About: Thomas Haller

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An Entity of Type : wikibase:Item, within Data Space : wikidata.demo.openlinksw.com associated with source document(s)

researcher ORCID ID = 0000-0001-9147-4081

Attributes	Values
rdf:type	Item
description	forskare (sv) 研究者 (ja) araştırmacı (tr) wetenschapper (nl) дослідник (uk) эрдэмтэн (mn) 研究者 (zh) researcher ORCID ID = 0000-0001-9147-4081 (en)
rdfs:label	Thomas Haller (en) Thomas Haller (es) Thomas Haller (nl) Thomas Haller (sl) Thomas Haller (ast)
skos:prefLabel	Thomas Haller (en) Thomas Haller (es) Thomas Haller (nl) Thomas Haller (sl) Thomas Haller (ast)
name	Thomas Haller (en) Thomas Haller (es) Thomas Haller (nl) Thomas Haller (sl) Thomas Haller (ast)
given name	wds:Q59700149-EEB2935B-0E44-4CE3-88E8-57BC5BCD5047
given name	Thomas
instance of	wds:Q59700149-1C4B3634-A1D7-4687-99E4-04CD8CFA9669
instance of	Homo sapiens
occupation	wds:Q59700149-FCD9800A-15FC-4855-8F0B-8DA1DB0FD9DA
employer	wds:Q59700149-52387A56-79AB-4B64-8CED-A28E94DD7505
CONOR.SI ID	wds:Q59700149-0BD1FA3B-81EF-4C33-B834-71EB6715CF4F
languages spoken, written or signed	wds:Q59700149-0E89FA8B-8D81-447F-BA63-FA986C6A1832
sex or gender	wds:Q59700149-ECA3DF94-12E1-4373-AA05-D9E7A3FC97F0
VIAF ID	wds:Q59700149-DE596D90-3CA2-4377-A0BE-50370C47F690
educated at	wds:Q59700149-4370EFDC-1B8F-44E8-BD25-BA5B2F53630C
family name	wds:Q59700149-959C2C3F-A03C-4C3E-8B1B-075F92064E6C
WorldCat Identities ID (superseded)	wds:Q59700149-98FAC06B-E788-41C1-8E99-801F0C6DDBC7
VIAF ID	http://viaf.org/viaf/63154440165035341870
occupation	researcher
employer	Medical University of Innsbruck
CONOR.SI ID	184706147
languages spoken, written or signed	German
sex or gender	male
VIAF ID	63154440165035341870
educated at	Medical University of Innsbruck
family name	Haller
WorldCat Identities ID (superseded)	viaf-63154440165035341870
ORCID iD	wds:Q59700149-8E2588B2-FB94-4F19-A613-391899F7A7D3
ORCID iD	https://orcid.org/0000-0001-9147-4081
ORCID iD	0000-0001-9147-4081
ResearcherID	wds:Q59700149-C98457B9-53E4-40A2-AFE3-2254BD243F04
ResearcherID	H-9036-2015
is about of	https://www.wikidata.org/wiki/Special:EntityData/Q59700149
is author of	Fusion-activated Ca2+ entry via vesicular P2X4 receptors promotes fusion pore opening and exocytotic content release in pneumocytes Dynamics of surfactant release in alveolar type II cells. Secretion in alveolar type II cells at the interface of constitutive and regulated exocytosis. A respirometer for investigating oxidative cell metabolism: toward optimization of respiratory studies Spatio-temporal aspects, pathways and actions of Ca(2+) in surfactant secreting pulmonary alveolar type II pneumocytes The lysosomal Ca2+ pool in MDCK cells can be released by ins(1,4,5)P3-dependent hormones or thapsigargin but does not activate store-operated Ca2+ entry. Signal transduction pathways in directed migration of human monocytes induced by human growth hormone in vitro. Long-term exposure to LPS enhances the rate of stimulated exocytosis and surfactant secretion in alveolar type II cells and upregulates P2Y2 receptor expression Tracing surfactant transformation from cellular release to insertion into an air-liquid interface Fusion pore expansion is a slow, discontinuous, and Ca2+-dependent process regulating secretion from alveolar type II cells The lysosomal compartment as intracellular calcium store in MDCK cells: a possible involvement in InsP3-mediated Ca2+ release. Ca2+ entry is essential for cell strain-induced lamellar body fusion in isolated rat type II pneumocytes. Two different store-operated Ca2+ entry pathways in MDCK cells Inhibition by cytoplasmic nucleotides of a new cation channel in freshly isolated human and rat type II pneumocytes. Fusion-activated Ca(2+) entry: an "active zone" of elevated Ca(2+) during the postfusion stage of lamellar body exocytosis in rat type II pneumocytes Activation of Na+/H(+)-exchanger by transforming Ha-ras requires stimulated cellular calcium influx and is associated with rearrangement of the actin cytoskeleton. A new role for an old drug: Ambroxol triggers lysosomal exocytosis via pH-dependent Ca²⁺ release from acidic Ca²⁺ stores. Nontoxic impact of PEG-coated gold nanospheres on functional pulmonary surfactant-secreting alveolar type II cells. Recent advances in alveolar biology: some new looks at the alveolar interface. Lamellar bodies form solid three-dimensional films at the respiratory air-liquid interface. Effects of taurine on Ca2(+)-dependent force development of skinned muscle fibre preparations Exocytosis in alveolar type II cells revealed by cell capacitance and fluorescence measurements. Screening for organic acids in fish tissues with special reference to the distribution of taurine inRutilus rutilus L Human decidua-derived mesenchymal stem cells differentiate into functional alveolar type II-like cells that synthesize and secrete pulmonary surfactant complexes. Polarized light microscopy reveals physiological and drug-induced changes in surfactant membrane assembly in alveolar type II pneumocytes. Threshold calcium levels for lamellar body exocytosis in type II pneumocytes Mechanical forces impeding exocytotic surfactant release revealed by optical tweezers. Persistent fusion pores but transient fusion in alveolar type II cells Existence of exocytotic hemifusion intermediates with a lifetime of up to seconds in type II pneumocytes. Effects of perfluorocarbons on surfactant exocytosis and membrane properties in isolated alveolar type II cells. Interfacial sensing by alveolar type II cells: a new concept in lung physiology? Interfacial stress affects rat alveolar type II cell signaling and gene expression. Mechanisms of surfactant exocytosis in alveolar type II cells in vitro and in vivo Pneumocytes Assemble Lung Surfactant as Highly Packed/Dehydrated States with Optimal Surface Activity Physiological variables affecting surface film formation by native lamellar body-like pulmonary surfactant particles. High-throughput evaluation of pulmonary surfactant adsorption and surface film formation The conception of fusion pores as rate-limiting structures for surfactant secretion. A small key unlocks a heavy door: The essential function of the small hydrophobic proteins SP-B and SP-C to trigger adsorption of pulmonary surfactant lamellar bodies. Mitochondrial calcium overload is restricted to a few mitochondria in endothelial cells after cold ischemia/reperfusion Migrating Lung Monocytes Internalize and Inhibit Growth of <i>Aspergillus fumigatus</i> Conidia Effect of exogenous surfactants on viability and DNA synthesis in A549, immortalized mouse type II and isolated rat alveolar type II cells Optical measurement of surface tension in a miniaturized air-liquid interface and its application in lung physiology. Activation of L-type Ca2+ channels after purinoceptor stimulation by ATP in an alveolar epithelial cell (L2). Reduction of intracellular pH inhibits constitutive expression of cyclooxygenase-2 in human colon cancer cells. Calcium entry stimulated by swelling of Madin-Darby canine kidney cells Stimulation of surfactant exocytosis in primary alveolar type II cells by A. fumigatus
is author of	wds:Q86742599-2A918BF3-84BB-4186-BE77-6D7F3D6A7587

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