About: Potential mechanisms by which certain foods promote or inhibit the development of spontaneous diabetes in BB rats: dose, timing, early effect on islet area, and switch in infiltrate from Th1 to Th2 cells

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About: Potential mechanisms by which certain foods promote or inhibit the development of spontaneous diabetes in BB rats: dose, timing, early effect on islet area, and switch in infiltrate from Th1 to Th2 cells Goto Sponge NotDistinct Permalink

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scientific article published on 01 April 1997

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rdf:type	Item
description	wetenschappelijk artikel (nl) наукова стаття, опублікована у квітні 1997 (uk) article scientifique publié en 1997 (fr) im April 1997 veröffentlichter wissenschaftlicher Artikel (de) artículu científicu espublizáu n'abril de 1997 (ast) scientific article published on 01 April 1997 (en) գիտական հոդված հրատարակված 1997 թվականի ապրիլի 1-ին (hy)
publication date	wds:Q73167339-52C8E277-65FE-40AB-A451-1AE673345E89
publication date	1997-04-01 00:00:00Z (xsd:dateTime)
language of work or name	wds:Q73167339-5EBAE949-8B45-466C-A2EB-5DAB18D9D72A
language of work or name	English
author name string	wds:Q73167339-222BDC00-8BAF-40CE-B812-F8829480258B wds:Q73167339-37134C16-8CE7-4483-A927-031E30246E33 wds:Q73167339-60E8F87C-D055-4CBF-AF42-DFB6645C93F4 wds:Q73167339-C4A7F2DB-C192-43FE-A7CF-DECFA9E26FAD wds:Q73167339-D14A2821-14D3-42DF-A340-96933A0E6BEA wds:Q73167339-D8454DB9-E90D-40AB-9CF0-E7D7C9420706 wds:Q73167339-EEF5AFC1-BF23-4B92-B428-2AE4C08A8FE1
author name string	F W Scott H W Modler H Kolb P Rowsell R Kleemann H E Cloutier U Wöerz-Pagenstert
rdfs:label	Potential mechanisms by which certain foods promote or inhibit the development of spontaneous diabetes in BB rats: dose, timing, early effect on islet area, and switch in infiltrate from Th1 to Th2 cells (en) Potential mechanisms by which certain foods promote or inhibit the development of spontaneous diabetes in BB rats: dose, timing, early effect on islet area, and switch in infiltrate from Th1 to Th2 cells (nl)
skos:prefLabel	Potential mechanisms by which certain foods promote or inhibit the development of spontaneous diabetes in BB rats: dose, timing, early effect on islet area, and switch in infiltrate from Th1 to Th2 cells (en) Potential mechanisms by which certain foods promote or inhibit the development of spontaneous diabetes in BB rats: dose, timing, early effect on islet area, and switch in infiltrate from Th1 to Th2 cells (nl)
name	Potential mechanisms by which certain foods promote or inhibit the development of spontaneous diabetes in BB rats: dose, timing, early effect on islet area, and switch in infiltrate from Th1 to Th2 cells (en) Potential mechanisms by which certain foods promote or inhibit the development of spontaneous diabetes in BB rats: dose, timing, early effect on islet area, and switch in infiltrate from Th1 to Th2 cells (nl)
title	wds:Q73167339-EF33153A-273F-413D-A8B1-6105D408ED81
title	Potential mechanisms by which certain foods promote or inhibit the development of spontaneous diabetes in BB rats: dose, timing, early effect on islet area, and switch in infiltrate from Th1 to Th2 cells (en)
page(s)	wds:Q73167339-EE57C90C-6729-40A9-B8DE-ADE852F3C991
page(s)	589-598
instance of	wds:Q73167339-D1A3E91D-11A0-4DF1-B14D-2311AEA6BB8C
instance of	scholarly article
PubMed ID	wds:Q73167339-873C5A77-3231-4EA9-BC69-2AE6BB77344E
PubMed ID	http://rdf.ncbi.nlm.nih.gov/pubchem/reference/9075798
PubMed ID	9075798
published in	wds:Q73167339-A66CDEC4-31DF-4FBA-A31C-B210F5D5229A
published in	Diabetes
issue	wds:Q73167339-E93B5236-6A29-49E6-8732-9460A09909C4
volume	wds:Q73167339-4DC3999F-3FCB-49E9-AC5D-FFC3C05603E7
issue	4
volume	46
DOI	wds:Q73167339-0F4473F2-40EA-4833-984F-9C71F56F8D08
DOI	http://dx.doi.org/10.2337/DIAB.46.4.589
DOI	10.2337/DIAB.46.4.589
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q73167339
is cites work of	Tight junctions, intestinal permeability, and autoimmunity: celiac disease and type 1 diabetes paradigms Gut and the induction of immune tolerance in type 1 diabetes Study design of the Trial to Reduce IDDM in the Genetically at Risk (TRIGR). Small intestinal enteropathy in non-obese diabetic mice fed a diet containing wheat Antibiotic treatment partially protects against type 1 diabetes in the Bio-Breeding diabetes-prone rat. Is the gut flora involved in the development of type 1 diabetes? Tubular complexes as a source for islet neogenesis in the pancreas of diabetes-prone BB rats Oral probiotic administration induces interleukin-10 production and prevents spontaneous autoimmune diabetes in the non-obese diabetic mouse Mucosal T-cell immunoregulation varies in early and late inflammatory bowel disease Promotion of autoimmune diabetes by cereal diet in the presence or absence of microbes associated with gut immune activation, regulatory imbalance, and altered cathelicidin antimicrobial Peptide Is the origin of type 1 diabetes in the gut? Dietary gluten and the development of type 1 diabetes Leaky gut and autoimmune diseases Neonatal oral administration of DiaPep277, combined with hydrolysed casein diet, protects against Type 1 diabetes in BB-DP rats. An experimental study Dietary gluten alters the balance of pro-inflammatory and anti-inflammatory cytokines in T cells of BALB/c mice. Where genes meet environment-integrating the role of gut luminal contents, immunity and pancreas in type 1 diabetes Wheat protein-induced proinflammatory T helper 1 bias in mesenteric lymph nodes of young diabetes-prone rats. The diabetes prone BB rat model of IDDM shows duration of breastfeeding to influence Type 1 diabetes development later in life Diet can influence the ability of nicotinamide to prevent diabetes in the non-obese diabetic mouse: a preliminary study Modulation of the diet and gastrointestinal microbiota normalizes systemic inflammation and β-cell chemokine expression associated with autoimmune diabetes susceptibility. Controversial topics series: milk proteins and diabetes Bioactive GLP-1 in gut, receptor expression in pancreas, and insulin response to GLP-1 in diabetes-prone rats. The Role of Gut Microbiota and Environmental Factors in Type 1 Diabetes Pathogenesis
is cites work of	wds:Q44550803-779D09E0-3658-4123-A7DA-A03E23AC6084 wds:Q28249095-9E248C51-67F0-4558-A34D-72FE1671C460 wds:Q52979489-31A835F9-6CF7-422A-BE8A-30203E8008E0 wds:Q81649672-7D9A2B0B-8131-4262-BCF3-C670B9004812 wds:Q28253767-F64D157E-C23B-499B-B963-80266916E811 wds:Q77977867-285E9208-D456-4E7B-AE9D-FF5B73B4C8B4 wds:Q36867412-60D726F4-0D4E-4AF5-B98C-B9DF41A97F36 wds:Q40409987-8E848460-262C-47CB-A297-7C4D21325043 wds:Q48321657-1CBFC459-3074-4503-AAE3-B11C7B450DA6 wds:Q79243393-A8DC28FC-45FE-4797-9B3A-35D3C7B1CFB8 wds:Q33727439-4FA817E4-4D83-4F27-85FA-685AB0E2BA07 wds:Q53235135-B865C1DA-EB2C-43AE-A4BA-A9E312740B89 wds:Q26823584-DA95AEAF-EAE7-4C39-81B1-298633E13FCF wds:Q33363965-F361E0AA-8DCE-483C-8C95-5C9573A5C44C wds:Q47234300-19B3A71D-105A-46A6-B073-4B168C1454B4 wds:Q44807616-9672FDCB-B34E-4566-95AA-B290FD56D1F4 wds:Q37980043-B958EB10-BD52-40EA-B591-B6E03B5C4918 wds:Q81517210-E97C87DC-F9D4-4BB0-9853-149349860DB4 wds:Q79830050-DD409D80-9B05-42C8-9FCD-B50443EDB749 wds:Q33786936-A4CE0112-DADA-432C-89A3-9204331808D5 wds:Q36172835-25E6EFA6-1F70-4A8D-9D87-5995B17E512A wds:Q90345025-34E6CE6C-6030-4453-8D95-B87D7D9D00CA

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