About: Martin Bornhäuser

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An Entity of Type : wikibase:Item, within Data Space : wikidata.demo.openlinksw.com associated with source document(s)

German researcher

Attributes	Values
rdf:type	Item
description	Forscher (de) investigador (es) chercheur (fr) дослідник (uk) Germany karimba ŋun nyɛ doo (dag) 研究者 (zh) 研究員 (ja) জার্মান গবেষক (bn) German researcher (en) باحث ألماني (ar) onderzoeker uit Duitsland (nl)
rdfs:label	Martin Bornhäuser (ca) Martin Bornhäuser (de) Martin Bornhäuser (en) Martin Bornhäuser (es) Martin Bornhäuser (nl) Martin Bornhäuser (sl) Martin Bornhäuser (dag) Martin Bornhäuser (ast)
skos:prefLabel	Martin Bornhäuser (ca) Martin Bornhäuser (de) Martin Bornhäuser (en) Martin Bornhäuser (es) Martin Bornhäuser (nl) Martin Bornhäuser (sl) Martin Bornhäuser (dag) Martin Bornhäuser (ast)
name	Martin Bornhäuser (ca) Martin Bornhäuser (de) Martin Bornhäuser (en) Martin Bornhäuser (es) Martin Bornhäuser (nl) Martin Bornhäuser (sl) Martin Bornhäuser (dag) Martin Bornhäuser (ast)
given name	wds:Q61053345-2360818A-F6BD-444D-B817-6783EAF0EF17
given name	Martin
instance of	wds:Q61053345-ce3afb15-4d1a-6d58-e8f6-ad4e167a3aeb
instance of	Homo sapiens
work location	wds:Q61053345-fc8c45fe-4e52-aa4a-3667-22decb1c2112
work location	Dresden
skos:altLabel	Martin Bornhaeuser (en)
occupation	wds:Q61053345-61062c09-4b51-1519-64f2-01b434b65d85
employer	wds:Q61053345-3ab2d994-4469-efed-5255-cf4ddc4f20bd
languages spoken, written or signed	wds:Q61053345-66897C70-D14B-4C04-870E-9D7712697163
sex or gender	wds:Q61053345-79a8b80b-4294-876e-d982-bafc50815715
VIAF ID	wds:Q61053345-47D4CE71-F310-4B20-A974-5F599412152E
GND ID	wds:Q61053345-9f119a6c-4a4a-e615-a0fc-fcc276cc41f8
country of citizenship	wds:Q61053345-13fb9f17-4c29-fb55-aaef-2d8730053c2f
date of birth	wds:Q61053345-063cd04e-4ab6-8aaf-d397-9396cf42c90b
educated at	wds:Q61053345-15d0bd36-4b1d-97e5-79e3-ca2afc78ea85
WorldCat Identities ID (superseded)	wds:Q61053345-FA5349E6-84CD-431E-898D-AB328ACDD877
VIAF ID	http://viaf.org/viaf/4263153775055061550008
GND ID	https://d-nb.info/gnd/1167559118
occupation	university teacher
employer	TU Dresden
languages spoken, written or signed	German
sex or gender	male
VIAF ID	4263153775055061550008
GND ID	1167559118
country of citizenship	Germany
date of birth	1966-01-01 00:00:00Z (xsd:dateTime)
educated at	University of Kiel
WorldCat Identities ID (superseded)	viaf-4263153775055061550008
ORCID iD	wds:Q61053345-1CB00A0C-49CC-4A1F-8D78-613362AFDAD1
ORCID iD	https://orcid.org/0000-0002-5916-3029
ORCID iD	0000-0002-5916-3029
ResearcherID	wds:Q61053345-fb40ba20-4552-58a6-245b-9d4fe895d739
Publons author ID	wds:Q61053345-3eda2e4f-4000-e443-f4c0-b1a012aa076c
ResearcherID	F-8101-2010
Publons author ID	2826029
is owl:sameAs of	Martin Bornhäuser
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q61053345
is author of	Autotaxin is expressed in FLT3-ITD positive acute myeloid leukemia and hematopoietic stem cells and promotes cell migration and proliferation Zoledronic acid induces apoptosis and changes the TRAIL/OPG ratio in breast cancer cells. Isolated trisomy 13 defines a homogeneous AML subgroup with high frequency of mutations in spliceosome genes and poor prognosis Allogeneic stem cell transplantation after reduced-intensity conditioning in patients with myelofibrosis: a prospective, multicenter study of the Chronic Leukemia Working Party of the European Group for Blood and Marrow Transplantation Inhibition of platelet-derived growth factor receptorbeta by imatinib mesylate suppresses proliferation and alters differentiation of human mesenchymal stem cells in vitro. Reduced-intensity conditioning versus standard conditioning before allogeneic haemopoietic cell transplantation in patients with acute myeloid leukaemia in first complete remission: a prospective, open-label randomised phase 3 trial. Donor lymphocyte infusions and second transplantation as salvage treatment for relapsed myelofibrosis after reduced-intensity allografting Impact of JAK2V617F mutation status, allele burden, and clearance after allogeneic stem cell transplantation for myelofibrosis CXCR4 blockade and Sphingosine-1-phosphate activation facilitate engraftment of haematopoietic stem and progenitor cells in a non-myeloablative transplant model Clinical-scale isolation of 'minimally manipulated' cytomegalovirus-specific donor lymphocytes for the treatment of refractory cytomegalovirus disease Azacitidine for treatment of imminent relapse in MDS or AML patients after allogeneic HSCT: results of the RELAZA trial. Minimal residual disease-directed preemptive treatment with azacitidine in patients with NPM1-mutant acute myeloid leukemia and molecular relapse A novel prognostic model in elderly patients with acute myeloid leukemia: results of 909 patients entered into the prospective AML96 trial Sequential combination of azacitidine and lenalidomide in del(5q) higher-risk myelodysplastic syndromes or acute myeloid leukemia: a phase I study Tightly anchored tissue-mimetic matrices as instructive stem cell microenvironments Clonal evolution including partial loss of human leukocyte antigen genes favoring extramedullary acute myeloid leukemia relapse after matched related allogeneic hematopoietic stem cell transplantation Standardization of Good Manufacturing Practice-compliant production of bone marrow-derived human mesenchymal stromal cells for immunotherapeutic applications Allogeneic hematopoietic cell transplantation in patients age 60-70 years with de novo high-risk myelodysplastic syndrome or secondary acute myelogenous leukemia: comparison with patients lacking donors who received azacitidine Alemtuzumab depletes dendritic cells more effectively in blood than in skin: a pilot study in patients with chronic lymphocytic leukemia Myelodysplasia is in the niche: novel concepts and emerging therapies Mesenchymal stromal cells from patients with myelodyplastic syndrome display distinct functional alterations that are modulated by lenalidomide Functional immobilization of signaling proteins enables control of stem cell fate Genetic variations of interleukin-23R (1143A>G) and BPI (A645G), but not of NOD2, are associated with acute graft-versus-host disease after allogeneic transplantation CD49d blockade by natalizumab in patients with multiple sclerosis affects steady-state hematopoiesis and mobilizes progenitors with a distinct phenotype and function. Mesenchymal stem cells can be differentiated into endothelial cells in vitro Classifying cytogenetics in patients with acute myelogenous leukemia in complete remission undergoing allogeneic transplantation: a Center for International Blood and Marrow Transplant Research study

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