLabel	Phenotypic and functional characterization of mice that lack the type I receptor for IL-1 (en) Phenotypic and functional characterization of mice that lack the type I receptor for IL-1 (nl)
name	Phenotypic and functional characterization of mice that lack the type I receptor for IL-1 (en) Phenotypic and functional characterization of mice that lack the type I receptor for IL-1 (nl)
title	wds:Q73748033-6FD9A26C-87AB-4359-92B4-772CB4E63E15
title	Phenotypic and functional characterization of mice that lack the type I receptor for IL-1 (en)
page(s)	wds:Q73748033-5785A0E0-9C61-43C9-9E5C-01917D7CE6C2
page(s)	3364-3371
instance of	wds:Q73748033-BA78B4AD-C77A-4623-B8AC-A199FB519E22
instance of	scholarly article
PubMed ID	wds:Q73748033-7EA6E627-9D51-4C5B-9EA3-D333A9794137
PubMed ID	http://rdf.ncbi.nlm.nih.gov/pubchem/reference/9317135
PubMed ID	9317135
published in	wds:Q73748033-663B8A58-5EA0-4DDC-8FA2-B9AD299E216C
published in	Journal of Immunology
issue	wds:Q73748033-E9728DF4-0E2C-4959-B686-DE6A8C4790DE
volume	wds:Q73748033-0D217EF9-37EA-4A61-A932-0428CCE093C6
issue	7
volume	159
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q73748033
is cites work of	NOD2 contributes to cutaneous defense against Staphylococcus aureus through alpha-toxin-dependent innate immune activation Inflammation triggers emergency granulopoiesis through a density-dependent feedback mechanism Interleukin-1β regulates CXCL8 release and influences disease outcome in response to Streptococcus pneumoniae, defining intercellular cooperation between pulmonary epithelial cells and macrophages Murine P-glycoprotein deficiency alters intestinal injury repair and blunts lipopolysaccharide-induced radioprotection Neutrophil swarms require LTB4 and integrins at sites of cell death in vivo A novel role for the NLRC4 inflammasome in mucosal defenses against the fungal pathogen Candida albicans MUCOSAL IMMUNOLOGY. Individual intestinal symbionts induce a distinct population of RORγ⁺ regulatory T cells. Signaling through the adaptor molecule MyD88 in CD4+ T cells is required to overcome suppression by regulatory T cells The IL-1-dependent sterile inflammatory response has a substantial caspase-1-independent component that requires cathepsin C. Paget disease of bone Tumor progression locus 2 (Map3k8) is critical for host defense against Listeria monocytogenes and IL-1 beta production. TRAF6 deficiency results in osteopetrosis and defective interleukin-1, CD40, and LPS signaling Opposing activities of two novel members of the IL-1 ligand family regulate skin inflammation Inflammasome-independent IL-1β mediates autoinflammatory disease in Pstpip2-deficient mice Reduced inflammation in the tumor microenvironment delays the accumulation of myeloid-derived suppressor cells and limits tumor progression Whole-cell but not acellular pertussis vaccines induce convulsive activity in mice: evidence of a role for toxin-induced interleukin-1beta in a new murine model for analysis of neuronal side effects of vaccination Sustained hippocampal IL-1 beta overexpression mediates chronic neuroinflammation and ameliorates Alzheimer plaque pathology The role of interleukin-1 in neuroinflammation and Alzheimer disease: an evolving perspective Interleukin-1 receptor 1 knockout has no effect on amyloid deposition in Tg2576 mice and does not alter efficacy following Abeta immunotherapy Interleukin-1 receptor null mutant mice show decreased anxiety-like behavior and enhanced fear memory Interleukin-1 receptor type 1-deficient mice fail to develop social stress-associated glucocorticoid resistance in the spleen. Chronic interleukin-1 exposure drives haematopoietic stem cells towards precocious myeloid differentiation at the expense of self-renewal System x(c)- activity and astrocytes are necessary for interleukin-1 beta-mediated hypoxic neuronal injury IL-1 and IL-23 mediate early IL-17A production in pulmonary inflammation leading to late fibrosis Characterization of a conditional interleukin-1 receptor 1 mouse mutant using the Cre/LoxP system IL-1β drives inflammatory responses to propionibacterium acnes in vitro and in vivo. Interleukin (IL)-1 receptor-associated kinase (IRAK) requirement for optimal induction of multiple IL-1 signaling pathways and IL-6 production Arterial inflammation in mice lacking the interleukin 1 receptor antagonist gene NLRP3 inflammasome plays a redundant role with caspase 8 to promote IL-1β-mediated osteomyelitis A systems analysis identifies a feedforward inflammatory circuit leading to lethal influenza infection Lipopolysaccharide-induced osteoclastogenesis from mononuclear precursors: a mechanism for osteolysis in chronic otitis Interleukin-1 and tumor necrosis factor activities partially account for calvarial bone resorption induced by local injection of lipopolysaccharide Fatal Mycobacterium tuberculosis infection despite adaptive immune response in the absence of MyD88. Identification of the cellular sensor that stimulates the inflammatory response to sterile cell death. IL-33 is a crucial amplifier of innate rather than acquired immunity Caspase-11 activation in response to bacterial secretion systems that access the host cytosol. Contribution of interleukin-11 and prostaglandin(s) in lipopolysaccharide-induced bone resorption in vivo. IL-17 suppresses immune effector functions in human papillomavirus-associated epithelial hyperplasia Costimulatory effects of IL-1 on the expansion/differentiation of CD4+CD25+Foxp3+ and CD4+CD25+Foxp3- T cells Hypoxic epithelial necrosis triggers neutrophilic inflammation via IL-1 receptor signaling in cystic fibrosis lung disease. IL-1beta is an essential mediator of the antineurogenic and anhedonic effects of stress Interleukin-1 is responsible for acute lung immunopathology but increases survival of respiratory influenza virus infection Inflammasome-mediated disease animal models reveal roles for innate but not adaptive immunity. Tumor necrosis factor receptor-associated factor 6 (TRAF6) deficiency results in exencephaly and is required for apoptosis within the developing CNS Vaccine-induced protection against 3 systemic mycoses endemic to North America requires Th17 cells in mice. IL-1R type I-dependent hemopoietic stem cell proliferation is necessary for inflammatory granulopoiesis and reactive neutrophilia Stressor-induced increase in microbicidal activity of splenic macrophages is dependent upon peroxynitrite production. Nod/Ripk2 signaling in dendritic cells activates IL-17A-secreting innate lymphoid cells and drives colitis in T-bet-/-.Rag2-/- (TRUC) mice. Interleukin-1 and tumor necrosis factor receptor signaling is not required for bacteria-induced osteoclastogenesis and bone loss but is essential for protecting the host from a mixed anaerobic infection Leptin actions on food intake and body temperature are mediated by IL-1

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