About: Shear-induced platelet aggregation can be mediated by vWF released from platelets, as well as by exogenous large or unusually large vWF multimers, requires adenosine diphosphate, and is resistant to aspirin

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About: Shear-induced platelet aggregation can be mediated by vWF released from platelets, as well as by exogenous large or unusually large vWF multimers, requires adenosine diphosphate, and is resistant to aspirin Goto Sponge NotDistinct Permalink

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scientific article published on 01 May 1988

Attributes	Values
rdf:type	Item
description	wetenschappelijk artikel (nl) наукова стаття, опублікована в травні 1988 (uk) im Mai 1988 veröffentlichter wissenschaftlicher Artikel (de) artículu científicu espublizáu en mayu de 1988 (ast) scientific article published on 01 May 1988 (en)
publication date	wds:Q68092762-CA2263CC-215A-4DEC-8AF7-066F7B415217
publication date	1988-05-01 00:00:00Z (xsd:dateTime)
language of work or name	wds:Q68092762-A5F70E26-D40C-440B-9B37-6C32AED658A9
language of work or name	English
author name string	wds:Q68092762-1B4DEC0B-CBD6-4192-A030-04029537CAA6 wds:Q68092762-22B80AA5-31BA-4072-AD37-950DA3FD6EAA wds:Q68092762-CEA41F10-0499-4F4F-984F-A6E32120EF78 wds:Q68092762-E92EA5FB-2F46-4B23-BEFC-7553AFACFF35 wds:Q68092762-FAB867FF-5A55-4C7D-873C-94679EA9457A
author name string	Moake JL Hellums JD Turner NA Stathopoulos NA Nolasco L
rdfs:label	Shear-induced platelet aggregation can be mediated by vWF released from platelets, as well as by exogenous large or unusually large vWF multimers, requires adenosine diphosphate, and is resistant to aspirin (en) Shear-induced platelet aggregation can be mediated by vWF released from platelets, as well as by exogenous large or unusually large vWF multimers, requires adenosine diphosphate, and is resistant to aspirin (nl)
skos:prefLabel	Shear-induced platelet aggregation can be mediated by vWF released from platelets, as well as by exogenous large or unusually large vWF multimers, requires adenosine diphosphate, and is resistant to aspirin (en) Shear-induced platelet aggregation can be mediated by vWF released from platelets, as well as by exogenous large or unusually large vWF multimers, requires adenosine diphosphate, and is resistant to aspirin (nl)
name	Shear-induced platelet aggregation can be mediated by vWF released from platelets, as well as by exogenous large or unusually large vWF multimers, requires adenosine diphosphate, and is resistant to aspirin (en) Shear-induced platelet aggregation can be mediated by vWF released from platelets, as well as by exogenous large or unusually large vWF multimers, requires adenosine diphosphate, and is resistant to aspirin (nl)
title	wds:Q68092762-C13A2453-7C9B-4455-BC10-99402901BF24
title	Shear-induced platelet aggregation can be mediated by vWF released from platelets, as well as by exogenous large or unusually large vWF multimers, requires adenosine diphosphate, and is resistant to aspirin (en)
page(s)	wds:Q68092762-AE0DC2BB-6225-467C-8489-7706EEBB3D0A
page(s)	1366-1374
instance of	wds:Q68092762-6529181D-CFC3-4B1B-9B51-2688ACD2FE8B
instance of	scholarly article
main subject	wds:Q68092762-37A0BD74-A616-4B44-809B-4295AD01E76A wds:Q68092762-877D170B-D3D4-4526-843A-60600A7A3700
main subject	aspirin platelet aggregation
PubMed ID	wds:Q68092762-387EB933-754C-4F30-8CE8-F81C945DB482
PubMed ID	http://rdf.ncbi.nlm.nih.gov/pubchem/reference/3258770
PubMed ID	3258770
published in	wds:Q68092762-6B216672-8562-4D0F-89C8-54F569412BEB
published in	Blood
issue	wds:Q68092762-8BF5BB35-902F-4237-A35F-E8F7A369C1B1
volume	wds:Q68092762-BF387F2A-86C0-47DB-ABB9-DD927BBC9782
issue	5
volume	71
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q68092762
is cites work of	Distinct mechanisms of platelet aggregation as a consequence of different shearing flow conditions Multiscale systems biology and physics of thrombosis under flow 1993 Whitaker Lecture: biorheology in thrombosis research In vitro bleeding test with PFA-100-aspects of controlling individual acetylsalicylic acid induced platelet inhibition in patients with cardiovascular disease von Willebrand factor binding to platelet GpIb initiates signals for platelet activation The P2X1 receptor and platelet function Von Willebrand factor-cleaving protease and Upshaw-Schulman syndrome The role of von Willebrand factor and fibrinogen in platelet aggregation under varying shear stress Platelet-endothelial interactions in atherothrombotic disease: therapeutic implications. Nonlinear flow affects hydrodynamic forces and neutrophil adhesion rates in cone-plate viscometers Thrombotic thrombocytopenic purpura: survival by "giving a dam" Aggregation and disaggregation kinetics of human blood platelets: Part II. Shear-induced platelet aggregation Intercellular calcium communication regulates platelet aggregation and thrombus growth Botrocetin/VWF-induced signaling through GPIb-IX-V produces TxA2 in an alphaIIbbeta3- and aggregation-independent manner Platelet adhesive dynamics. Part II: high shear-induced transient aggregation via GPIbalpha-vWF-GPIbalpha bridging Fluid Mechanics of Blood Clot Formation Adenosine diphosphate-induced aggregation of human platelets in flow through tubes. II. Effect of shear rate, donor sex, and ADP concentration Mathematical analysis of mural thrombogenesis. Concentration profiles of platelet-activating agents and effects of viscous shear flow ARF6 inhibition stabilizes the vasculature and enhances survival during endotoxic shock Low shear stress can initiate von Willebrand factor-dependent platelet aggregation in patients with type IIB and platelet-type von Willebrand disease A risk-benefit assessment of aprotinin in cardiac surgical procedures Shear-stress-induced von Willebrand factor binding to platelets causes the activation of tyrosine kinase(s). Bruton tyrosine kinase is essential for botrocetin/VWF-induced signaling and GPIb-dependent thrombus formation in vivo Microfluidics and coagulation biology Antiplatelet drugs. A comparative review Obstacles in haemocompatibility testing Acquired von Willebrand syndrome associated with left ventricular assist device Direct observation of von Willebrand factor elongation and fiber formation on collagen during acute whole blood exposure to pathological flow Decreased platelet function in aortic valve stenosis: high shear platelet activation then inactivation Transport physics and biorheology in the setting of hemostasis and thrombosis Hemodynamic Performance and Thrombogenic Properties of a Superhydrophobic Bileaflet Mechanical Heart Valve. Hemodynamic force triggers rapid NETosis within sterile thrombotic occlusions. Modulation of the von Willebrand factor-dependent platelet adhesion through alternative proteolytic pathways A mechanistic investigation of thrombotic microangiopathy associated with IV abuse of Opana ER. Targeting von Willebrand Factor in Ischaemic Stroke: Focus on Clinical Evidence Current understanding of the pathophysiology of thrombotic thrombocytopenic purpura Aspirin has limited ability to modulate shear-mediated platelet activation associated with elevated shear stress of ventricular assist devices. Three-dimensional numerical simulations of physiological flows in a stented coronary bifurcation Catheter thrombosis and percutaneous coronary intervention: fundamental perspectives on blood, artificial surfaces and antithrombotic drugs Direct demonstration of radiolabeled von Willebrand factor binding to platelet glycoprotein Ib and IIb-IIIa in the presence of shear stress A comparative study of the antithrombotic effect of aurintricarboxylic acid on arterial thrombosis in rats and guinea pigs Quantification of Von Willebrand Factor Cleavage by adamts-13 in Patients Supported by Left Ventricular Assist Devices. von Willebrand factor proteolysis by ADAMTS-13 in patients on left ventricular assist device support. Thrombotic thrombocytopenic purpura associated with the acquired immune deficiency syndrome Von Willebrand factor, paravalvular leak, and a new vista for TAVR. Historical perspective on ADP-induced platelet activation Platelet size distribution measurements as indicators of shear stress-induced platelet aggregation. Aspirin in patients with coronary artery disease: is it simply irresistible? Impaired platelet inhibitory effect of a single dose of acetylsalicylic acid in patients with unstable coronary artery syndrome in comparison with healthy volunteers
is cites work of	wds:Q34125286-B0B23622-99B0-414F-A194-68AA00DBEAAB wds:Q28201162-70E59214-881B-48E0-8919-7CA7D9B57492 wds:Q34917691-FAFB4560-FCE2-4D2D-BC07-B2FBCE6FF080 wds:Q36323179-D51607F2-A9CC-4036-8EDC-329CD438CD28 wds:Q40432300-3489ADF7-AD8A-4B42-A064-645A2DBD4301

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