About: François Paris

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An Entity of Type : wikibase:Item, within Data Space : wikidata.demo.openlinksw.com associated with source document(s)

medical researcher

Attributes	Values
rdf:type	Item
description	ikertzailea (eu) 研究者 (ja) araştırmacı (tr) onderzoeker (nl) medical researcher (en) медичний дослідник (uk)
rdfs:label	François Paris (en) François Paris (es) François Paris (fr) François Paris (it) François Paris (nl) François Paris (ast)
skos:prefLabel	François Paris (en) François Paris (es) François Paris (fr) François Paris (it) François Paris (nl) François Paris (ast)
name	François Paris (en) François Paris (es) François Paris (fr) François Paris (it) François Paris (nl) François Paris (ast)
Scopus author ID	wds:Q64859984-90D7B52B-AE2A-476D-BAAD-F99B9F3683D4
Scopus author ID	35498372700
given name	wds:Q64859984-3A6DF3C0-DA91-406A-A5D1-BF599C4C14DB
given name	François
instance of	wds:Q64859984-451E7574-CDF1-4E49-8F9B-A7DE71369A10
instance of	Homo sapiens
Loop ID	wds:Q64859984-057CA266-0C8F-463D-97AE-34023900AA0B
Loop ID	26765
skos:altLabel	François E. Paris (en)
occupation	wds:Q64859984-6D82554F-3FF8-4C99-9896-0CCC1527524F
employer	wds:Q64859984-2B1D1E74-56C3-495A-A127-4C252F8CB8EA wds:Q64859984-4035BE49-6544-43B8-8414-625A48EBB289 wds:Q64859984-410742DD-03B0-4AFD-A373-348800099B91 wds:Q64859984-85A64912-D688-4E14-953E-C33413137965 wds:Q64859984-8F5FA1B2-B15C-4967-AF03-FA1C9925E7FE wds:Q64859984-C6EDFB0D-C16A-4863-833C-713D11976596 wds:Q64859984-F35FB0AE-E53D-402F-B6DF-293E6B9454B1
sex or gender	wds:Q64859984-F1253BBD-D67F-4FEA-9014-56A7C1CC8C0A
VIAF ID	wds:Q64859984-41E028F7-7B58-4CEF-99D5-B32A9A61F7F1
IdRef ID	wds:Q64859984-044B32CB-38C1-421F-8E19-B0B37B3C9ACE
educated at	wds:Q64859984-31CFAF87-95A0-4C94-B0CA-0F123D04A603 wds:Q64859984-6BD65120-E832-407A-BB7E-D84D36F1DC68
VIAF ID	http://viaf.org/viaf/190753438
IdRef ID	http://www.idref.fr/113355939/id
occupation	researcher
employer	Curie Institute Memorial Sloan Kettering Cancer Center UCB Medical Research Council Harwell French National Institute of Health and Medical Research Laval University CEA Fontenay-Aux-Roses
sex or gender	male
VIAF ID	190753438
IdRef ID	113355939
educated at	University of Nantes Pierre and Marie Curie University
ORCID iD	wds:Q64859984-FB7532C9-3611-42D9-A9A4-1779CBAA169E
Dimensions author ID	wds:Q64859984-4B5DEBAD-4546-4E97-9ADA-B02243C912B5
ORCID iD	https://orcid.org/0000-0002-0176-7348
ORCID iD	0000-0002-0176-7348
Dimensions author ID	01003222672.77
ResearcherID	wds:Q64859984-D78C0661-14D3-43F3-A48C-5513ADFA8E98
Publons author ID	wds:Q64859984-492a84ed-4f81-6e5f-6b7a-3ed4e4d07280
ResearcherID	L-4915-2015
Publons author ID	2362214
is owl:sameAs of	François Paris
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q64859984
is author of	Role of acidic sphingomyelinase in Fas/CD95-mediated cell death. Endothelial apoptosis as the primary lesion initiating intestinal radiation damage in mice. CD95-mediated apoptosis in vivo involves acid sphingomyelinase. Natural ceramide reverses Fas resistance of acid sphingomyelinase(-/-) hepatocytes. Differentiation-related response to DNA breaks in human mesenchymal stem cells Oocyte apoptosis is suppressed by disruption of the acid sphingomyelinase gene or by sphingosine-1-phosphate therapy Chimeric antibody c.8B6 to O-acetyl-GD2 mediates the same efficient anti-neuroblastoma effects as therapeutic ch14.18 antibody to GD2 without antibody induced allodynia S1P lyase regulates DNA damage responses through a novel sphingolipid feedback mechanism. Membrane signaling induced by high doses of ionizing radiation in the endothelial compartment. Relevance in radiation toxicity Sphingosine-1-phosphate protects proliferating endothelial cells from ceramide-induced apoptosis but not from DNA damage-induced mitotic death. Review and meta-analysis of epidemiological associations between low/moderate doses of ionizing radiation and circulatory disease risks, and their possible mechanisms Cell cycle arrest and apoptosis induced by O-acetyl-GD2-specific monoclonal antibody 8B6 inhibits tumor growth in vitro and in vivo. Radiation-induced PGE2 sustains human glioma cells growth and survival through EGF signaling. Intestinal epithelial cell dysfunction is mediated by an endothelial-specific radiation-induced bystander effect. Improved functionality of the vasculature during conventionally fractionated radiation therapy of prostate cancer RhoA GTPase regulates radiation-induced alterations in endothelial cell adhesion and migration. The p38 pathway, a major pleiotropic cascade that transduces stress and metastatic signals in endothelial cells Krüppel-like factor 4: A new potential biomarker of lung cancer Plasma membrane reorganization links acid sphingomyelinase/ceramide to p38 MAPK pathways in endothelial cells apoptosis. Radiosensitization of prostate cancer cells by the dual PI3K/mTOR inhibitor BEZ235 under normoxic and hypoxic conditions. Ionizing radiation induces long-term senescence in endothelial cells through mitochondrial respiratory complex II dysfunction and superoxide generation. Plasma ceramide, a real-time predictive marker of pulmonary and hepatic metastases response to stereotactic body radiation therapy combined with irinotecan. Underestimation of dose delivery in preclinical irradiation due to scattering conditions. Evaluation of tumor hypoxia prior to radiotherapy in intermediate-risk prostate cancer using 18F-fluoromisonidazole PET/CT: a pilot study. ["Dose-painting": myth or reality?] Binding activities and antitumor properties of a new mouse/human chimeric antibody specific for GD2 ganglioside antigen Bioactive lipids and the control of Bax pro-apoptotic activity

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