About: Activation of CaMKIIdeltaC is a common intermediate of diverse death stimuli-induced heart muscle cell apoptosis

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About: Activation of CaMKIIdeltaC is a common intermediate of diverse death stimuli-induced heart muscle cell apoptosis Goto Sponge NotDistinct Permalink

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scientific article published on 12 February 2007

Attributes	Values
rdf:type	Item
description	wetenschappelijk artikel (nl) наукова стаття, опублікована у квітні 2007 (uk) article scientifique publié en 2007 (fr) im April 2007 veröffentlichter wissenschaftlicher Artikel (de) scientific article published on 12 February 2007 (en) 2007 թվականի փետրվարի 12-ին հրատարակված գիտական հոդված (hy) artículu científicu espublizáu en febreru de 2007 (ast)
publication date	wds:Q64377247-8F03E8C6-73D0-4A1B-BA37-A5DB4AE5D11D
publication date	2007-02-12 00:00:00Z (xsd:dateTime)
language of work or name	wds:Q64377247-D978EFC6-99A1-46C7-BAC5-7D6719E1613C
language of work or name	English
cites work	wds:Q64377247-744C7CD9-29F2-4E1D-BE28-940E9DEFD7D4 wds:Q64377247-C4877811-9DF0-4F26-853B-A0E09BF6233C
cites work	RETRACTED: L-type Ca2+ channel facilitation mediated by phosphorylation of the beta subunit by CaMKII. Calmodulin kinase determines calcium-dependent facilitation of L-type calcium channels
author name string	wds:Q64377247-1D5C0224-8C7D-4D92-BC7B-870199C6B5A2 wds:Q64377247-2F493ABD-69F2-456B-8FE5-83ABFCA804E4 wds:Q64377247-3F29752E-73AE-43E8-86A8-36E3FC0BD80A wds:Q64377247-D7D11F2F-FBAA-4FEC-AE51-7B7A2CF0DDAA wds:Q64377247-EB2DAA96-E286-43D3-B764-B022A783AA5A
author name string	Heping Cheng Dongmei Yang Rui-Ping Xiao Weizhong Zhu Michael T Crow
rdfs:label	Activation of CaMKIIdeltaC is a common intermediate of diverse death stimuli-induced heart muscle cell apoptosis (en) Activation of CaMKIIdeltaC is a common intermediate of diverse death stimuli-induced heart muscle cell apoptosis (nl)
skos:prefLabel	Activation of CaMKIIdeltaC is a common intermediate of diverse death stimuli-induced heart muscle cell apoptosis (en) Activation of CaMKIIdeltaC is a common intermediate of diverse death stimuli-induced heart muscle cell apoptosis (nl)
name	Activation of CaMKIIdeltaC is a common intermediate of diverse death stimuli-induced heart muscle cell apoptosis (en) Activation of CaMKIIdeltaC is a common intermediate of diverse death stimuli-induced heart muscle cell apoptosis (nl)
author	wds:Q64377247-F2DD2B6D-C219-4D72-B956-3E491DFF4791
author	Anthony Yiu-Ho Woo
title	wds:Q64377247-76A4BAAB-AF6E-47E7-8DF9-59EC64A66F08
title	Activation of CaMKIIdeltaC is a common intermediate of diverse death stimuli-induced heart muscle cell apoptosis (en)
page(s)	wds:Q64377247-82A07251-AC4A-409A-8B65-63D35277F65F
page(s)	10833-10839
instance of	wds:Q64377247-709F0FB1-D165-4C24-AC6C-A23600A65952
instance of	scholarly article
main subject	wds:Q64377247-00BDF255-F120-4E16-A6CB-AA972819D722
main subject	apoptotic process
PubMed ID	wds:Q64377247-DE94A952-10C4-469A-862E-993B01450B5A
PubMed ID	http://rdf.ncbi.nlm.nih.gov/pubchem/reference/17296607
PubMed ID	17296607
published in	wds:Q64377247-2B413AEC-812B-49B5-A472-922C06A05AF3
published in	Journal of Biological Chemistry
issue	wds:Q64377247-070325D9-9123-4EAA-940A-AF79CFAA8FBB
volume	wds:Q64377247-B21A82D2-5D40-4ADE-8122-0C3C50ED20EB
issue	14
volume	282
DOI	wds:Q64377247-799AD30F-0CE3-4CE1-9C5B-56F8FF27CA4A
DOI	http://dx.doi.org/10.1074/JBC.M611507200
DOI	10.1074/JBC.M611507200
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q64377247
is cites work of	Increased Na⁺/Ca²⁺ exchanger expression/activity constitutes a point of inflection in the progression to heart failure of hypertensive rats Early apoptosis in different models of cardiac hypertrophy induced by high renin-angiotensin system activity involves CaMKII. The signalling pathway of CaMKII-mediated apoptosis and necrosis in the ischemia/reperfusion injury. Ca2+/calmodulin-dependent kinase IIdelta causes heart failure by accumulation of p53 in dilated cardiomyopathy A β(IV)-spectrin/CaMKII signaling complex is essential for membrane excitability in mice Location matters: clarifying the concept of nuclear and cytosolic CaMKII subtypes CaMKII in myocardial hypertrophy and heart failure CaMKII negatively regulates calcineurin-NFAT signaling in cardiac myocytes A dynamic pathway for calcium-independent activation of CaMKII by methionine oxidation β-Adrenergic Receptor-Stimulated Cardiac Myocyte Apoptosis: Role of β1 Integrins Na+/K+-ATPase inhibition by ouabain induces CaMKII-dependent apoptosis in adult rat cardiac myocytes Role of CaMKII and ROS in rapid pacing-induced apoptosis Role of activated CaMKII in abnormal calcium homeostasis and I(Na) remodeling after myocardial infarction: insights from mathematical modeling The role of CaMKII in calcium-activated death pathways in bone marrow B cells Calcium/calmodulin-dependent protein kinase II links ER stress with Fas and mitochondrial apoptosis pathways. Differential integration of Ca2+-calmodulin signal in intact ventricular myocytes at low and high affinity Ca2+-calmodulin targets. β₂ AR agonists in treatment of chronic heart failure: long path to translation Cardioprotection by CaMKII-deltaB is mediated by phosphorylation of heat shock factor 1 and subsequent expression of inducible heat shock protein 70. Hydrogen peroxide sensing and signaling by protein kinases in the cardiovascular system. Strong synaptic transmission impact by copy number variations in schizophrenia A pathway and network review on beta-adrenoceptor signaling and beta blockers in cardiac remodeling beta-Arrestin-dependent activation of Ca(2+)/calmodulin kinase II after beta(1)-adrenergic receptor stimulation Oxidative-stress-induced afterdepolarizations and calmodulin kinase II signaling Protein Kinase Signaling at the Crossroads of Myocyte Life and Death in Ischemic Heart Disease CaMKII in the cardiovascular system: sensing redox states. Oestrogen confers cardioprotection by suppressing Ca2+/calmodulin-dependent protein kinase II. β-Adrenergic receptor subtype signaling in heart: from bench to bedside Beta-adrenergic receptor signaling in the heart: role of CaMKII. Phosphorylation of CEACAM1 molecule by calmodulin kinase IID in a three-dimensional model of mammary gland lumen formation Beta-adrenergic receptor mediated protection against doxorubicin-induced apoptosis in cardiomyocytes: the impact of high ambient glucose CaM Kinase II mediates maladaptive post-infarct remodeling and pro-inflammatory chemoattractant signaling but not acute myocardial ischemia/reperfusion injury S-Nitrosylation Induces Both Autonomous Activation and Inhibition of Calcium/Calmodulin-dependent Protein Kinase II δ. Critical role of nuclear calcium/calmodulin-dependent protein kinase IIdeltaB in cardiomyocyte survival in cardiomyopathy Ca2+/calmodulin-dependent protein kinase II-γ (CaMKIIγ) negatively regulates vascular smooth muscle cell proliferation and vascular remodeling Cardiotoxic and cardioprotective features of chronic β-adrenergic signaling CaMKII induces permeability transition through Drp1 phosphorylation during chronic β-AR stimulation CaMKII is a RIP3 substrate mediating ischemia- and oxidative stress-induced myocardial necroptosis. Visualizing CaMKII and CaM activity: a paradigm of compartmentalized signaling CaMKIIδ mediates β-adrenergic effects on RyR2 phosphorylation and SR Ca(2+) leak and the pathophysiological response to chronic β-adrenergic stimulation. Cardiac hypertrophy and heart failure development through Gq and CaM kinase II signaling CaMKIIdelta subtypes: localization and function CaMKII is a nodal signal for multiple programmed cell death pathways in heart Tbx5-mediated expression of Ca(2+)/calmodulin-dependent protein kinase II is necessary for zebrafish cardiac and pectoral fin morphogenesis Signaling Pathways in Cardiac Myocyte Apoptosis. The role of CaMKII in diabetic heart dysfunction

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