About: ATP-sensitive K+ channels may control glucose-induced electrical activity in pancreatic B-cells

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About: ATP-sensitive K+ channels may control glucose-induced electrical activity in pancreatic B-cells Goto Sponge NotDistinct Permalink

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scientific article published on 01 October 1988

Attributes	Values
rdf:type	Item
description	wetenschappelijk artikel (nl) наукова стаття, опублікована в жовтні 1988 (uk) im Oktober 1988 veröffentlichter wissenschaftlicher Artikel (de) artículu científicu espublizáu n'ochobre de 1988 (ast) scientific article published on 01 October 1988 (en)
publication date	wds:Q70225986-007503AE-D05C-4D07-93C9-7CB82D4474F9
publication date	1988-10-01 00:00:00Z (xsd:dateTime)
language of work or name	wds:Q70225986-162070BB-81DB-4E9D-8F76-6D1E5E798011
language of work or name	English
cites work	wds:Q70225986-0D291E06-4EDB-4F77-B311-6BAB27E3689C wds:Q70225986-136A5100-D337-419C-AF9E-540612DEE5DA wds:Q70225986-178245C7-006A-4AD8-ACDB-8AC3C124B2FC wds:Q70225986-1B60A527-754B-4D3E-9838-9F38C7342482 wds:Q70225986-4C09A0F4-C57B-4D50-B423-9293B6B48400 wds:Q70225986-5758F763-BC9B-4C20-8A83-A8F051886560 wds:Q70225986-5D62E1A1-D3B2-434B-A1FB-A251F4B497DB wds:Q70225986-6C368357-829D-441E-B0D5-25DA9BC12BC5 wds:Q70225986-72441649-5972-4B43-BF67-BFC3F109BF2B wds:Q70225986-7C86CA85-D422-49EC-8705-A594C69F4F84 wds:Q70225986-804E7079-63B5-4FA3-BC05-FD8D88993D74 wds:Q70225986-8A715D46-927F-4014-87D7-27118F970B4C wds:Q70225986-8E87F92B-AAB6-425C-BC5C-03D836590DB9 wds:Q70225986-94DCD622-CE6E-41E3-87B4-D0D17232D008 wds:Q70225986-A32D244F-E8C3-4542-A32F-344CC602349C wds:Q70225986-A5B54799-DAC9-4339-9C16-AE3E13C5271C wds:Q70225986-ADEDA463-29DB-4ACF-B404-563AD1E528E2 wds:Q70225986-B1DB8217-EDA5-42E6-8B96-879C2EF473FE wds:Q70225986-C60F86FD-941F-4CE2-847D-959D5C990E73 wds:Q70225986-DA4DDBC5-ED74-4EA2-B8E9-4CEBBAFE1A8F wds:Q70225986-DB48CAF1-0FBD-449C-93C5-21F09BFA227C wds:Q70225986-F6241C3C-7374-4D93-83EA-221245520329 wds:Q70225986-F6CC85B0-D574-451E-9EAA-4479748D0358 wds:Q70225986-FB6AA4E7-0E55-4B19-ADA6-29E114BB79D4
cites work	Cyclic changes in potential and resistance of the beta-cell membrane induced by glucose in islets of Langerhans from mouse Glucose-induced electrical activity in pancreatic islet cells Muscarinic control of pancreatic B cell function involves sodium-dependent depolarization and calcium influx Intracellular ATP directly blocks K+ channels in pancreatic B-cells Glucose induces closure of single potassium channels in isolated rat pancreatic beta-cells Regulation of insulin release by calcium Opposite effects of tolbutamide and diazoxide on 86Rb+ fluxes and membrane potential in pancreatic B cells Electrophysiology of the pancreas Opposite effects of tolbutamide and diazoxide on the ATP-dependent K+ channel in mouse pancreatic beta-cells ATP-sensitive K+ channels in pancreatic beta-cells. Spare-channel hypothesis. Significance of ionic fluxes and changes in membrane potential for stimulus-secretion coupling in pancreatic B-cells D-Glucose inhibits potassium efflux from pancreatic islet cells Glucose-induced decrease in Rb+ permeability in pancreatic β cells Tolbutamide stimulation and inhibition of insulin release: studies of the underlying ionic mechanisms in isolated rat islets Regulation of insulin release by ionic and electrical events in B cells The sulphonylurea receptor may be an ATP-sensitive potassium channel. Glucose dependent K+-channels in pancreatic beta-cells are regulated by intracellular ATP. ATP-sensitive inward rectifier and voltage- and calcium-activated K+ channels in cultured pancreatic islet cells Properties of single potassium channels modulated by glucose in rat pancreatic beta-cells Membrane potential of beta-cells in pancreatic islets. Concentration-dependent effects of tolbutamide, meglitinide, glipizide, glibenclamide and diazoxide on ATP-regulated K+ currents in pancreatic B-cells. Interaction of diazoxide, tolbutamide and ATP4- on nucleotide-dependent K+ channels in an insulin-secreting cell line. Opposite effects of intracellular Ca2+ and glucose on K+ permeability of pancreatic islet cells Efflux of 86Rb from rat and mouse pancreatic islets: the role of membrane depolarization
author name string	wds:Q70225986-A2D6C050-2FF7-4632-A10B-5A9A7EB4A9F2
author name string	J C Henquin
rdfs:label	ATP-sensitive K+ channels may control glucose-induced electrical activity in pancreatic B-cells (en) ATP-sensitive K+ channels may control glucose-induced electrical activity in pancreatic B-cells (nl)
skos:prefLabel	ATP-sensitive K+ channels may control glucose-induced electrical activity in pancreatic B-cells (en) ATP-sensitive K+ channels may control glucose-induced electrical activity in pancreatic B-cells (nl)
name	ATP-sensitive K+ channels may control glucose-induced electrical activity in pancreatic B-cells (en) ATP-sensitive K+ channels may control glucose-induced electrical activity in pancreatic B-cells (nl)
title	wds:Q70225986-4E61311F-FB64-4ECA-B240-13AEDCCD2DC8
title	ATP-sensitive K+ channels may control glucose-induced electrical activity in pancreatic B-cells (en)
page(s)	wds:Q70225986-BA21A0D7-2B42-4959-A623-011AC77F7AA6
page(s)	769-775
instance of	wds:Q70225986-43F6C2DC-FC5C-4800-86D0-678333A80E9B
instance of	scholarly article
PubMed ID	wds:Q70225986-9A02FAEC-E7FF-4679-8F85-7F0D74E4B531
PubMed ID	http://rdf.ncbi.nlm.nih.gov/pubchem/reference/3056403
PubMed ID	3056403
published in	wds:Q70225986-3FA5C12C-AA02-4628-B6BB-6E63EB89BD49
published in	Biochemical and Biophysical Research Communications
issue	wds:Q70225986-AFFC70B3-6581-4560-B7C0-3CFE758DE29D
volume	wds:Q70225986-9F80E460-6189-4A2A-B9DE-C458D1009348
issue	2
volume	156
number of pages	wds:Q70225986-8633A313-F28F-481D-8686-AD6A184B3F1A
number of pages	7 (xsd:decimal)
DOI	wds:Q70225986-0A124DC5-3728-4AF4-A2C3-1E86C3135783
DOI	http://dx.doi.org/10.1016/S0006-291X(88)80910-0
DOI	10.1016/S0006-291X(88)80910-0
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q70225986
is cites work of	Specificity of tetraethylammonium and quinine for three K channels in insulin-secreting cells Calcium and glycolysis mediate multiple bursting modes in pancreatic islets. Interaction of glycolysis and mitochondrial respiration in metabolic oscillations of pancreatic islets. Evidence that glucose can control insulin release independently from its action on ATP-sensitive K+ channels in mouse B cells Activation of Ca(2+)-dependent K(+) channels contributes to rhythmic firing of action potentials in mouse pancreatic beta cells Tolbutamide stimulation of pancreatic beta-cells involves both cell recruitment and increase in the individual Ca(2+) response Regulation of insulin secretion: a matter of phase control and amplitude modulation Glucose stimulation of insulin release in the absence of extracellular Ca2+ and in the absence of any increase in intracellular Ca2+ in rat pancreatic islets. Magnitude and modulation of pancreatic beta-cell gap junction electrical conductance in situ Bursting electrical activity in pancreatic ?-cells: evidence that the channel underlying the burst is sensitive to Ca2+ influx through L-type Ca2+ channels Charybdotoxin-sensitive K(Ca) channel is not involved in glucose-induced electrical activity in pancreatic β-cells K(ATP) channels and islet hormone secretion: new insights and controversies Possible links between glucose-induced changes in the energy state of pancreatic B cells and insulin release. Unmasking by decreasing a stable pool of adenine nucleotides in mouse islets

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