Attributes | Values |
---|
rdf:type
| |
description
| - wetenschappelijk artikel (nl)
- bài báo khoa học (vi)
- наукова стаття, опублікована в лютому 1994 (uk)
- im Februar 1994 veröffentlichter wissenschaftlicher Artikel (de)
- artículu científicu espublizáu en febreru de 1994 (ast)
- artikull shkencor i botuar më 01 shkurt 1994 (sq)
- scientific article published on 01 February 1994 (en)
- գիտական հոդված հրատարակված 1994 թվականի փետրվարի 1-ին (hy)
|
publication date
| |
publication date
| |
language of work or name
| |
language of work or name
| |
author name string
| |
author name string
| - Chen P
- Luyten FP
- Reddi AH
- Paralkar V
|
rdfs:label
| - Recombinant bone morphogenetic protein-4, transforming growth factor-beta 1, and activin A enhance the cartilage phenotype of articular chondrocytes in vitro (en)
- Recombinant bone morphogenetic protein-4, transforming growth factor-beta 1, and activin A enhance the cartilage phenotype of articular chondrocytes in vitro (nl)
- Recombinant bone morphogenetic protein-4, transforming growth factor-beta 1, and activin A enhance the cartilage phenotype of articular chondrocytes in vitro (sq)
|
skos:prefLabel
| - Recombinant bone morphogenetic protein-4, transforming growth factor-beta 1, and activin A enhance the cartilage phenotype of articular chondrocytes in vitro (en)
- Recombinant bone morphogenetic protein-4, transforming growth factor-beta 1, and activin A enhance the cartilage phenotype of articular chondrocytes in vitro (nl)
- Recombinant bone morphogenetic protein-4, transforming growth factor-beta 1, and activin A enhance the cartilage phenotype of articular chondrocytes in vitro (sq)
|
name
| - Recombinant bone morphogenetic protein-4, transforming growth factor-beta 1, and activin A enhance the cartilage phenotype of articular chondrocytes in vitro (en)
- Recombinant bone morphogenetic protein-4, transforming growth factor-beta 1, and activin A enhance the cartilage phenotype of articular chondrocytes in vitro (nl)
- Recombinant bone morphogenetic protein-4, transforming growth factor-beta 1, and activin A enhance the cartilage phenotype of articular chondrocytes in vitro (sq)
|
title
| |
title
| - Recombinant bone morphogenetic protein-4, transforming growth factor-beta 1, and activin A enhance the cartilage phenotype of articular chondrocytes in vitro (en)
|
page(s)
| |
page(s)
| |
instance of
| |
instance of
| |
main subject
| |
main subject
| |
PubMed ID
| |
PubMed ID
| |
PubMed ID
| |
published in
| |
published in
| |
issue
| |
volume
| |
issue
| |
volume
| |
DOI
| |
DOI
| |
DOI
| |
is about
of | |
is cites work
of | - Advances in drug delivery for articular cartilage
- Characterization and cloning of a receptor for BMP-2 and BMP-4 from NIH 3T3 cells.
- Roles of bone morphogenetic protein type I receptors and Smad proteins in osteoblast and chondroblast differentiation.
- Functional analysis of activins during mammalian development.
- Primary structure and tissue distribution of FRZB, a novel protein related to Drosophila frizzled, suggest a role in skeletal morphogenesis
- Regulation of osteogenic proteins by chondrocytes
- Early response genes induced in chondrocytes stimulated with the inflammatory cytokine interleukin-1beta
- Distribution and characterization of specific cellular binding proteins for bone morphogenetic protein-2.
- Molecular analysis of expansion, differentiation, and growth factor treatment of human chondrocytes identifies differentiation markers and growth-related genes
- Interleukin-6 reduces cartilage destruction during experimental arthritis. A study in interleukin-6-deficient mice
- Embryonic stem cell-derived chondrogenic differentiation in vitro: activation by BMP-2 and BMP-4.
- The influence of tissue microenvironment on stem cell-based cartilage repair
- Bone morphogenetic protein signaling is required for maintenance of differentiated phenotype, control of proliferation, and hypertrophy in chondrocytes
- Smad3-deficient chondrocytes have enhanced BMP signaling and accelerated differentiation
- Differential effects of osteogenic protein-1 (BMP-7) on gene expression of BMP and GDF family members during differentiation of the mouse MC615 chondrocyte cells
- Mechanical injury and cytokines cause loss of cartilage integrity and upregulate proteins associated with catabolism, immunity, inflammation, and repair
- Characterization of engineered tissue construct mechanical function by magnetic resonance imaging
- Effect of transforming growth factor-beta on proteoglycan synthesis by chondrocytes in relation to differentiation stage and the presence of pericellular matrix
- Altered responsiveness to TGF-β results in reduced Papss2 expression and alterations in the biomechanical properties of mouse articular cartilage
- Retention and activity of BMP-2 in hyaluronic acid-based scaffolds in vitro
- Assessment of TGF-β3 on production of aggrecan by human articular chondrocytes in pellet culture system
- TGFβ/BMP Signaling Pathway in Cartilage Homeostasis
- Local administration of activin promotes fracture healing in the rat fibula fracture model
- Bone morphogenetic protein-6 and parathyroid hormone-related protein coordinately regulate the hypertrophic conversion in mouse clonal chondrogenic EC cells, ATDC5
- Cartilage morphogenetic proteins: role in joint development, homoeostasis, and regeneration
- Effect of osteogenic protein-1 on the development and mineralization of primary cultures of avian growth plate chondrocytes: modulation by retinoic acid
- [Osteoinduction and -reparation].
- TGFβ1 - induced recruitment of human bone mesenchymal stem cells is mediated by the primary cilium in a SMAD3-dependent manner.
- Parathyroid hormone-related peptide inhibits the expression of bone morphogenetic protein-4 mRNA through a cyclic AMP/protein kinase A pathway in mouse clonal chondrogenic EC cells, ATDC5.
- Modulation of equine articular chondrocyte messenger RNA levels following brief exposures to recombinant equine interleukin-1beta
- Analysis of the native murine bone morphogenetic protein serine threonine kinase type I receptor (ALK-3)
- TGF-β family ligands exhibit distinct signalling dynamics that are driven by receptor localisation
|
is cites work
of | |