About: Genetic associations and regulation of expression indicate an independent role for 14q32 snoRNAs in Human Cardiovascular Disease     Goto   Sponge   NotDistinct   Permalink

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scientific article published on 01 August 2019

AttributesValues
rdf:type
description
  • wetenschappelijk artikel (nl)
  • наукова стаття, опублікована в грудні 2018 (uk)
  • im Dezember 2018 veröffentlichter wissenschaftlicher Artikel (de)
  • artículu científicu espublizáu n'agostu de 2019 (ast)
  • scientific article published on 01 August 2019 (en)
publication date
publication date
language of work or name
language of work or name
author name string
author name string
  • Jens Kastrup
  • Saskia Le Cessie
  • Rasmus S Ripa
  • Christoph Kaun
  • Stefan Böhringer
  • Eva van Ingen
rdfs:label
  • Genetic associations and regulation of expression indicate an independent role for 14q32 snoRNAs in Human Cardiovascular Disease (en)
  • Genetic associations and regulation of expression indicate an independent role for 14q32 snoRNAs in Human Cardiovascular Disease (nl)
skos:prefLabel
  • Genetic associations and regulation of expression indicate an independent role for 14q32 snoRNAs in Human Cardiovascular Disease (en)
  • Genetic associations and regulation of expression indicate an independent role for 14q32 snoRNAs in Human Cardiovascular Disease (nl)
name
  • Genetic associations and regulation of expression indicate an independent role for 14q32 snoRNAs in Human Cardiovascular Disease (en)
  • Genetic associations and regulation of expression indicate an independent role for 14q32 snoRNAs in Human Cardiovascular Disease (nl)
author
author
title
title
  • Genetic associations and regulation of expression indicate an independent role for 14q32 snoRNAs in human cardiovascular disease (en)
page(s)
page(s)
  • 1519-1532
instance of
instance of
main subject
main subject
PubMed ID
PubMed ID
PubMed ID
  • 30544252
published in
published in
issue
volume
issue
  • 10
volume
  • 115
DOI
DOI
DOI
  • 10.1093/CVR/CVY309
is about of
is cites work of
is cites work of
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