About: Mice homozygous for the ablm1 mutation show poor viability and depletion of selected B and T cell populations

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About: Mice homozygous for the ablm1 mutation show poor viability and depletion of selected B and T cell populations Goto Sponge NotDistinct Permalink

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scientific article published on 01 June 1991

Attributes	Values
rdf:type	Item
description	wetenschappelijk artikel (nl) наукова стаття, опублікована в червні 1991 (uk) article scientifique publié en 1991 (fr) im Juni 1991 veröffentlichter wissenschaftlicher Artikel (de) artículu científicu espublizáu en xunu de 1991 (ast) scientific article published on 01 June 1991 (en)
publication date	wds:Q70220517-B3FAF8BF-6D33-41C1-A2F3-3D5141D24A29
publication date	1991-06-01 00:00:00Z (xsd:dateTime)
language of work or name	wds:Q70220517-754BDA87-1000-4E91-B341-54B53857245D
language of work or name	English
cites work	wds:Q70220517-82EBBDBF-4338-4C57-AA60-AB0513D22097 wds:Q70220517-C9E84323-82FA-4E12-9D24-3DF1DFFBCED1
cites work	Targeted disruption of the c-src proto-oncogene leads to osteopetrosis in mice Xenogeneic Monoclonal Antibodies to Mouse Lymphoid Differentiation Antigens*
author name string	wds:Q70220517-10493001-C03A-4ABB-86E2-74697FF2A07F wds:Q70220517-11B0AB22-64AB-4DFF-9DE0-E7ADF42A7EA1 wds:Q70220517-7D37C028-EBA1-43EB-9562-06976D2DFBA3 wds:Q70220517-8EFA8D04-8A1E-41E8-B08A-076B3C002064 wds:Q70220517-A2F2D1B9-05D8-4490-979F-69553FFD86F2 wds:Q70220517-AE21FA7B-A8A2-4B83-B7EF-F0A271E5DFE8 wds:Q70220517-BA4085E9-73C5-4050-BA3D-83A18EFA7811 wds:Q70220517-BB251C01-6FA8-403D-ABBA-416F9AB56711 wds:Q70220517-EDB1F776-965C-424B-AD92-4FDE2F57B7BD
author name string	S P Goff E J Robertson P L Schwartzberg A M Stall J D Hardin K S Bowdish M L Harbison S Boast T Humaran
rdfs:label	Mice homozygous for the ablm1 mutation show poor viability and depletion of selected B and T cell populations (en) Mice homozygous for the ablm1 mutation show poor viability and depletion of selected B and T cell populations (nl)
skos:prefLabel	Mice homozygous for the ablm1 mutation show poor viability and depletion of selected B and T cell populations (en) Mice homozygous for the ablm1 mutation show poor viability and depletion of selected B and T cell populations (nl)
name	Mice homozygous for the ablm1 mutation show poor viability and depletion of selected B and T cell populations (en) Mice homozygous for the ablm1 mutation show poor viability and depletion of selected B and T cell populations (nl)
title	wds:Q70220517-D7FA7CCE-730F-4D4E-BD1C-323A1198711F
title	Mice homozygous for the ablm1 mutation show poor viability and depletion of selected B and T cell populations (en)
page(s)	wds:Q70220517-521807BF-8F9B-4F8C-8AA5-13A4E972768C
page(s)	1165-1175
instance of	wds:Q70220517-0958B1F9-2378-4F3A-996D-EA5D4A222D6B
instance of	scholarly article
main subject	wds:Q70220517-DE27BBDA-07A9-4781-BE80-26E33289959B
main subject	homozygosity
PubMed ID	wds:Q70220517-172BB39D-DFE7-4D00-9DBC-E3085800A469
PubMed ID	http://rdf.ncbi.nlm.nih.gov/pubchem/reference/2065353
PubMed ID	2065353
published in	wds:Q70220517-E9491962-A940-4E9C-884D-3F6BB7F8ED62
published in	Cell
issue	wds:Q70220517-006F8FF2-0978-4FA1-A045-83DFC0F36A5B
volume	wds:Q70220517-10F6CA3D-4327-4FCE-9D37-ECCD472BC6B7
issue	7
volume	65
DOI	wds:Q70220517-64F16F22-4603-41AA-B5ED-4AA094EBAD49
DOI	http://dx.doi.org/10.1016/0092-8674(91)90012-N
DOI	10.1016/0092-8674(91)90012-N
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q70220517
is cites work of	The c-Abl tyrosine kinase is regulated downstream of the B cell antigen receptor and interacts with CD19 Phosphorylation by the c-Abl protein tyrosine kinase inhibits parkin's ubiquitination and protective function Cytoskeletal protein PSTPIP1 directs the PEST-type protein tyrosine phosphatase to the c-Abl kinase to mediate Abl dephosphorylation Essential roles for the Abl and Arg tyrosine kinases in neurulation NF-kappaB activity is constitutively elevated in c-Abl null fibroblasts c-Abl-deficient mice exhibit reduced numbers of peritoneal B-1 cells and defects in BCR-induced B cell activation The cytostatic function of c-Abl is controlled by multiple nuclear localization signals and requires the p53 and Rb tumor suppressor gene products c-Abl is required for development and optimal cell proliferation in the context of p53 deficiency Abl family tyrosine kinases are essential for basement membrane integrity and cortical lamination in the cerebellum The c-Abl tyrosine kinase regulates actin remodeling at the immune synapse c-Abl-mediated tyrosine phosphorylation of the T-bet DNA-binding domain regulates CD4+ T-cell differentiation and allergic lung inflammation Transgenic and gene knockout mice in cancer research MitoInteractome: mitochondrial protein interactome database, and its application in 'aging network' analysis A small molecule Abl kinase inhibitor induces differentiation of Abelson virus-transformed pre-B cell lines Differential requirements for alpha4 integrins during fetal and adult hematopoiesis Formin binding proteins bear WWP/WW domains that bind proline-rich peptides and functionally resemble SH3 domains Regulation of the hTERT telomerase catalytic subunit by the c-Abl tyrosine kinase The kinase-deficient Src acts as a suppressor of the Abl kinase for Cbl phosphorylation From Abl to actin: Abl tyrosine kinase and associated proteins in growth cone motility c-Abl promotes osteoblast expansion by differentially regulating canonical and non-canonical BMP pathways and p16INK4a expression Loss of WAVE-1 causes sensorimotor retardation and reduced learning and memory in mice Tyrosine kinase oncogenes in normal hematopoiesis and hematological disease Critical role for Kit-mediated Src kinase but not PI 3-kinase signaling in pro T and pro B cell development ABL tyrosine kinases: evolution of function, regulation, and specificity Regulation of cell migration and morphogenesis by Abl-family kinases: emerging mechanisms and physiological contexts Multifunctional Abl kinases in health and disease Role of ABL family kinases in cancer: from leukaemia to solid tumours ABL1 regulates spindle orientation in adherent cells and mammalian skin. Enteropathogenic Escherichia coli use redundant tyrosine kinases to form actin pedestals. The capable ABL: what is its biological function? Acute lymphoblastic leukemia and developmental biology: a crucial interrelationship Mice deficient in Abl are osteoporotic and have defects in osteoblast maturation Effects of a selective inhibitor of the Abl tyrosine kinase on the growth of Bcr-Abl positive cells Abl tyrosine kinase in signal transduction and cell-cycle regulation Signal transduction by wild-type and leukemogenic Abl proteins. The amphiphysin-like protein 1 (ALP1) interacts functionally with the cABL tyrosine kinase and may play a role in cytoskeletal regulation Aberrant splicing of a mouse disabled homolog, mdab1, in the scrambler mouse Promotion and inhibition of activation-induced apoptosis in T-cell hybridomas by oncogenes and related signals Normal development and growth of mice carrying a targeted disruption of the alpha 1 retinoic acid receptor gene c-Abl: activation and nuclear targets

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