About: David H. Munn

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An Entity of Type : wikibase:Item, within Data Space : wikidata.demo.openlinksw.com associated with source document(s)

researcher

Attributes	Values
rdf:type	Item
description	Forscher (de) forskare (sv) researcher (en) 연구원 (ko) ricercatore (it) investigador (es) araştırmacı (tr) chercheur (fr) wetenschapper (nl) дослідник (uk) эрдэмтэн (mn) 研究者 (zh)
rdfs:label	David H. Munn (en) Munn DH (es) Munn DH (nl) Munn DH (ast)
skos:prefLabel	David H. Munn (en) Munn DH (es) Munn DH (nl) Munn DH (ast)
name	David H. Munn (en) Munn DH (es) Munn DH (nl) Munn DH (ast)
Scopus author ID	wds:Q67401826-25D1DCA8-9B35-4226-96B0-9316BE6C2D38
Scopus author ID	7005526265
given name	wds:Q67401826-86514A24-1D0C-480A-B1F9-CA15E3CCC718
given name	David
instance of	wds:Q67401826-7A2CB24E-BEC2-4CE0-82AE-074CC60A7522
instance of	Homo sapiens
skos:altLabel	David Munn (en) DH Munn (en) Munn DH (en)
occupation	wds:Q67401826-152846D1-CBA6-4A1A-9E70-4BF0A635CFA4
employer	wds:Q67401826-4C2A153E-29EB-4587-829C-C79740770349
sex or gender	wds:Q67401826-1E524237-444C-46DB-B311-CA6C32B5D330
ISNI	wds:Q67401826-223F7B29-CB5A-4BFF-9FE9-67A88295AAA5
VIAF ID	wds:Q67401826-6887FE5D-92CA-4BC3-9078-FB762F0BB188
IdRef ID	wds:Q67401826-C4B3C4C2-2953-4EF2-AEAC-4EB601D2292A
PLWABN ID	wds:Q67401826-A64DC0DE-3E78-4EB7-A9F0-8203AB0043F0
official website	wds:Q67401826-E15BB492-2F8A-406B-8713-E47F69F0CF3B
VIAF ID	http://viaf.org/viaf/69145304389278571093
IdRef ID	http://www.idref.fr/109253949/id
occupation	researcher
employer	Augusta University
sex or gender	male
ISNI	0000000049249860
VIAF ID	69145304389278571093
IdRef ID	109253949
PLWABN ID	9810537167005606
official website	https://augusta.pure.elsevier.com/en/persons/1d0d8708-1ddd-4f3c-b8ac-f741a306cb37
ORCID iD	wds:Q67401826-983BC8B4-F404-49DF-BB3C-D7DFE25AE559
ORCID iD	https://orcid.org/0000-0002-7711-2858
ORCID iD	0000-0002-7711-2858
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q67401826
is author of	Inhibition of indoleamine 2,3-dioxygenase (IDO) enhances elimination of virus-infected macrophages in an animal model of HIV-1 encephalitis Cytokine regulation of human monocyte differentiation in vitro: the tumor-cytotoxic phenotype induced by macrophage colony-stimulating factor is developmentally regulated by gamma-interferon Indoleamine 2,3-dioxygenase controls conversion of Foxp3+ Tregs to TH17-like cells in tumor-draining lymph nodes. Tumor-Expressed IDO Recruits and Activates MDSCs in a Treg-Dependent Manner Tryptophan catabolism and T cell responses. Expression of indoleamine 2,3-dioxygenase by plasmacytoid dendritic cells in tumor-draining lymph nodes. Indoleamine 2,3-dioxygenase contributes to tumor cell evasion of T cell-mediated rejection. TLR2-dependent activation of β-catenin pathway in dendritic cells induces regulatory responses and attenuates autoimmune inflammation Program death-1 signaling and regulatory T cells collaborate to resist the function of adoptively transferred cytotoxic T lymphocytes in advanced acute myeloid leukemia. Indoleamine 2,3-dioxygenase in lung dendritic cells promotes Th2 responses and allergic inflammation Toxicology and pharmacokinetics of 1-methyl-d-tryptophan: absence of toxicity due to saturating absorption. Deficient tryptophan catabolism along the kynurenine pathway reveals that the epididymis is in a unique tolerogenic state. Host indoleamine 2,3-dioxygenase: contribution to systemic acquired tumor tolerance. Tolerance to apoptotic cells is regulated by indoleamine 2,3-dioxygenase. GVHD-associated, inflammasome-mediated loss of function in adoptively transferred myeloid-derived suppressor cells. Creating immune privilege: active local suppression that benefits friends, but protects foes. Prevention of T cell-driven complement activation and inflammation by tryptophan catabolism during pregnancy. GCN2 kinase in T cells mediates proliferative arrest and anergy induction in response to indoleamine 2,3-dioxygenase. Ligation of B7-1/B7-2 by human CD4+ T cells triggers indoleamine 2,3-dioxygenase activity in dendritic cells. Monocyte-induced downregulation of nitric oxide synthase in cultured aortic endothelial cells Antigen-specific bacterial vaccine combined with anti-PD-L1 rescues dysfunctional endogenous T cells to reject long-established cancer. An inherently bifunctional subset of Foxp3+ T helper cells is controlled by the transcription factor eos. Lineage-specific transcription factors in unexpected places. IDO inhibits a tryptophan sufficiency signal that stimulates mTOR: A novel IDO effector pathway targeted by D-1-methyl-tryptophan Cells expressing indoleamine 2,3-dioxygenase inhibit T cell responses. Activation-induced apoptosis in human macrophages: developmental regulation of a novel cell death pathway by macrophage colony-stimulating factor and interferon gamma. Therapeutic regulatory T-cell adoptive transfer ameliorates established murine chronic GVHD in a CXCR5-dependent manner The countervailing actions of myeloid and plasmacytoid dendritic cells control autoimmune diabetes in the nonobese diabetic mouse. Blockade of programmed death-1 pathway rescues the effector function of tumor-infiltrating T cells and enhances the antitumor efficacy of lentivector immunization. B-lymphoid cells with attributes of dendritic cells regulate T cells via indoleamine 2,3-dioxygenase Blocking IDO activity to enhance anti-tumor immunity. Cell-autonomous control of interferon type I expression by indoleamine 2,3-dioxygenase in regulatory CD19+ dendritic cells. Recombinant human macrophage colony-stimulating factor in nonhuman primates: selective expansion of a CD16+ monocyte subset with phenotypic similarity to primate natural killer cells. Dendritic cells have the option to express IDO-mediated suppression or not Chronic inflammation that facilitates tumor progression creates local immune suppression by inducing indoleamine 2,3 dioxygenase. Indoleamine 2,3-dioxygenase 1 (ido1) is involved in the control of mouse caput epididymis immune environment Cutting edge: induced indoleamine 2,3 dioxygenase expression in dendritic cell subsets suppresses T cell clonal expansion. Epitope-optimized alpha-fetoprotein genetic vaccines prevent carcinogen-induced murine autochthonous hepatocellular carcinoma Immunosuppressive myeloid cells induced by chemotherapy attenuate antitumor CD4+ T-cell responses through the PD-1-PD-L1 axis Generation of cytotoxic T cells against virus-infected human brain macrophages in a murine model of HIV-1 encephalitis. Indoleamine 2,3-dioxygenase-expressing mature human monocyte-derived dendritic cells expand potent autologous regulatory T cells. Activation of the STING adaptor attenuates experimental autoimmune encephalitis. STING Promotes the Growth of Tumors Characterized by Low Antigenicity via IDO Activation Cutting edge: DNA sensing via the STING adaptor in myeloid dendritic cells induces potent tolerogenic responses

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