About: Deficient iNOS in inflammatory bowel disease intestinal microvascular endothelial cells results in increased leukocyte adhesion     Goto   Sponge   NotDistinct   Permalink

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scientific article published on 01 November 2000

AttributesValues
rdf:type
description
  • wetenschappelijk artikel (nl)
  • наукова стаття, опублікована в листопаді 2000 (uk)
  • im November 2000 veröffentlichter wissenschaftlicher Artikel (de)
  • artículu científicu espublizáu en payares de 2000 (ast)
  • scientific article published on 01 November 2000 (en)
publication date
publication date
cites work
cites work
author name string
author name string
  • S Fu
  • C P Johnson
  • M T Rivera
  • P J Fisher
  • K S Ramanujam
  • P Rafiee
  • D G Binion
  • G L Telford
  • M F Otterson
rdfs:label
  • Deficient iNOS in inflammatory bowel disease intestinal microvascular endothelial cells results in increased leukocyte adhesion (en)
  • Deficient iNOS in inflammatory bowel disease intestinal microvascular endothelial cells results in increased leukocyte adhesion (nl)
skos:prefLabel
  • Deficient iNOS in inflammatory bowel disease intestinal microvascular endothelial cells results in increased leukocyte adhesion (en)
  • Deficient iNOS in inflammatory bowel disease intestinal microvascular endothelial cells results in increased leukocyte adhesion (nl)
name
  • Deficient iNOS in inflammatory bowel disease intestinal microvascular endothelial cells results in increased leukocyte adhesion (en)
  • Deficient iNOS in inflammatory bowel disease intestinal microvascular endothelial cells results in increased leukocyte adhesion (nl)
author
author
title
title
  • Deficient iNOS in inflammatory bowel disease intestinal microvascular endothelial cells results in increased leukocyte adhesion (en)
page(s)
page(s)
  • 881-888
instance of
instance of
main subject
main subject
PubMed ID
PubMed ID
PubMed ID
  • 11063913
published in
published in
issue
volume
issue
  • 9
volume
  • 29
DOI
DOI
DOI
  • 10.1016/S0891-5849(00)00391-9
is about of
is cites work of
is cites work of
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