About: Cryopreserved veins in myocardial revascularization: possible mechanism for their increased failure

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About: Cryopreserved veins in myocardial revascularization: possible mechanism for their increased failure Goto Sponge NotDistinct Permalink

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scientific article published on 01 April 1997

Attributes	Values
rdf:type	Item
description	wetenschappelijk artikel (nl) наукова стаття, опублікована у квітні 1997 (uk) im April 1997 veröffentlichter wissenschaftlicher Artikel (de) artículu científicu espublizáu n'abril de 1997 (ast) scientific article published on 01 April 1997 (en)
publication date	wds:Q73283102-C2D9D3E1-370C-4C3D-915E-D10F6E58A6B9
publication date	1997-04-01 00:00:00Z (xsd:dateTime)
language of work or name	wds:Q73283102-39E299F8-A932-45B6-9B41-89F2D8F36699
language of work or name	English
cites work	wds:Q73283102-16E25892-CF8B-40BA-91EE-A8A08550A837 wds:Q73283102-1DCC6910-4638-4253-B634-F44AFF6C7B30 wds:Q73283102-2CE1ED39-3AA7-4917-95F6-DE07280ACF14 wds:Q73283102-31457DC8-2CA4-4BBB-9804-D109B1563919 wds:Q73283102-49CAD439-4EDF-46E9-8D43-C16CDE76C461 wds:Q73283102-53EC16F0-91FB-4C1B-B384-DCB11A455D99 wds:Q73283102-5EBA7DAB-557E-4AD5-AC11-39CF1823ABD8 wds:Q73283102-626DC306-0E1C-41F1-9FC9-EBE7D46C872D wds:Q73283102-8D013D14-8D7F-4408-BBF2-E4D61A6B7CF8 wds:Q73283102-93A9E268-6EF5-4EBD-B80F-3ADDF60902AC wds:Q73283102-A5A10B83-5B4A-46A8-84B8-F35949E6ECDE wds:Q73283102-C2BB7835-7623-45BF-9AF4-84CA04B3C2B1 wds:Q73283102-C5E4ED2A-0A37-4843-9E43-0D3B541706AE wds:Q73283102-D6B1506C-7BBC-4FB1-9D95-4405FE18CFE6 wds:Q73283102-E76EBEC8-A290-4CB1-929F-C9BCD6ED20F6 wds:Q73283102-EC15C7ED-0BA7-4DC5-A86E-F447A8BE1D99 wds:Q73283102-F3C173B0-8B13-4252-8467-64DE1D2FD0B2 wds:Q73283102-F433E64F-9A54-4FB9-B0A4-666E807E8C07 wds:Q73283102-F8721B44-D2E5-48AA-A28F-8C4E31CD6BE1
cites work	Histologic Fate and Endothelial Changes of Distended and Nondistended Vein Grafts Nitric oxide decreases cytokine-induced endothelial activation. Nitric oxide selectively reduces endothelial expression of adhesion molecules and proinflammatory cytokines Morphine and codeine are endogenous components of human cerebrospinal fluid [D-Ala2]deltorphin I binding and pharmacological evidence for a special subtype of delta opioid receptor on human and invertebrate immune cells Nature and pressure dependence of damage induced by distension of human saphenous vein coronary artery bypass grafts. Changes in saphenous veins used as aortocoronary bypass grafts Induction of superoxide anion and nitric oxide production in cultured microglia. Opiate-like substances in an invertebrate, an opiate receptor on invertebrate and human immunocytes, and a role in immunosuppression. Amplification of the inflammatory response: adhesion molecules associated with platelet/white cell responses Constitutive nitric oxide release is impaired after ischemia and reperfusion Presence of the mu3 opiate receptor in endothelial cells. Coupling to nitric oxide production and vasodilation Multiple effects of ryanodine on intracellular free Ca2+ in smooth muscle cells from bovine and porcine coronary artery: modulation of sarcoplasmic reticulum function The immune-neuro-link and the macrophage: postcardiotomy delirium, HIV-associated dementia and psychiatry Cryopreserved allograft veins as alternative coronary artery bypass conduits: early phase results Effects of cryopreservation on the proliferation and anticoagulant activity of human saphenous vein endothelial cells Long-term Performance of 555 Aortic Homografts in the Aortic Position Cryopreserved saphenous vein allografts for below-knee lower extremity revascularization Reactivity of human saphenous veins at arterial perfusion pressures Simultaneous manipulation of the nitric oxide and prostanoid pathways reduces myocardial reperfusion injury.
author name string	wds:Q73283102-7854B66A-AAED-42D0-B287-96D0695710EB wds:Q73283102-AFCEBAB5-3D32-4B64-96CA-A851FD515349 wds:Q73283102-F77A0BA6-A4CD-4A35-B468-58C981D9EDC6 wds:Q73283102-FC1B2672-5B6C-46E9-BCE4-BFE7772D7104
author name string	Y Liu T V Bilfinger H I Magazine A R Hartman
rdfs:label	Cryopreserved veins in myocardial revascularization: possible mechanism for their increased failure (en) Cryopreserved veins in myocardial revascularization: possible mechanism for their increased failure (nl)
skos:prefLabel	Cryopreserved veins in myocardial revascularization: possible mechanism for their increased failure (en) Cryopreserved veins in myocardial revascularization: possible mechanism for their increased failure (nl)
name	Cryopreserved veins in myocardial revascularization: possible mechanism for their increased failure (en) Cryopreserved veins in myocardial revascularization: possible mechanism for their increased failure (nl)
author	wds:Q73283102-8AA2D4E2-BF60-4201-A870-E973FE9D00B5
author	George Stefano
title	wds:Q73283102-963D6A2C-95EE-440A-9C7C-132D8B1919D2
title	Cryopreserved veins in myocardial revascularization: possible mechanism for their increased failure (en)
page(s)	wds:Q73283102-7F64B89C-58DD-4A46-A584-8E73C6538D83
page(s)	1063-1069
instance of	wds:Q73283102-CE054725-3E13-47DF-B2C6-AEBC515394DD
instance of	scholarly article
main subject	wds:Q73283102-98587C42-045F-44F6-B765-8CA7B1631A43
main subject	revascularization
PubMed ID	wds:Q73283102-5CC24169-2356-4009-820F-170D57EF3558
PubMed ID	http://rdf.ncbi.nlm.nih.gov/pubchem/reference/9124906
PubMed ID	9124906
published in	wds:Q73283102-11F688D9-B422-4FFC-8D73-EB868ADB8F19
published in	The Annals of Thoracic Surgery
issue	wds:Q73283102-BE5C4C2F-B7AC-4553-82C4-3FB2EF84D761
volume	wds:Q73283102-2DD73F84-02B5-4A7C-BEAF-F62173EBAC3D
issue	4
volume	63
DOI	wds:Q73283102-6CB22459-981C-4CEE-8F00-998CFB12F6FB
DOI	http://dx.doi.org/10.1016/S0003-4975(97)00167-7
DOI	10.1016/S0003-4975(97)00167-7
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q73283102
is cites work of	Pharmacological evidence for anandamide amidase in human cardiac and vascular tissues Rebound from nitric oxide inhibition triggers enhanced monocyte activation and chemotaxis The presence of endogenous morphine signaling in animals Endogenous morphine levels increase following cardiac surgery as part of the antiinflammatory response? Morphine suppresses complement receptor expression, phagocytosis, and respiratory burst in neutrophils by a nitric oxide and mu(3) opiate receptor-dependent mechanism. Morphine and anandamide coupling to nitric oxide stimulates GnRH and CRF release from rat median eminence: neurovascular regulation. Vitreous cryopreservation maintains the function of vascular grafts Vascular dysfunction associated with type 2 diabetes and Alzheimer's disease: a potential etiological linkage Human aortocoronary grafts and nitric oxide release: relationship to pulsatile pressure Basal nitric oxide limits immune, nervous and cardiovascular excitation: human endothelia express a mu opiate receptor. Autoimmunovascular regulation: morphine and anandamide and ancondamide stimulated nitric oxide release Effect of central and peripheral administration of a nitric oxide synthase inhibitor on morphine hyperthermia in rats The biochemical substrate of nitric oxide signaling is present in primitive non-cognitive organisms. Delta2 opioid receptor subtype on human vascular endothelium uncouples morphine stimulated nitric oxide release Morphine's immunoregulatory actions are not shared by fentanyl.

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