About: Adverse Reproductive Outcomes in the Transgenic Ah Receptor-Deficient Mouse

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scientific article published on 01 February 1999

Attributes	Values
rdf:type	Item
description	wetenschappelijk artikel (nl) наукова стаття, опублікована в лютому 1999 (uk) im Februar 1999 veröffentlichter wissenschaftlicher Artikel (de) artículu científicu espublizáu en febreru de 1999 (ast) artikull shkencor i botuar më 01 shkurt 1999 (sq) scientific article published on 01 February 1999 (en)
publication date	wds:Q60352568-E381D6AC-E293-453B-A08C-BF269786A7A2
publication date	1999-02-01 00:00:00Z (xsd:dateTime)
author name string	wds:Q60352568-441614E0-919F-4778-A380-18A66893A669 wds:Q60352568-53818F5A-F4A7-493E-B50E-E131F3533A86 wds:Q60352568-6141CE23-2CB9-4DE2-A4DC-540B094BC248 wds:Q60352568-AE120109-C397-4882-A4DC-7768F8990F0C wds:Q60352568-EA841F99-939C-4B49-B568-4E8FAB74EFE4
author name string	Held GA Wood CR Schmid JE Pitt JA Buckalew AR
rdfs:label	Adverse Reproductive Outcomes in the Transgenic Ah Receptor-Deficient Mouse (en) Adverse Reproductive Outcomes in the Transgenic Ah Receptor-Deficient Mouse (nl) Adverse Reproductive Outcomes in the Transgenic Ah Receptor-Deficient Mouse (sq)
skos:prefLabel	Adverse Reproductive Outcomes in the Transgenic Ah Receptor-Deficient Mouse (en) Adverse Reproductive Outcomes in the Transgenic Ah Receptor-Deficient Mouse (nl) Adverse Reproductive Outcomes in the Transgenic Ah Receptor-Deficient Mouse (sq)
name	Adverse Reproductive Outcomes in the Transgenic Ah Receptor-Deficient Mouse (en) Adverse Reproductive Outcomes in the Transgenic Ah Receptor-Deficient Mouse (nl) Adverse Reproductive Outcomes in the Transgenic Ah Receptor-Deficient Mouse (sq)
author	wds:Q60352568-3AF465A7-2087-4211-AD66-09AE66E4E4DA wds:Q60352568-F1915E7E-ED44-4A61-80B7-554ADEB3CBDE
author	Janet J. Diliberto Barbara D. Abbott
title	wds:Q60352568-D286211F-2CB8-4FF2-8A9F-8ADCF94FC564
title	Adverse reproductive outcomes in the transgenic Ah receptor-deficient mouse (en)
page(s)	wds:Q60352568-D6ED308F-14AB-4EF9-A0DA-A80930137B5E
page(s)	62-70
instance of	wds:Q60352568-56E93ECE-DD26-44D4-980D-A4696BFAB52E
instance of	scholarly article
PubMed ID	wds:Q60352568-52620A9C-63CE-4EDC-99B4-81333BBC9F4D
PubMed ID	http://rdf.ncbi.nlm.nih.gov/pubchem/reference/10036219
PubMed ID	10036219
published in	wds:Q60352568-ABBD4079-52ED-41F1-9C85-6C0D69372C56
published in	Toxicology and Applied Pharmacology
issue	wds:Q60352568-502378B6-88B9-4263-B541-1301EA5A1D1D
volume	wds:Q60352568-F2D54AEC-C878-4C26-9413-E64BAAFD3DE0
issue	1
volume	155
DOI	wds:Q60352568-2000B33F-5C0A-40DB-BAA6-0526E2459490
DOI	http://dx.doi.org/10.1006/TAAP.1998.8601
DOI	10.1006/TAAP.1998.8601
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q60352568
is cites work of	Dioxin: a review of its environmental effects and its aryl hydrocarbon receptor biology The aryl hydrocarbon receptor complex and the control of gene expression Hypersensitivity of aryl hydrocarbon receptor-deficient mice to lipopolysaccharide-induced septic shock Role of the aromatic hydrocarbon receptor and [Ah] gene battery in the oxidative stress response, cell cycle control, and apoptosis Expression of aryl hydrocarbon receptor in human placentas and fetal tissues Homology modelling of the nuclear receptors: human oestrogen receptorbeta (hERbeta), the human pregnane-X-receptor (PXR), the Ah receptor (AhR) and the constitutive androstane receptor (CAR) ligand binding domains from the human oestrogen receptor a Intrinsic function of the aryl hydrocarbon (dioxin) receptor as a key factor in female reproduction AHR2 mutant reveals functional diversity of aryl hydrocarbon receptors in zebrafish Functional role of AhR in the expression of toxic effects by TCDD Portosystemic shunting and persistent fetal vascular structures in aryl hydrocarbon receptor-deficient mice The RelA NF-kappaB subunit and the aryl hydrocarbon receptor (AhR) cooperate to transactivate the c-myc promoter in mammary cells Dioxins, the aryl hydrocarbon receptor and the central regulation of energy balance How can chemical compounds alter human fertility? CYP1A in TCDD toxicity and in physiology-with particular reference to CYP dependent arachidonic acid metabolism and other endogenous substrates Omeprazole stimulates the induction of human insulin-like growth factor binding protein-1 through aryl hydrocarbon receptor activation Anti-remodeling and anti-fibrotic effects of the neuregulin-1β glial growth factor 2 in a large animal model of heart failure Regulation of aryl hydrocarbon receptor function by selective estrogen receptor modulators The role of the aryl hydrocarbon receptor in the female reproductive system Role of the aryl hydrocarbon receptor in carcinogenesis and potential as a drug target Effects of Adipocyte Aryl Hydrocarbon Receptor Deficiency on PCB-Induced Disruption of Glucose Homeostasis in Lean and Obese Mice Molecular mechanisms of the physiological functions of the aryl hydrocarbon (dioxin) receptor, a multifunctional regulator that senses and responds to environmental stimuli Inhibition of cytochrome P4501-dependent clearance of the endogenous agonist FICZ as a mechanism for activation of the aryl hydrocarbon receptor. The aryl hydrocarbon receptor (AhR) and its nuclear translocator (Arnt) are dispensable for normal mammary gland development but are required for fertility Ah receptor binding to its cognate response element is required for dioxin-mediated toxicity Ligand selectivity and gene regulation by the human aryl hydrocarbon receptor in transgenic mice The ability of the aryl hydrocarbon receptor to regulate ovarian follicle growth and estradiol biosynthesis in mice depends on stage of sexual maturity The aryl hydrocarbon receptor is required for normal gonadotropin responsiveness in the mouse ovary Sexually dimorphic behavioral responses to prenatal dioxin exposure Interference of endocrine disrupting chemicals with aromatase CYP19 expression or activity, and consequences for reproduction of teleost fish Nuclear receptor coactivator SRC-1 interacts with the Q-rich subdomain of the AhR and modulates its transactivation potential. Disruption of Ah Receptor Signaling during Mouse Development Leads to Abnormal Cardiac Structure and Function in the Adult Dietary fat alters body composition, mammary development, and cytochrome p450 induction after maternal TCDD exposure in DBA/2J mice with low-responsive aryl hydrocarbon receptors Keratinocyte Growth Factor Regulation of Aryl Hydrocarbon Receptor Activation in Colorectal Cancer Cells The Q-rich subdomain of the human Ah receptor transactivation domain is required for dioxin-mediated transcriptional activity Transgenic Humanized AHR Mouse Reveals Differences between Human and Mouse AHR Ligand Selectivity An Aryl Hydrocarbon Receptor-Mediated Amplification Loop That Enforces Cell Migration in ER-/PR-/Her2- Human Breast Cancer Cells The mechanism of AH receptor protein down-regulation (degradation) and its impact on AH receptor-mediated gene regulation Sun exposure: what molecular photodermatology tells us about its good and bad sides Transcriptional factor aryl hydrocarbon receptor (Ahr) controls cardiovascular and respiratory functions by regulating the expression of the Vav3 proto-oncogene The mouse and human Ah receptor differ in recognition of LXXLL motifs A possible role of aryl hydrocarbon receptor in spontaneous preterm birth Co-planar 3,3',4,4',5-pentachlorinated biphenyl and non-co-planar 2,2',4,6,6'-pentachlorinated biphenyl differentially induce recruitment of oestrogen receptor alpha to aryl hydrocarbon receptor target genes. Tryptamine serves as a proligand of the AhR transcriptional pathway whose activation is dependent of monoamine oxidases Induction of oxidative stress responses by dioxin and other ligands of the aryl hydrocarbon receptor AhR signaling pathways and regulatory functions Aryl Hydrocarbon Receptor Plays Protective Roles against High Fat Diet (HFD)-induced Hepatic Steatosis and the Subsequent Lipotoxicity via Direct Transcriptional Regulation of Socs3 Gene Expression Expression, Localization, and Activity of the Aryl Hydrocarbon Receptor in the Human Placenta FSH/LH-Dependent Upregulation of Ahr in Murine Granulosa Cells Is Controlled by PKA Signaling and Involves Epigenetic Regulation Specific blockage of ligand-induced degradation of the Ah receptor by proteasome but not calpain inhibitors in cell culture lines from different species

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